Shani Bialik

5.6k total citations · 1 hit paper
46 papers, 4.7k citations indexed

About

Shani Bialik is a scholar working on Molecular Biology, Epidemiology and Cell Biology. According to data from OpenAlex, Shani Bialik has authored 46 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Molecular Biology, 23 papers in Epidemiology and 17 papers in Cell Biology. Recurrent topics in Shani Bialik's work include Autophagy in Disease and Therapy (23 papers), Endoplasmic Reticulum Stress and Disease (12 papers) and Cell death mechanisms and regulation (12 papers). Shani Bialik is often cited by papers focused on Autophagy in Disease and Therapy (23 papers), Endoplasmic Reticulum Stress and Disease (12 papers) and Cell death mechanisms and regulation (12 papers). Shani Bialik collaborates with scholars based in Israel, United States and Czechia. Shani Bialik's co-authors include Adi Kimchi, Avital Eisenberg‐Lerner, Hans‐Uwe Simon, Gidi Shani, Richard N. Kitsis, Santosh K. Dasari, Yaara Ber, Ilana Sabanay, Boaz Inbal and Miriam Eisenstein and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Shani Bialik

45 papers receiving 4.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Life and death partners: apoptosis, autophagy and the cross-talk between them

2009 1000 citations Shani Bialik, Adi Kimchi et al. Cell Death and Differentiation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Shani Bialik Israel	28	3.0k	2.1k	859	516	443	46	4.7k
Rita Garuti Italy	20	2.4k 0.8×	2.8k 1.3×	897 1.0×	698 1.4×	560 1.3×	30	6.0k
Yushan Zhu China	33	3.1k 1.0×	2.3k 1.1×	814 0.9×	439 0.9×	288 0.7×	66	4.8k
Marco Corazzari Italy	35	2.7k 0.9×	2.7k 1.3×	1.4k 1.6×	589 1.1×	602 1.4×	87	5.6k
Kurt Degenhardt United States	22	3.0k 1.0×	2.1k 1.0×	720 0.8×	713 1.4×	401 0.9×	26	4.5k
Anne Hamacher‐Brady United States	28	2.5k 0.8×	2.2k 1.0×	508 0.6×	532 1.0×	204 0.5×	36	4.3k
Maria Perander Norway	17	2.1k 0.7×	2.2k 1.1×	804 0.9×	411 0.8×	220 0.5×	20	3.7k
Chi Li United States	25	2.5k 0.8×	1.2k 0.6×	801 0.9×	429 0.8×	403 0.9×	70	3.7k
Ezgi Tasdemir France	16	1.9k 0.6×	2.1k 1.0×	538 0.6×	426 0.8×	250 0.6×	16	3.4k
Brian Beaudoin United States	6	2.6k 0.9×	3.1k 1.5×	759 0.9×	734 1.4×	309 0.7×	6	4.2k
Hai‐Xin Yuan China	22	2.8k 0.9×	1.8k 0.9×	2.0k 2.3×	626 1.2×	304 0.7×	45	5.0k

Countries citing papers authored by Shani Bialik

Since Specialization

Citations

This map shows the geographic impact of Shani Bialik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shani Bialik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shani Bialik more than expected).

Fields of papers citing papers by Shani Bialik

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shani Bialik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shani Bialik. The network helps show where Shani Bialik may publish in the future.

Co-authorship network of co-authors of Shani Bialik

This figure shows the co-authorship network connecting the top 25 collaborators of Shani Bialik. A scholar is included among the top collaborators of Shani Bialik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shani Bialik. Shani Bialik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Eisenstein, Miriam, Tsviya Olender, Alon Savidor, et al.. (2025). The RNA-binding protein PRRC2B preserves 5′ TOP mRNA during starvation to maintain ribosome biogenesis during nutrient recovery. Nucleic Acids Research. 53(22).

Bialik, Shani, Sílvia Carvalho, Noga Kozer, et al.. (2024). Structure–activity relationship study of small-molecule inhibitor of Atg12-Atg3 protein–protein interaction. Bioorganic & Medicinal Chemistry Letters. 112. 129939–129939. 2 indexed citations

Olender, Tsviya, et al.. (2024). Loss of EIF4G2 mediates aggressiveness in distinct human endometrial cancer subpopulations with poor survival outcome in patients. Oncogene. 43(15). 1098–1112. 1 indexed citations

Eisenstein, Miriam, Alon Savidor, Yishai Levin, et al.. (2023). Loss-of-function cancer-linked mutations in the EIF4G2 non-canonical translation initiation factor. Life Science Alliance. 7(3). e202302338–e202302338. 3 indexed citations

Carvalho, Sílvia, Noga Kozer, Haim Barr, et al.. (2023). Identifying a selective inhibitor of autophagy that targets ATG12-ATG3 protein-protein interaction. Autophagy. 19(8). 2372–2385. 10 indexed citations

Olender, Tsviya, Orel Mizrahi, Shira Weingarten-Gabbay, et al.. (2022). DAP5 drives translation of specific mRNA targets with upstream ORFs in human embryonic stem cells. RNA. 28(10). 1325–1336. 15 indexed citations

Shiloh, Ruth, Yaara Ber, Miriam Eisenstein, et al.. (2018). Non-canonical activation of DAPK2 by AMPK constitutes a new pathway linking metabolic stress to autophagy. Nature Communications. 9(1). 1759–1759. 31 indexed citations

Friedlander, Gilgi, Ester Feldmesser, Elena Ainbinder, et al.. (2016). Cap-independent translation by DAP5 controls cell fate decisions in human embryonic stem cells. Genes & Development. 30(17). 1991–2004. 46 indexed citations

Ber, Yaara, et al.. (2014). DAPK2 is a novel regulator of mTORC1 activity and autophagy. Cell Death and Differentiation. 22(3). 465–475. 51 indexed citations

10.

Bialik, Shani, et al.. (2013). DAP-kinase and autophagy. APOPTOSIS. 19(2). 346–356. 65 indexed citations

11.

Weingarten-Gabbay, Shira, Debjit Khan, Noa Liberman, et al.. (2013). The translation initiation factor DAP5 promotes IRES-driven translation of p53 mRNA. Oncogene. 33(5). 611–618. 67 indexed citations

12.

Cohen, Shmuel Jaffe, Sara W. Feigelson, Shani Bialik, et al.. (2011). Tumor Suppressor Death-Associated Protein Kinase Attenuates Inflammatory Responses in the Lung. American Journal of Respiratory Cell and Molecular Biology. 46(3). 313–322. 24 indexed citations

13.

Brownstein, Zippora, Yaara Ber, Shani Bialik, et al.. (2010). SPIKE: a database of highly curated human signaling pathways. Nucleic Acids Research. 39(suppl_1). D793–D799. 61 indexed citations

14.

Bialik, Shani & Adi Kimchi. (2009). Lethal weapons: DAP-kinase, autophagy and cell death. Current Opinion in Cell Biology. 22(2). 199–205. 52 indexed citations

15.

Bialik, Shani, Tal Raveh, Géraldine Mitou, et al.. (2008). DAP-kinase is a mediator of endoplasmic reticulum stress-induced caspase activation and autophagic cell death. Cell Death and Differentiation. 15(12). 1875–1886. 219 indexed citations

16.

Bialik, Shani & Adi Kimchi. (2004). DAP-kinase as a target for drug design in cancer and diseases associated with accelerated cell death. Seminars in Cancer Biology. 14(4). 283–294. 82 indexed citations

17.

Buvoli, Massimo, et al.. (2002). Potential limitations of transcription terminators used as transgene insulators in adenoviral vectors. Gene Therapy. 9(3). 227–231. 17 indexed citations

18.

Shohat-Ophir, Galit, Taly R. Spivak-Kroizman, Ofer Cohen, et al.. (2001). The Pro-apoptotic Function of Death-associated Protein Kinase Is Controlled by a Unique Inhibitory Autophosphorylation-based Mechanism. Journal of Biological Chemistry. 276(50). 47460–47467. 138 indexed citations

19.

Bialik, Shani & Tanya Gottlieb. (2000). Death at the Dead Sea. Cell Death and Differentiation. 7(9). 851–859. 2 indexed citations

20.

Evans, Sydney M., M. Bergeron, Donna M. Ferriero, et al.. (1997). Imaging Hypoxia in Diseased Tissues. Advances in experimental medicine and biology. 428. 595–603. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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