Jung H. Kim
- Molecular Biology top 5%
- Cancer Research top 2%
- Pulmonary and Respiratory Medicine top 10%
- Health, Toxicology and Mutagenesis top 5%
- Oncology
- Co-authors
- Xuhong CaoSooryanarayana VaramballyQi CaoRam S. ManiJindan YuBharathi LaxmanDana C. DolinoyChandan Kumar‐Sinha
- Topics
- Epigenetics and DNA Methylation (11 papers)Effects and risks of endocrine disrupting chemicals (6 papers)Cancer-related gene regulation (3 papers)
- Journals
- ScienceMolecular CellPLoS ONE
- Partner nations
- United StatesSouth KoreaRussia
In The Last Decade
Jung H. Kim
39 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 109
- Molecular Biology 1.7k
- Cancer Research 827
- Pulmonary and Respiratory Medicine 240
- Health, Toxicology and Mutagenesis 237
- Oncology 199
Countries citing papers authored by Jung H. Kim
This map shows the geographic impact of Jung H. Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jung H. Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jung H. Kim more than expected).
Fields of papers citing papers by Jung H. Kim
This network shows the impact of papers produced by Jung H. Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jung H. Kim. The network helps show where Jung H. Kim may publish in the future.
Co-authorship network of co-authors of Jung H. Kim
This figure shows the co-authorship network connecting the top 25 collaborators of Jung H. Kim. A scholar is included among the top collaborators of Jung H. Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jung H. Kim. Jung H. Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 7 | |
| 3 | 51 | |
| 4 | 14 | |
| 5 | 62 | |
| 6 | 62 | |
| 7 | 9 | |
| 8 | 60 | |
| 9 | 116 | |
| 10 | 60 | |
| 11 | 24 | |
| 12 | 29 | |
| 13 | Genomic Loss of microRNA-101 Leads to Overexpression of Histone Methyltransferase EZH2 in Cancerbreakdown → | 833 |
| 14 | 452 | |
| 15 | 88 | |
| 16 | 111 | |
| 17 | 44 | |
| 18 | 11 | |
| 19 | 26 | |
| 20 | 2 |
About Jung H. Kim
Jung H. Kim is a scholar working on Cancer Research, Health, Toxicology and Mutagenesis and Molecular Biology, having authored 40 papers that have together received 2.3k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (11 papers), Effects and risks of endocrine disrupting chemicals (6 papers) and Cancer-related gene regulation (3 papers). The work is most often cited by research in Cancer Research (827 citations), Molecular Biology (1.7k citations) and Health, Toxicology and Mutagenesis (237 citations). Jung H. Kim has collaborated with scholars based in United States, South Korea and Russia. Frequent co-authors include Xuhong Cao, Sooryanarayana Varambally, Qi Cao, Ram S. Mani, Jindan Yu, Bharathi Laxman, Dana C. Dolinoy, Chandan Kumar‐Sinha, Maureen A. Sartor and Nallasivam Palanisamy. Their work appears in journals such as Science, Molecular Cell and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.