José M. Inácio

462 total citations
27 papers, 342 citations indexed

About

José M. Inácio is a scholar working on Molecular Biology, Biotechnology and Genetics. According to data from OpenAlex, José M. Inácio has authored 27 papers receiving a total of 342 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 5 papers in Biotechnology and 4 papers in Genetics. Recurrent topics in José M. Inácio's work include Congenital heart defects research (15 papers), Pluripotent Stem Cells Research (5 papers) and Enzyme Production and Characterization (5 papers). José M. Inácio is often cited by papers focused on Congenital heart defects research (15 papers), Pluripotent Stem Cells Research (5 papers) and Enzyme Production and Characterization (5 papers). José M. Inácio collaborates with scholars based in Portugal, United States and Spain. José M. Inácio's co-authors include Isabel Sá‐Nogueira, José António Belo, Sara Marques, Carla Costa, Isabel Bento, Daniele de Sanctis, Peter F. Lindley, Luı́s Jaime Mota, Fernando Bonet and Paulo N. G. Pereira and has published in prestigious journals such as PLoS ONE, Journal of Bacteriology and International Journal of Molecular Sciences.

In The Last Decade

José M. Inácio

25 papers receiving 340 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
José M. Inácio Portugal 11 180 119 114 72 62 27 342
Chien-Chung Hsia Taiwan 8 83 0.5× 122 1.0× 42 0.4× 154 2.1× 57 0.9× 10 397
Peihong Jiang China 11 193 1.1× 47 0.4× 29 0.3× 66 0.9× 19 0.3× 31 343
Youssef Ben Ammar Japan 10 186 1.0× 52 0.4× 184 1.6× 99 1.4× 139 2.2× 12 374
Wan Sun China 10 228 1.3× 68 0.6× 22 0.2× 85 1.2× 24 0.4× 19 406
Giles O. Elliott United Kingdom 10 251 1.4× 123 1.0× 139 1.2× 129 1.8× 50 0.8× 11 450
C. Granzow Germany 11 144 0.8× 94 0.8× 64 0.6× 47 0.7× 17 0.3× 31 307
Stefanie Fischer Germany 7 272 1.5× 37 0.3× 65 0.6× 17 0.2× 16 0.3× 11 374
Mark Rabinowitz United States 7 271 1.5× 31 0.3× 72 0.6× 27 0.4× 22 0.4× 10 460
Deqiang Kong China 11 275 1.5× 77 0.6× 28 0.2× 61 0.8× 4 0.1× 33 411
Nergiz Doğan Türkiye 4 266 1.5× 52 0.4× 48 0.4× 98 1.4× 22 0.4× 5 395

Countries citing papers authored by José M. Inácio

Since Specialization
Citations

This map shows the geographic impact of José M. Inácio's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by José M. Inácio with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites José M. Inácio more than expected).

Fields of papers citing papers by José M. Inácio

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by José M. Inácio. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by José M. Inácio. The network helps show where José M. Inácio may publish in the future.

Co-authorship network of co-authors of José M. Inácio

This figure shows the co-authorship network connecting the top 25 collaborators of José M. Inácio. A scholar is included among the top collaborators of José M. Inácio based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with José M. Inácio. José M. Inácio is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Inácio, José M., et al.. (2025). Cell Reprogramming, Transdifferentiation, and Dedifferentiation Approaches for Heart Repair. International Journal of Molecular Sciences. 26(7). 3063–3063.
2.
Lozano-Velasco, Estefanía, et al.. (2024). miRNAs in Heart Development and Disease. International Journal of Molecular Sciences. 25(3). 1673–1673. 14 indexed citations
3.
Inácio, José M., et al.. (2023). Gene-Edited Human-Induced Pluripotent Stem Cell Lines to Elucidate DAND5 Function throughout Cardiac Differentiation. Cells. 12(4). 520–520. 3 indexed citations
4.
Inácio, José M., Miguel Pinheiro, Francisco Vasques‐Nóvoa, et al.. (2023). Myocardial RNA Sequencing Reveals New Potential Therapeutic Targets in Heart Failure with Preserved Ejection Fraction. Biomedicines. 11(8). 2131–2131. 3 indexed citations
5.
Bonet, Fernando, et al.. (2022). CCBE1 in Cardiac Development and Disease. Frontiers in Genetics. 13. 836694–836694. 4 indexed citations
6.
Inácio, José M., et al.. (2021). DAND5 Inactivation Enhances Cardiac Differentiation in Mouse Embryonic Stem Cells. Frontiers in Cell and Developmental Biology. 9. 629430–629430. 2 indexed citations
7.
8.
Silva, Marta M., Patrícia Gomes‐Alves, Sara Sousa Rosa, et al.. (2018). Full-length human CCBE1 production and purification: leveraging bioprocess development for high quality glycosylation attributes and functionality. Journal of Biotechnology. 285. 6–14. 2 indexed citations
9.
Pereira, Paulo N. G., João Facucho-Oliveira, José M. Inácio, et al.. (2018). Loss of Ccbe1 affects cardiac-specification and cardiomyocyte differentiation in mouse embryonic stem cells. PLoS ONE. 13(10). e0205108–e0205108. 5 indexed citations
10.
Bonet, Fernando, et al.. (2018). CCBE1 is required for coronary vessel development and proper coronary artery stem formation in the mouse heart. Developmental Dynamics. 247(10). 1135–1145. 19 indexed citations
11.
Inácio, José M., Isabel M. Carreira, Joana Barbosa Melo, et al.. (2018). Generation and characterization of a human iPS cell line from a patient-related control to study disease mechanisms associated with DAND5 p.R152H alteration. Stem Cell Research. 29. 202–206. 2 indexed citations
12.
Inácio, José M., Isabel M. Carreira, Joana Barbosa Melo, et al.. (2017). Generation of human iPSC line from a patient with laterality defects and associated congenital heart anomalies carrying a DAND5 missense alteration. Stem Cell Research. 25. 152–156. 8 indexed citations
13.
Vitorino, Marta, Ana Cristina Silva, José M. Inácio, et al.. (2015). Xenopus Pkdcc1 and Pkdcc2 Are Two New Tyrosine Kinases Involved in the Regulation of JNK Dependent Wnt/PCP Signaling Pathway. PLoS ONE. 10(8). e0135504–e0135504. 11 indexed citations
14.
Vitorino, Marta, et al.. (2014). Expression pattern of zcchc24 during early Xenopus development. The International Journal of Developmental Biology. 58(1). 45–50. 4 indexed citations
15.
Inácio, José M., et al.. (2014). Expression and Function of Ccbe1 in the Chick Early Cardiogenic Regions Are Required for Correct Heart Development. PLoS ONE. 9(12). e115481–e115481. 10 indexed citations
16.
Inácio, José M., Sara Marques, Tetsuya Nakamura, et al.. (2013). The Dynamic Right-to-Left Translocation of Cerl2 Is Involved in the Regulation and Termination of Nodal Activity in the Mouse Node. PLoS ONE. 8(3). e60406–e60406. 27 indexed citations
17.
Sanctis, Daniele de, José M. Inácio, Peter F. Lindley, Isabel Sá‐Nogueira, & Isabel Bento. (2010). New evidence for the role of calcium in the glycosidase reaction of GH43 arabinanases. FEBS Journal. 277(21). 4562–4574. 41 indexed citations
18.
Sanctis, Daniele de, et al.. (2008). Overproduction, crystallization and preliminary X-ray characterization of Abn2, an endo-1,5-α-arabinanase fromBacillus subtilis. Acta Crystallographica Section F Structural Biology and Crystallization Communications. 64(7). 636–638. 3 indexed citations
19.
Inácio, José M. & Isabel Sá‐Nogueira. (2007). trans -Acting Factors and cis Elements Involved in Glucose Repression of Arabinan Degradation in Bacillus subtilis. Journal of Bacteriology. 189(22). 8371–8376. 8 indexed citations
20.
Inácio, José M., Carla Costa, & Isabel Sá‐Nogueira. (2003). Distinct molecular mechanisms involved in carbon catabolite repression of the arabinose regulon in Bacillus subtilis. Microbiology. 149(9). 2345–2355. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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