Friedrich Schlesinger

670 total citations
20 papers, 541 citations indexed

About

Friedrich Schlesinger is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Pharmacology. According to data from OpenAlex, Friedrich Schlesinger has authored 20 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Cellular and Molecular Neuroscience, 18 papers in Molecular Biology and 2 papers in Pharmacology. Recurrent topics in Friedrich Schlesinger's work include Neuroscience and Neuropharmacology Research (16 papers), Ion channel regulation and function (15 papers) and Nicotinic Acetylcholine Receptors Study (6 papers). Friedrich Schlesinger is often cited by papers focused on Neuroscience and Neuropharmacology Research (16 papers), Ion channel regulation and function (15 papers) and Nicotinic Acetylcholine Receptors Study (6 papers). Friedrich Schlesinger collaborates with scholars based in Germany, China and Canada. Friedrich Schlesinger's co-authors include Johannes Bufler, Klaus Krampfl, Reinhard Dengler, Julian Großkreutz, W. Melzer, Joana Cobilanschi, Daniel Ursu, Armin Heils, Snezana Maljevic and Patrick Cossette and has published in prestigious journals such as Annals of Neurology, Neuroscience and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Friedrich Schlesinger

20 papers receiving 538 citations

Peers

Friedrich Schlesinger
Xue-Min Gao United States
Olsen Rw United States
YogendraSinh H. Raol United States
Ruth Henteleff United States
Friedrich Schlesinger
Citations per year, relative to Friedrich Schlesinger Friedrich Schlesinger (= 1×) peers María Ramos

Countries citing papers authored by Friedrich Schlesinger

Since Specialization
Citations

This map shows the geographic impact of Friedrich Schlesinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Friedrich Schlesinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Friedrich Schlesinger more than expected).

Fields of papers citing papers by Friedrich Schlesinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Friedrich Schlesinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Friedrich Schlesinger. The network helps show where Friedrich Schlesinger may publish in the future.

Co-authorship network of co-authors of Friedrich Schlesinger

This figure shows the co-authorship network connecting the top 25 collaborators of Friedrich Schlesinger. A scholar is included among the top collaborators of Friedrich Schlesinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Friedrich Schlesinger. Friedrich Schlesinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Jin, Lingjing, et al.. (2012). The Two Different Effects of the Potential Neuroprotective Compound Minocycline on AMPA-Type Glutamate Receptors. Pharmacology. 89(3-4). 156–162. 5 indexed citations
2.
Song, Yunping, et al.. (2010). Changes in extracellular pH affect glycine receptor channels expressed in HEK 293 cells. European Journal of Pharmacology. 636(1-3). 59–64. 8 indexed citations
3.
Song, Yunping, et al.. (2009). Synaptic currents and transmitter responses in human NT2 neurons differentiated in aggregate culture. Neuroscience Letters. 468(3). 207–210. 21 indexed citations
4.
Jahn, Klaus, Alexandra Kotsiari, Friedrich Schlesinger, et al.. (2009). Analysis of Neuroprotective Effects of Valproic Acid on Primary Motor Neurons in Monoculture or Co-cultures with Astrocytes or Schwann Cells. Cellular and Molecular Neurobiology. 29(6-7). 1037–1043. 10 indexed citations
5.
Jin, Lingjing, Friedrich Schlesinger, Yunping Song, Reinhard Dengler, & Klaus Krampfl. (2009). The Interaction of the Neuroprotective Compounds Riluzole and Phenobarbital with AMPA-Type Glutamate Receptors: A Patch-Clamp Study. Pharmacology. 85(1). 54–62. 17 indexed citations
6.
Jahn, Klaus, et al.. (2008). Molecular mechanisms of interaction between the neuroprotective substance riluzole and GABAA-receptors. Naunyn-Schmiedeberg s Archives of Pharmacology. 378(1). 53–63. 21 indexed citations
7.
Maljevic, Snezana, Klaus Krampfl, Joana Cobilanschi, et al.. (2006). A mutation in the GABAA receptor α1‐subunit is associated with absence epilepsy. Annals of Neurology. 59(6). 983–987. 154 indexed citations
8.
Jahn, Klaus, Julian Großkreutz, Kirsten Haastert‐Talini, et al.. (2006). Temporospatial coupling of networked synaptic activation of AMPA-type glutamate receptor channels and calcium transients in cultured motoneurons. Neuroscience. 142(4). 1019–1029. 29 indexed citations
9.
10.
Schlesinger, Friedrich, et al.. (2005). Two mechanisms of action of the adamantane derivative IEM‐1460 at human AMPA‐type glutamate receptors. British Journal of Pharmacology. 145(5). 656–663. 20 indexed citations
11.
Krampfl, Klaus, et al.. (2005). Effects of propofol on recombinant AMPA receptor channels. European Journal of Pharmacology. 511(1). 1–7. 8 indexed citations
12.
Timmer, Marco, Julian Großkreutz, Friedrich Schlesinger, et al.. (2005). Dopaminergic properties and function after grafting of attached neural precursor cultures. Neurobiology of Disease. 21(3). 587–606. 38 indexed citations
13.
Schlesinger, Friedrich, et al.. (2005). Desensitization and resensitization are independently regulated in human recombinant GluR subunit coassemblies. Synapse. 55(3). 176–182. 13 indexed citations
14.
Klingler, Werner, J.J.A. Heffron, Karin Jurkat‐Rott, et al.. (2005). 3,4-Methylenedioxymethamphetamine (Ecstasy) Activates Skeletal Muscle Nicotinic Acetylcholine Receptors. Journal of Pharmacology and Experimental Therapeutics. 314(3). 1267–1273. 28 indexed citations
15.
Schlesinger, Friedrich, et al.. (2004). Ligand‐gated channels in early mesencephalic neuronal precursors: immunocytochemical and electrophysiological analysis. European Journal of Neuroscience. 19(9). 2371–2376. 9 indexed citations
16.
Schlesinger, Friedrich, Klaus Krampfl, Gertrud Haeseler, Reinhard Dengler, & Johannes Bufler. (2004). Competitive and open channel block of recombinant nAChR channels by different antibiotics. Neuromuscular Disorders. 14(5). 307–312. 14 indexed citations
17.
Großkreutz, Julian, et al.. (2003). Kinetic properties of human AMPA‐type glutamate receptors expressed in HEK293 cells. European Journal of Neuroscience. 17(6). 1173–1178. 55 indexed citations
18.
Krampfl, Klaus, et al.. (2002). Control of kinetic properties of GluR2 flop AMPA‐type channels: impact of R/G nuclear editing. European Journal of Neuroscience. 15(1). 51–62. 56 indexed citations
19.
Schlesinger, Friedrich, et al.. (2001). Functional diversity of recombinant human AMPA type glutamate receptors: possible implications for selective vulnerability of motor neurons. Journal of the Neurological Sciences. 191(1-2). 19–23. 14 indexed citations
20.
Krampfl, Klaus, et al.. (2000). Pentobarbital Has Curare-Like Effects on Adult-Type Nicotinic Acetylcholine Receptor Channel Currents. Anesthesia & Analgesia. 90(4). 970–974. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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