Jon Reed

931 total citations
24 papers, 739 citations indexed

About

Jon Reed is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Jon Reed has authored 24 papers receiving a total of 739 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 5 papers in Physiology and 4 papers in Neurology. Recurrent topics in Jon Reed's work include Metabolomics and Mass Spectrometry Studies (5 papers), Alzheimer's disease research and treatments (5 papers) and Mitochondrial Function and Pathology (4 papers). Jon Reed is often cited by papers focused on Metabolomics and Mass Spectrometry Studies (5 papers), Alzheimer's disease research and treatments (5 papers) and Mitochondrial Function and Pathology (4 papers). Jon Reed collaborates with scholars based in United States, United Kingdom and Norway. Jon Reed's co-authors include Michael Mullan, Fiona Crawford, Gogce Crynen, Nils Aall Barricelli, Laila Abdullah, Ghania Ait‐Ghezala, Benoit Mouzon, James Evans, Venkatarajan S. Mathura and Corbin Bachmeier and has published in prestigious journals such as PLoS ONE, The FASEB Journal and European Journal of Pharmacology.

In The Last Decade

Jon Reed

24 papers receiving 713 citations

Peers

Jon Reed
Hojin Choi South Korea
Ai‐Ling Lin United States
Seong A. Shin South Korea
Yuanbo Wu China
H. Frey Finland
Yubo Xie China
Hojin Choi South Korea
Jon Reed
Citations per year, relative to Jon Reed Jon Reed (= 1×) peers Hojin Choi

Countries citing papers authored by Jon Reed

Since Specialization
Citations

This map shows the geographic impact of Jon Reed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon Reed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon Reed more than expected).

Fields of papers citing papers by Jon Reed

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon Reed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon Reed. The network helps show where Jon Reed may publish in the future.

Co-authorship network of co-authors of Jon Reed

This figure shows the co-authorship network connecting the top 25 collaborators of Jon Reed. A scholar is included among the top collaborators of Jon Reed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon Reed. Jon Reed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cui, Jie, Jon Reed, Gogce Crynen, et al.. (2019). Proteomic Identification of Pathways Responsible for the Estradiol Therapeutic Window in AD Animal Models. Frontiers in Cellular Neuroscience. 13. 437–437. 3 indexed citations
2.
Carmo, Sonia Do, Gogce Crynen, Tiffany Paradis, et al.. (2017). Hippocampal Proteomic Analysis Reveals Distinct Pathway Deregulation Profiles at Early and Late Stages in a Rat Model of Alzheimer’s-Like Amyloid Pathology. Molecular Neurobiology. 55(4). 3451–3476. 23 indexed citations
3.
Zakirova, Zuchra, Jon Reed, Gogce Crynen, et al.. (2017). Complementary proteomic approaches reveal mitochondrial dysfunction, immune and inflammatory dysregulation in a mouse model of Gulf War Illness. PROTEOMICS - CLINICAL APPLICATIONS. 11(9-10). 29 indexed citations
4.
Abdullah, Laila, James Evans, Gogce Crynen, et al.. (2016). Translational potential of long-term decreases in mitochondrial lipids in a mouse model of Gulf War Illness. Toxicology. 372. 22–33. 50 indexed citations
5.
Emmerich, Tanja, Laila Abdullah, Joseph Ojo, et al.. (2016). Mild TBI Results in a Long-Term Decrease in Circulating Phospholipids in a Mouse Model of Injury. NeuroMolecular Medicine. 19(1). 122–135. 28 indexed citations
6.
Tzekov, Radouil, Megan S. Orlando, Benoit Mouzon, et al.. (2016). Sub-Chronic Neuropathological and Biochemical Changes in Mouse Visual System after Repetitive Mild Traumatic Brain Injury. PLoS ONE. 11(4). e0153608–e0153608. 41 indexed citations
7.
Abdullah, Laila, James Evans, Ben Shackleton, et al.. (2016). P3‐124: APOE4 Genotype‐Dependent Deficits in DHA Containing Phospholipids and DHA Transporters in the Cerebrovasculature of Alzheimer's Disease Patients. Alzheimer s & Dementia. 12(7S_Part_18). 3 indexed citations
8.
Zakirova, Zuchra, Gogce Crynen, Jon Reed, et al.. (2015). Gulf War Agent Exposure Causes Impairment of Long-Term Memory Formation and Neuropathological Changes in a Mouse Model of Gulf War Illness. PLoS ONE. 10(3). e0119579–e0119579. 69 indexed citations
9.
Emmerich, Tanja, Laila Abdullah, Gogce Crynen, et al.. (2015). Plasma Lipidomic Profiling in a Military Population of Mild Traumatic Brain Injury and Post-Traumatic Stress Disorder with Apolipoprotein E ɛ4–Dependent Effect. Journal of Neurotrauma. 33(14). 1331–1348. 43 indexed citations
10.
Abdullah, Laila, James Evans, Scott Ferguson, et al.. (2014). Lipidomic analyses identify injury‐specific phospholipid changes 3 mo after traumatic brain injury. The FASEB Journal. 28(12). 5311–5321. 65 indexed citations
11.
Bachmeier, Corbin, Gary M. Levin, David Beaulieu‐Abdelahad, Jon Reed, & Michael Mullan. (2013). Effect of Venlafaxine and Desvenlafaxine on Drug Efflux Protein Expression and Biodistribution In Vivo. Journal of Pharmaceutical Sciences. 102(10). 3838–3843. 20 indexed citations
12.
Paris, Daniel, David Beaulieu‐Abdelahad, Laila Abdullah, et al.. (2012). Anti-inflammatory activity of anatabine via inhibition of STAT3 phosphorylation. European Journal of Pharmacology. 698(1-3). 145–153. 46 indexed citations
13.
Abdullah, Laila, Gogce Crynen, Jon Reed, et al.. (2011). Proteomic CNS Profile of Delayed Cognitive Impairment in Mice Exposed to Gulf War Agents. NeuroMolecular Medicine. 13(4). 275–288. 59 indexed citations
14.
Paris, Daniel, David Beaulieu‐Abdelahad, Corbin Bachmeier, et al.. (2011). Anatabine lowers Alzheimer's Aβ production in vitro and in vivo. European Journal of Pharmacology. 670(2-3). 384–391. 51 indexed citations
15.
Crawford, Fiona, Gogce Crynen, Jon Reed, et al.. (2011). Identification of Plasma Biomarkers of TBI Outcome Using Proteomic Approaches in an APOE Mouse Model. Journal of Neurotrauma. 29(2). 246–260. 23 indexed citations
16.
17.
Berhane, Beniam T., et al.. (2009). Structural optimization of a CXCR2-directed antagonist that indirectly inhibits γ-secretase and reduces Aβ. Bioorganic & Medicinal Chemistry. 17(23). 8102–8112. 18 indexed citations
18.
Abdullah, Laila, et al.. (2009). Proteomic Analysis of Human Neuronal Cells Treated with the Gulf War Agent Pyridostigmine Bromide. Journal of Proteomics & Bioinformatics. 2(10). 439–444. 1 indexed citations
20.
Reed, Jon & Edith D. Hendley. (1994). Blood cell changes in spontaneously hypertensive rats are not all associated with the hypertensive phenotype. Journal of Hypertension. 12(4). 391???400–391???400. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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