Johannes Bufler

3.4k total citations
101 papers, 2.7k citations indexed

About

Johannes Bufler is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Johannes Bufler has authored 101 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 70 papers in Molecular Biology, 69 papers in Cellular and Molecular Neuroscience and 26 papers in Neurology. Recurrent topics in Johannes Bufler's work include Ion channel regulation and function (64 papers), Neuroscience and Neuropharmacology Research (47 papers) and Nicotinic Acetylcholine Receptors Study (27 papers). Johannes Bufler is often cited by papers focused on Ion channel regulation and function (64 papers), Neuroscience and Neuropharmacology Research (47 papers) and Nicotinic Acetylcholine Receptors Study (27 papers). Johannes Bufler collaborates with scholars based in Germany, United Kingdom and United States. Johannes Bufler's co-authors include Reinhard Dengler, Christian Franke, Klaus Krampfl, Gertrud Haeseler, M. Leuwer, Friedrich Schlesinger, Julian Großkreutz, J. Dudél, K Krampfl and S. Piepenbrock and has published in prestigious journals such as The Journal of Chemical Physics, PLoS ONE and Neurology.

In The Last Decade

Johannes Bufler

99 papers receiving 2.7k citations

Peers

Johannes Bufler
Gonzalo E. Torres United States
Paul McGonigle United States
John Sharkey United Kingdom
Emanuele Sher United Kingdom
Andrew D. Medhurst United Kingdom
Matthew P. Galloway United States
Gonzalo E. Torres United States
Johannes Bufler
Citations per year, relative to Johannes Bufler Johannes Bufler (= 1×) peers Gonzalo E. Torres

Countries citing papers authored by Johannes Bufler

Since Specialization
Citations

This map shows the geographic impact of Johannes Bufler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Johannes Bufler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Johannes Bufler more than expected).

Fields of papers citing papers by Johannes Bufler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Johannes Bufler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Johannes Bufler. The network helps show where Johannes Bufler may publish in the future.

Co-authorship network of co-authors of Johannes Bufler

This figure shows the co-authorship network connecting the top 25 collaborators of Johannes Bufler. A scholar is included among the top collaborators of Johannes Bufler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Johannes Bufler. Johannes Bufler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mohammadi, Bahram, et al.. (2007). Correlation between distal motor latency and compound muscle action potential in amyotrophic lateral sclerosis. Neurological Research. 29(5). 425–428. 5 indexed citations
2.
Maljevic, Snezana, Klaus Krampfl, Joana Cobilanschi, et al.. (2006). A mutation in the GABAA receptor α1‐subunit is associated with absence epilepsy. Annals of Neurology. 59(6). 983–987. 154 indexed citations
3.
Krampfl, Klaus, Snezana Maljevic, Patrick Cossette, et al.. (2005). Molecular analysis of the A322D mutation in the GABAA receptor α1‐subunit causing juvenile myoclonic epilepsy. European Journal of Neuroscience. 22(1). 10–20. 52 indexed citations
4.
5.
Schlesinger, Friedrich, et al.. (2005). Two mechanisms of action of the adamantane derivative IEM‐1460 at human AMPA‐type glutamate receptors. British Journal of Pharmacology. 145(5). 656–663. 20 indexed citations
6.
Dengler, Reinhard, Nils von Neuhoff, Johannes Bufler, et al.. (2005). Amyotrophic Lateral Sclerosis: New Developments in Diagnostic Markers. Neurodegenerative Diseases. 2(3-4). 177–184. 24 indexed citations
7.
Krampfl, Klaus, et al.. (2005). Effects of propofol on recombinant AMPA receptor channels. European Journal of Pharmacology. 511(1). 1–7. 8 indexed citations
8.
Schlesinger, Friedrich, et al.. (2004). Ligand‐gated channels in early mesencephalic neuronal precursors: immunocytochemical and electrophysiological analysis. European Journal of Neuroscience. 19(9). 2371–2376. 9 indexed citations
9.
Ahrens, Jörg, Gertrud Haeseler, M. Leuwer, et al.. (2004). 2,6 Di-tert-butylphenol, a Nonanesthetic Propofol Analog, Modulates α1β Glycine Receptor Function in a Manner Distinct from Propofol. Anesthesia & Analgesia. 99(1). 91–96. 23 indexed citations
10.
Krampfl, K, Susanne Petri, Friedrich Götz, Bahram Mohammadi, & Johannes Bufler. (2003). Amyotrophic lateral sclerosis (ALS) and mirror movements in a patient with polymicrogyria. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders. 4(4). 266–269. 10 indexed citations
11.
Großkreutz, Julian, et al.. (2003). Kinetic properties of human AMPA‐type glutamate receptors expressed in HEK293 cells. European Journal of Neuroscience. 17(6). 1173–1178. 55 indexed citations
12.
Petri, Susanne, K Krampfl, Claudia Grothe, et al.. (2003). Distribution of GABAAReceptor mRNA in the Motor Cortex of ALS Patients. Journal of Neuropathology & Experimental Neurology. 62(10). 1041–1051. 80 indexed citations
13.
Schmidt, Carolin, Angela Abicht, K Krampfl, et al.. (2003). Congenital myasthenic syndrome due to a novel missense mutation in the gene encoding choline acetyltransferase. Neuromuscular Disorders. 13(3). 245–251. 41 indexed citations
14.
Bufler, Johannes, et al.. (2001). Pentobarbital and brilliant green modulate the current response of recombinant rat kainate-type GluR6 receptor channels differentially. Neuroscience Letters. 312(2). 91–94. 6 indexed citations
15.
Schlesinger, Friedrich, et al.. (2001). Functional diversity of recombinant human AMPA type glutamate receptors: possible implications for selective vulnerability of motor neurons. Journal of the Neurological Sciences. 191(1-2). 19–23. 14 indexed citations
16.
Bufler, Johannes, Wolfram Kreß, Almuth Brandis, et al.. (2000). Okulopharyngeale Muskeldystrophie. Der Nervenarzt. 71(12). 1003–1006.
17.
Krampfl, K, Johannes Bufler, Alexandra Lepier, J. Dudél, & Helmuth Adelsberger. (2000). Desensitization characteristics of rat recombinant GABA A receptors consisting of α 1 β 2 γ 2S and α 1 β 2 subunits expressed in HEK293 cells. Neuroscience Letters. 278(1-2). 21–24. 21 indexed citations
18.
Jahn, Klaus, Johannes Bufler, Helmuth Adelsberger, et al.. (1997). Activation kinetics and single channel properties of recombinant α1δ2γ2L GABAA receptor channels. Neuroreport. 8(16). 3443–3446. 17 indexed citations
19.
Bufler, Johannes, A. Weindl, Thomas Arzberger, Klaus Jahn, & Hanns Hatt. (1996). Patch-clamp study on membrane properties and transmitter activated currents of rabbit area postrema neurons. Journal of Comparative Physiology A. 178(6). 771–8. 13 indexed citations
20.
Bufler, Johannes, Christian Franke, H. Parnas, & J. Dudél. (1996). Open Channel Block by Physostigmine and Procaine in Embryonic‐like Nicotinic Receptors of Mouse Muscle. European Journal of Neuroscience. 8(4). 677–687. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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