Julia Bollrath
- Immunology top 2%
- Molecular Biology top 10%
- Oncology top 5%
- Cancer Research top 5%
- Surgery top 10%
- Co-authors
- Florian R. GretenFiona PowrieMelek C. ArkanRoland M. SchmidChris SchieringKrista AdelmannThomas KrausgruberThomas Kirchner
- Topics
- NF-κB Signaling Pathways (4 papers)Cytokine Signaling Pathways and Interactions (4 papers)Immune Cell Function and Interaction (3 papers)
- Cited by
- ImmunologyCancer ResearchOncology
- Journals
- NatureCellCancer Cell
- Partner nations
- GermanyUnited KingdomUnited States
In The Last Decade
Julia Bollrath
8 papers receiving 2.5k citations
Hit Papers
Peers
Comparison fields: 5 of 98
- Immunology 1.5k
- Molecular Biology 827
- Oncology 725
- Cancer Research 466
- Surgery 423
Countries citing papers authored by Julia Bollrath
This map shows the geographic impact of Julia Bollrath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Julia Bollrath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Julia Bollrath more than expected).
Fields of papers citing papers by Julia Bollrath
This network shows the impact of papers produced by Julia Bollrath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Julia Bollrath. The network helps show where Julia Bollrath may publish in the future.
Co-authorship network of co-authors of Julia Bollrath
This figure shows the co-authorship network connecting the top 25 collaborators of Julia Bollrath. A scholar is included among the top collaborators of Julia Bollrath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Julia Bollrath. Julia Bollrath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 33 | |
| 2 | 18 | |
| 3 | The alarmin IL-33 promotes regulatory T-cell function in the intestinebreakdown → | 784 |
| 4 | 67 | |
| 5 | gp130-Mediated Stat3 Activation in Enterocytes Regulates Cell Survival and Cell-Cycle Progression during Colitis-Associated Tumorigenesisbreakdown → | 780 |
| 6 | 3 | |
| 7 | 315 | |
| 8 | NF-κB Is a Negative Regulator of IL-1β Secretion as Revealed by Genetic and Pharmacological Inhibition of IKKβbreakdown → | 503 |
About Julia Bollrath
Julia Bollrath is a scholar working on Immunology, Cancer Research and Oncology, having authored 8 papers that have together received 2.5k indexed citations. Recurring topics across this work include NF-κB Signaling Pathways (4 papers), Cytokine Signaling Pathways and Interactions (4 papers) and Immune Cell Function and Interaction (3 papers). The work is most often cited by research in Immunology (1.5k citations), Cancer Research (466 citations) and Oncology (725 citations). Julia Bollrath has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Florian R. Greten, Fiona Powrie, Melek C. Arkan, Roland M. Schmid, Chris Schiering, Krista Adelmann, Thomas Krausgruber, Thomas Kirchner, Özge Canli and Tim Nebelsiek. Their work appears in journals such as Nature, Cell and Cancer Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.