Gitta Seleznik

995 total citations
16 papers, 505 citations indexed

About

Gitta Seleznik is a scholar working on Surgery, Oncology and Immunology. According to data from OpenAlex, Gitta Seleznik has authored 16 papers receiving a total of 505 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Surgery, 6 papers in Oncology and 6 papers in Immunology. Recurrent topics in Gitta Seleznik's work include Pancreatitis Pathology and Treatment (8 papers), Pancreatic and Hepatic Oncology Research (6 papers) and Neuroendocrine Tumor Research Advances (3 papers). Gitta Seleznik is often cited by papers focused on Pancreatitis Pathology and Treatment (8 papers), Pancreatic and Hepatic Oncology Research (6 papers) and Neuroendocrine Tumor Research Advances (3 papers). Gitta Seleznik collaborates with scholars based in Switzerland, Germany and France. Gitta Seleznik's co-authors include Mathias Heikenwälder, Nicolas Zeller, Maximilian Wolf, Sergei A. Nedospasov, Sergei I. Grivennikov, Dan R. Littman, Dmitry V. Kuprash, Gérard Eberl, Alexei V. Tumanov and Andrey Kruglov and has published in prestigious journals such as Science, Oncogene and Gut.

In The Last Decade

Gitta Seleznik

16 papers receiving 500 citations

Peers

Gitta Seleznik

IMMUN

SURGE

ONCOL

EPIDE

Bingyi Shi China

Julia Jackson United States

Francesca Ammoscato United Kingdom

Chaoying Zhou China

Pieter Ruytinx Belgium

Gabriela Schiechl Germany

Shinichiro Yokota United States

Runhua Hou United States

Helena Cucak Denmark

Ushā Śarmā India

Bingyi Shi China

Gitta Seleznik

343 ×1.5

IMMUN

83 ×0.4

SURGE

120 ×0.8

101 ×0.8

ONCOL

81 ×1.2

EPIDE

Citations per year, relative to Gitta Seleznik Gitta Seleznik (= 1×) peers Bingyi Shi

Countries citing papers authored by Gitta Seleznik

Since Specialization

Citations

This map shows the geographic impact of Gitta Seleznik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gitta Seleznik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gitta Seleznik more than expected).

Fields of papers citing papers by Gitta Seleznik

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gitta Seleznik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gitta Seleznik. The network helps show where Gitta Seleznik may publish in the future.

Co-authorship network of co-authors of Gitta Seleznik

This figure shows the co-authorship network connecting the top 25 collaborators of Gitta Seleznik. A scholar is included among the top collaborators of Gitta Seleznik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gitta Seleznik. Gitta Seleznik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

16 of 16 papers shown

Seleznik, Gitta, Theresia Reding, Daniela Lenggenhager, et al.. (2022). De novo expression of gastrokines in pancreatic precursor lesions impede the development of pancreatic cancer. Oncogene. 41(10). 1507–1517. 4 indexed citations

Malagola, Ermanno, Rong Chen, Gitta Seleznik, et al.. (2018). Enhanced proliferation of pancreatic acinar cells in MRL/MpJ mice is driven by severe acinar injury but independent of inflammation. Scientific Reports. 8(1). 9391–9391. 1 indexed citations

Visentin, Michele, Francesco Baschieri, Gitta Seleznik, et al.. (2018). Serotonin uptake is required for Rac1 activation in Kras‐induced acinar‐to‐ductal metaplasia in the pancreas. The Journal of Pathology. 246(3). 352–365. 18 indexed citations

Seleznik, Gitta, Theresia Reding, Anurag Gupta, et al.. (2017). Development of autoimmune pancreatitis is independent of CDKN1A/p21-mediated pancreatic inflammation. Gut. 67(9). 1663–1673. 16 indexed citations

Reding, Theresia, Cristian Palmière, Marc Schiesser, et al.. (2017). The pancreas responds to remote damage and systemic stress by secretion of the pancreatic secretory proteins PSP/regI and PAPregIII. Pancreatology. 17(3). S3–S3. 1 indexed citations

Reding, Theresia, Cristian Palmière, Marc Schiesser, et al.. (2017). The pancreas responds to remote damage and systemic stress by secretion of the pancreatic secretory proteins PSP/regI and PAP/regIII. Oncotarget. 8(18). 30162–30174. 25 indexed citations

Malagola, Ermanno, Rong Chen, Gitta Seleznik, et al.. (2017). Class I histone deacetylase inhibition improves pancreatitis outcome by limiting leukocyte recruitment and acinar‐to‐ductal metaplasia. British Journal of Pharmacology. 174(21). 3865–3880. 24 indexed citations

Bertrand, Mathieu J.M., Caroline Remouchamps, Gitta Seleznik, et al.. (2015). NIK promotes tissue destruction independently of the alternative NF-κB pathway through TNFR1/RIP1-induced apoptosis. Cell Death and Differentiation. 22(12). 2020–2033. 33 indexed citations

Reding, Theresia, Gitta Seleznik, Ermanno Malagola, et al.. (2015). Inactivation of TGFβ receptor II signalling in pancreatic epithelial cells promotes acinar cell proliferation, acinar‐to‐ductal metaplasia and fibrosis during pancreatitis. The Journal of Pathology. 238(3). 434–445. 18 indexed citations

10.

Buzzi, Raphael M., Alberto B. Silva, Ermanno Malagola, et al.. (2015). Serotonin promotes acinar dedifferentiation following pancreatitis‐induced regeneration in the adult pancreas. The Journal of Pathology. 237(4). 495–507. 16 indexed citations

11.

Seleznik, Gitta, Jessica Zöller, Tracy O’Connor, Rolf Graf, & Mathias Heikenwälder. (2014). The role of lymphotoxin signaling in the development of autoimmune pancreatitis and associated secondary extra-pancreatic pathologies. Cytokine & Growth Factor Reviews. 25(2). 125–137. 10 indexed citations

12.

Hehl, Adrian B., Gitta Seleznik, Richard A. Zuellig, et al.. (2014). p21^WAF1^/Cip1 limits senescence and acinar‐to‐ductal metaplasia formation during pancreatitis. The Journal of Pathology. 235(3). 502–514. 22 indexed citations

13.

Kruglov, Andrey, Sergei I. Grivennikov, Dmitry V. Kuprash, et al.. (2013). Nonredundant Function of Soluble LTα ₃ Produced by Innate Lymphoid Cells in Intestinal Homeostasis. Science. 342(6163). 1243–1246. 203 indexed citations

14.

Wolf, Maximilian, Gitta Seleznik, Nicolas Zeller, & Mathias Heikenwälder. (2010). The unexpected role of lymphotoxin β receptor signaling in carcinogenesis: from lymphoid tissue formation to liver and prostate cancer development. Oncogene. 29(36). 5006–5018. 79 indexed citations

15.

Wolf, Monika, Gitta Seleznik, & Mathias Heikenwälder. (2010). Lymphotoxin’s Link to Carcinogenesis: Friend or Foe? From Lymphoid Neogenesis to Hepatocellular Carcinoma and Prostate Cancer. Advances in experimental medicine and biology. 691. 231–249. 3 indexed citations

16.

Westrich, Jason R., Chufa He, Raymond Chen, et al.. (2010). Factors Affecting Residence Time of Mesenchymal Stromal Cells (MSC) Injected into the Myocardium. Cell Transplantation. 19(8). 937–948. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact