Jonathan Cremer

1.0k citations
41 papers · 428 · h-index 13

Impact in

    • IL-33, ST2, and ILC Pathways
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Inflammatory Bowel Disease

Papers in

    • IL-33, ST2, and ILC Pathways 17
    • Immune Cell Function and Interaction 6
    • Galectins and Cancer Biology 2
    • Eosinophilic Esophagitis 11

Jonathan Cremer

34 papers receiving 426 citations

Peers

Jonathan Cremer
Comparison fields: 5 of 75
  • Immunology 188
  • Genetics 155
  • Gastroenterology 15
  • Developmental Neuroscience 10
  • Surgery 94
Replace Francesca Ammoscato with:
Francesca Ammoscato United Kingdom
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Shiroh Tanoue Japan
Na Diao China
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Citations per field
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Citations per year

Countries citing papers authored by Jonathan Cremer

Since Specialization
Citations

This map shows the geographic impact of Jonathan Cremer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan Cremer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan Cremer more than expected).

Fields of papers citing papers by Jonathan Cremer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan Cremer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan Cremer. The network helps show where Jonathan Cremer may publish in the future.

Co-authors

The 25 scholars most cited alongside Jonathan Cremer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonathan Cremer Line = papers co-authored together Jonathan Cremer links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 41 papers — load more, or switch the sort, to bring in the rest.

#Work
1 201251
2 201345
3 201539
4 201638
5 202032
6 202029
7 201722
8 201018
9 202118
10 201916
11 202215
12 201814
13 201713
14 201211
15 201911
16 201910
17 20239
18 20205
19 20205
20 20244

About Jonathan Cremer

Jonathan Cremer is a scholar working on Immunology, Surgery, Genetics, Physiology and Epidemiology, having authored 41 papers that have together received 428 indexed citations. Recurring topics across this work include IL-33, ST2, and ILC Pathways (17 papers), Eosinophilic Esophagitis (11 papers), Inflammatory Bowel Disease (9 papers), Immune Cell Function and Interaction (6 papers), Asthma and respiratory diseases (5 papers), Microscopic Colitis (4 papers), Chronic Obstructive Pulmonary Disease (COPD) Research (3 papers) and Galectins and Cancer Biology (2 papers). The work is most often cited by research in Immunology (188 citations), Genetics (155 citations), Gastroenterology (15 citations), Developmental Neuroscience (10 citations) and Surgery (94 citations). Jonathan Cremer has collaborated with scholars based in Belgium, United States and Netherlands. Frequent co-authors include Jan Ceuppens, Séverine Vermeire, Gert Van Assche, Marc Ferrante, Christine Breynaert, Gert De Hertogh, Clémentine Perrier, Bram Verstockt, Louis Boon and Paul Rutgeerts. Their work appears in journals such as Frontiers in Immunology, Journal of Crohn s and Colitis, Inflammatory Bowel Diseases, PLoS ONE and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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