Julia Sim

4.7k total citations · 1 hit paper
37 papers, 3.8k citations indexed

About

Julia Sim is a scholar working on Immunology, Molecular Biology and Neurology. According to data from OpenAlex, Julia Sim has authored 37 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Immunology, 11 papers in Molecular Biology and 6 papers in Neurology. Recurrent topics in Julia Sim's work include T-cell and B-cell Immunology (15 papers), Immunotherapy and Immune Responses (13 papers) and Immune Cell Function and Interaction (6 papers). Julia Sim is often cited by papers focused on T-cell and B-cell Immunology (15 papers), Immunotherapy and Immune Responses (13 papers) and Immune Cell Function and Interaction (6 papers). Julia Sim collaborates with scholars based in United States, United Kingdom and Singapore. Julia Sim's co-authors include Douglas R. Spitz, John H. Russell, Lisa A. Ridnour, Kenneth M. Murphy, Yong J. Lee, Sandra Galoforo, Theresa L. Murphy, Larry W. Oberley, Natalie Zimmerman and Michelle A. Hurchla and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Julia Sim

37 papers receiving 3.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

BTLA is a lymphocyte inhibitory receptor with similarities to CTLA-4 and PD-1

2003 695 citations Julia Sim, Theresa L. Murphy et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Julia Sim United States	25	1.7k	1.2k	654	429	405	37	3.8k
Ralf M. Zwacka United States	33	695 0.4×	2.2k 1.8×	533 0.8×	329 0.8×	677 1.7×	57	4.2k
Gadiparthi N. Rao United States	41	897 0.5×	3.0k 2.4×	512 0.8×	300 0.7×	711 1.8×	130	5.2k
H. Martin Seidel United States	23	1.1k 0.6×	2.0k 1.6×	762 1.2×	149 0.3×	260 0.6×	37	3.3k
Karen Block United States	35	1.1k 0.7×	2.0k 1.6×	269 0.4×	298 0.7×	613 1.5×	47	4.5k
Shan Zhu China	34	1.2k 0.7×	2.9k 2.4×	846 1.3×	176 0.4×	1.3k 3.2×	89	5.3k
Byong Chul Yoo South Korea	37	523 0.3×	2.7k 2.2×	755 1.2×	246 0.6×	786 1.9×	137	4.5k
Kenji Tago Japan	34	1.3k 0.8×	1.9k 1.6×	454 0.7×	125 0.3×	281 0.7×	105	4.0k
Maddalena Fratelli Italy	33	422 0.3×	2.7k 2.2×	584 0.9×	241 0.6×	413 1.0×	89	4.8k
Russell M. Medford United States	32	1.6k 1.0×	2.7k 2.2×	342 0.5×	215 0.5×	519 1.3×	47	6.3k
Zhiheng He China	35	558 0.3×	1.7k 1.4×	552 0.8×	175 0.4×	388 1.0×	99	4.8k

Countries citing papers authored by Julia Sim

Since Specialization

Citations

This map shows the geographic impact of Julia Sim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia Sim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia Sim more than expected).

Fields of papers citing papers by Julia Sim

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Julia Sim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia Sim. The network helps show where Julia Sim may publish in the future.

Co-authorship network of co-authors of Julia Sim

This figure shows the co-authorship network connecting the top 25 collaborators of Julia Sim. A scholar is included among the top collaborators of Julia Sim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia Sim. Julia Sim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Harp, Chelsea R. Parker, Angela S. Archambault, Julia Sim, et al.. (2018). B cells are capable of independently eliciting rapid reactivation of encephalitogenic CD4 T cells in a murine model of multiple sclerosis. PLoS ONE. 13(6). e0199694–e0199694. 16 indexed citations

Lin, Chih‐Chung, Tara R. Bradstreet, Elizabeth A. Schwarzkopf, et al.. (2016). IL-1–induced Bhlhe40 identifies pathogenic T helper cells in a model of autoimmune neuroinflammation. The Journal of Experimental Medicine. 213(2). 251–271. 66 indexed citations

Archambault, Angela S., Javier A. Carrero, Lisa G. Barnett, et al.. (2013). Cutting Edge: Conditional MHC Class II Expression Reveals a Limited Role for B Cell Antigen Presentation in Primary and Secondary CD4 T Cell Responses. The Journal of Immunology. 191(2). 545–550. 31 indexed citations

Edelson, Brian T., Tara R. Bradstreet, Wumesh KC, et al.. (2011). Batf3-Dependent CD11blow/− Peripheral Dendritic Cells Are GM-CSF-Independent and Are Not Required for Th Cell Priming after Subcutaneous Immunization. PLoS ONE. 6(10). e25660–e25660. 95 indexed citations

Graham, Daniel B., Grzegorz B. Gmyrek, Laura Piccio, et al.. (2010). ITAM signaling in dendritic cells controls T helper cell priming by regulating MHC class II recycling. Blood. 116(17). 3208–3218. 15 indexed citations

Boomer, Jonathan, Christine Deppong, Julia Sim, et al.. (2009). Targeted Knock-In Mice Expressing Mutations of CD28 Reveal an Essential Pathway for Costimulation. Molecular and Cellular Biology. 29(13). 3710–3721. 63 indexed citations

Schraml, Barbara U., Kai Hildner, Wataru Ise, et al.. (2009). The AP-1 transcription factor Batf controls TH17 differentiation. Nature. 460(7253). 405–409. 466 indexed citations

Lees, Jason R., Paul T. Golumbek, Julia Sim, Denise Dorsey, & John H. Russell. (2008). Regional CNS responses to IFN-γ determine lesion localization patterns during EAE pathogenesis. The Journal of Experimental Medicine. 205(11). 2633–2642. 145 indexed citations

Sim, Julia, et al.. (2006). A Tumor Necrosis Factor Receptor 1-Dependent Conversation between Central Nervous System-Specific T Cells and the Central Nervous System Is Required for Inflammatory Infiltration of the Spinal Cord. American Journal Of Pathology. 168(4). 1200–1209. 44 indexed citations

10.

Laszlo, Andrei, Amanda Harvey, Julia Sim, et al.. (2006). Alterations in heat-induced radiosensitization accompanied by nuclear structure alterations in Chinese hamster cells. International Journal of Hyperthermia. 22(1). 43–60. 12 indexed citations

11.

Archambault, Angela S., Julia Sim, Erin E. McCandless, Robyn S. Klein, & John H. Russell. (2006). Region-specific regulation of inflammation and pathogenesis in experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 181(1-2). 122–132. 24 indexed citations

12.

Archambault, Angela S., et al.. (2005). Defining antigen‐dependent stages of T cell migration from the blood to the central nervous system parenchyma. European Journal of Immunology. 35(4). 1076–1085. 56 indexed citations

13.

Ridnour, Lisa A., Julia Sim, Jinah Choi, et al.. (2005). Nitric oxide-induced resistance to hydrogen peroxide stress is a glutamate cysteine ligase activity-dependent process. Free Radical Biology and Medicine. 38(10). 1361–1371. 22 indexed citations

14.

Hook, G. J., Douglas R. Spitz, Julia Sim, et al.. (2004). Evaluation of Parameters of Oxidative Stress afterIn VitroExposure to FMCW- and CDMA-Modulated Radiofrequency Radiation Fields. Radiation Research. 162(5). 497–504. 40 indexed citations

15.

Spitz, Douglas R., Julia Sim, Lisa A. Ridnour, Sandra Galoforo, & Yong J. Lee. (2000). Glucose Deprivation‐Induced Oxidative Stress in Human Tumor Cells: A Fundamental Defect in Metabolism?. Annals of the New York Academy of Sciences. 899(1). 349–362. 275 indexed citations

16.

Lee, Yong J., Sandra Galoforo, Julia Sim, et al.. (2000). Dominant-negative Jun N-terminal protein kinase (JNK-1) inhibits metabolic oxidative stress during glucose deprivation in a human breast carcinoma cell line. Free Radical Biology and Medicine. 28(4). 575–584. 28 indexed citations

17.

Goswami, Prabhat C., Jamie Sheren, Lee D. Albee, et al.. (2000). Cell Cycle-coupled Variation in Topoisomerase IIα mRNA Is Regulated by the 3′-Untranslated Region. Journal of Biological Chemistry. 275(49). 38384–38392. 61 indexed citations

18.

Ridnour, Lisa A., Julia Sim, Michael A. Hayward, et al.. (2000). A Spectrophotometric Method for the Direct Detection and Quantitation of Nitric Oxide, Nitrite, and Nitrate in Cell Culture Media. Analytical Biochemistry. 281(2). 223–229. 196 indexed citations

19.

Polavarapu, Rathnagiri, Douglas R. Spitz, Julia Sim, et al.. (1998). Increased lipid peroxidation and impaired antioxidant enzyme function is associated with pathological liver injury in experimental alcoholic liver disease in rats fed diets high in corn oil and fish oil. Hepatology. 27(5). 1317–1323. 256 indexed citations

20.

Lee, Yong J., Sandra Galoforo, Christine M. Berns, et al.. (1998). Glucose Deprivation-induced Cytotoxicity and Alterations in Mitogen-activated Protein Kinase Activation Are Mediated by Oxidative Stress in Multidrug-resistant Human Breast Carcinoma Cells. Journal of Biological Chemistry. 273(9). 5294–5299. 205 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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