Nadee Nissanka

1.6k total citations
19 papers, 1.2k citations indexed

About

Nadee Nissanka is a scholar working on Molecular Biology, Clinical Biochemistry and Cellular and Molecular Neuroscience. According to data from OpenAlex, Nadee Nissanka has authored 19 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Clinical Biochemistry and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Nadee Nissanka's work include Mitochondrial Function and Pathology (13 papers), ATP Synthase and ATPases Research (6 papers) and Metabolism and Genetic Disorders (6 papers). Nadee Nissanka is often cited by papers focused on Mitochondrial Function and Pathology (13 papers), ATP Synthase and ATPases Research (6 papers) and Metabolism and Genetic Disorders (6 papers). Nadee Nissanka collaborates with scholars based in United States, United Kingdom and Brazil. Nadee Nissanka's co-authors include Carlos T. Moraes, Milena Pinto, Sandra R. Bacman, Francisca Díaz, Siôn L. Williams, Cláudia V. Pereira, Susana Peralta, Michal Minczuk, James B. Stewart and María Miranda and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Nature Communications.

In The Last Decade

Nadee Nissanka

19 papers receiving 1.2k citations

Peers

Nadee Nissanka

PHYSI

CMN

NEURO

Lorena Perrone Italy

Anna Majander Finland

Nicole J. Van Bergen Australia

Andrea Bernacchia Italy

Jessica Hui United States

Alessandra Torraco Italy

Lee Stanyer United Kingdom

Verónica Eisner Chile

Liron Boyman United States

Wojciech Hilgier Poland

Lorena Perrone Italy

Nadee Nissanka

742 ×0.9

317 ×1.1

455 ×2.4

PHYSI

120 ×1.1

CMN

145 ×1.5

NEURO

Citations per year, relative to Nadee Nissanka Nadee Nissanka (= 1×) peers Lorena Perrone

Countries citing papers authored by Nadee Nissanka

Since Specialization

Citations

This map shows the geographic impact of Nadee Nissanka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadee Nissanka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadee Nissanka more than expected).

Fields of papers citing papers by Nadee Nissanka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadee Nissanka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadee Nissanka. The network helps show where Nadee Nissanka may publish in the future.

Co-authorship network of co-authors of Nadee Nissanka

This figure shows the co-authorship network connecting the top 25 collaborators of Nadee Nissanka. A scholar is included among the top collaborators of Nadee Nissanka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadee Nissanka. Nadee Nissanka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Nissanka, Nadee, et al.. (2024). Absence of both MGME1 and POLG EXO abolishes mtDNA whereas absence of either creates unique mtDNA duplications. Journal of Biological Chemistry. 300(4). 107128–107128. 3 indexed citations

Lang, Anna L., Nadee Nissanka, Ruy A. Louzada, et al.. (2023). A Defect in Mitochondrial Complex III but Not in Complexes I or IV Causes Early β-Cell Dysfunction and Hyperglycemia in Mice. Diabetes. 72(9). 1262–1276. 13 indexed citations

Pinto, Milena, Francisca Díaz, Nadee Nissanka, et al.. (2022). Adult-Onset Deficiency of Mitochondrial Complex III in a Mouse Model of Alzheimer’s Disease Decreases Amyloid Beta Plaque Formation. Molecular Neurobiology. 59(10). 6552–6566. 3 indexed citations

García, Sofía, Ami P. Raval, Nadee Nissanka, et al.. (2021). Enhanced glycolysis and GSK3 inactivation promote brain metabolic adaptations following neuronal mitochondrial stress. Human Molecular Genetics. 31(5). 692–704. 5 indexed citations

Nissanka, Nadee & Carlos T. Moraes. (2020). Mitochondrial DNA heteroplasmy in disease and targeted nuclease‐based therapeutic approaches. EMBO Reports. 21(3). e49612–e49612. 75 indexed citations

Nissanka, Nadee, et al.. (2020). Hypoxia Promotes Mitochondrial Complex I Abundance via HIF-1α in Complex III and Complex IV Deficient Cells. Cells. 9(10). 2197–2197. 11 indexed citations

Nissanka, Nadee, Michal Minczuk, & Carlos T. Moraes. (2019). Mechanisms of Mitochondrial DNA Deletion Formation. Trends in Genetics. 35(3). 235–244. 60 indexed citations

Pereira, Cláudia V., Nadee Nissanka, Tania Arguello, et al.. (2019). Photobiomodulation enhancement of cell proliferation at 660 nm does not require cytochrome c oxidase. Journal of Photochemistry and Photobiology B Biology. 194. 71–75. 58 indexed citations

Bacman, Sandra R., Nadee Nissanka, & Carlos T. Moraes. (2019). Cybrid technology. Methods in cell biology. 155. 415–439. 8 indexed citations

10.

Bacman, Sandra R., Johanna H.K. Kauppila, Cláudia V. Pereira, et al.. (2018). MitoTALEN reduces mutant mtDNA load and restores tRNAAla levels in a mouse model of heteroplasmic mtDNA mutation. Nature Medicine. 24(11). 1696–1700. 200 indexed citations

11.

Nissanka, Nadee, Carlos T. Moraes, & Milena Pinto. (2018). Image-Based Analysis of Mitochondrial Area and Counting from Adult Mouse Dopaminergic Neurites. BIO-PROTOCOL. 8(16). e2471–e2471. 3 indexed citations

12.

Nissanka, Nadee, et al.. (2018). The mitochondrial DNA polymerase gamma degrades linear DNA fragments precluding the formation of deletions. Nature Communications. 9(1). 2491–2491. 97 indexed citations

13.

Peralta, Susana, Steffi Goffart, Siôn L. Williams, et al.. (2018). ATAD3 controls mitochondrial cristae structure in mouse muscle, influencing mtDNA replication and cholesterol levels. Journal of Cell Science. 131(13). 74 indexed citations

14.

García, Sofía, Nadee Nissanka, Edson Assunção Mareco, et al.. (2018). Overexpression of PGC‐1α in aging muscle enhances a subset of young‐like molecular patterns. Aging Cell. 17(2). 60 indexed citations

15.

Pinto, Milena, Nadee Nissanka, & Carlos T. Moraes. (2017). Lack of Parkin Anticipates the Phenotype and Affects Mitochondrial Morphology and mtDNA Levels in a Mouse Model of Parkinson's Disease. Journal of Neuroscience. 38(4). 1042–1053. 47 indexed citations

16.

Nissanka, Nadee & Carlos T. Moraes. (2017). Mitochondrial DNA damage and reactive oxygen species in neurodegenerative disease. FEBS Letters. 592(5). 728–742. 331 indexed citations

17.

Pinto, Milena, Nadee Nissanka, Susana Peralta, et al.. (2016). Pioglitazone ameliorates the phenotype of a novel Parkinson’s disease mouse model by reducing neuroinflammation. Molecular Neurodegeneration. 11(1). 25–25. 60 indexed citations

18.

Zakirova, Zuchra, Gogce Crynen, Jon Reed, et al.. (2015). Gulf War Agent Exposure Causes Impairment of Long-Term Memory Formation and Neuropathological Changes in a Mouse Model of Gulf War Illness. PLoS ONE. 10(3). e0119579–e0119579. 69 indexed citations

19.

Barmettler, Anne, et al.. (2014). Magnetic Systems for Tarsorrhaphy. Ophthalmic Plastic and Reconstructive Surgery. 30(4). 305–308. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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