Jesús Aldudo

7.4k total citations
28 papers, 1.1k citations indexed

About

Jesús Aldudo is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Jesús Aldudo has authored 28 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Physiology, 14 papers in Molecular Biology and 7 papers in Neurology. Recurrent topics in Jesús Aldudo's work include Alzheimer's disease research and treatments (23 papers), Neuroinflammation and Neurodegeneration Mechanisms (6 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Jesús Aldudo is often cited by papers focused on Alzheimer's disease research and treatments (23 papers), Neuroinflammation and Neurodegeneration Mechanisms (6 papers) and Endoplasmic Reticulum Stress and Disease (4 papers). Jesús Aldudo collaborates with scholars based in Spain, United States and Canada. Jesús Aldudo's co-authors include María J. Bullido, Fernando Valdivieso, Marı́a Recuero, Isabel Sastre, Soraya Santana, María J. Artiga, Jesús Vázquez, Ana Frank, Miguel A. Garcı́a and Miguel García and has published in prestigious journals such as Nature Genetics, PLoS ONE and FEBS Letters.

In The Last Decade

Jesús Aldudo

27 papers receiving 1.1k citations

Peers

Jesús Aldudo
W. Sue T. Griffin United States
Kai Zhong China
Nicole Maphis United States
Rahasson R. Ager United States
Chulman Jo South Korea
Jesús Aldudo
Citations per year, relative to Jesús Aldudo Jesús Aldudo (= 1×) peers Marı́a Recuero

Countries citing papers authored by Jesús Aldudo

Since Specialization
Citations

This map shows the geographic impact of Jesús Aldudo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jesús Aldudo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jesús Aldudo more than expected).

Fields of papers citing papers by Jesús Aldudo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jesús Aldudo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jesús Aldudo. The network helps show where Jesús Aldudo may publish in the future.

Co-authorship network of co-authors of Jesús Aldudo

This figure shows the co-authorship network connecting the top 25 collaborators of Jesús Aldudo. A scholar is included among the top collaborators of Jesús Aldudo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jesús Aldudo. Jesús Aldudo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Sastre, Isabel, et al.. (2023). Herpes Simplex Virus Type 1 Induces AD-like Neurodegeneration Markers in Human Progenitor and Differentiated ReNcell VM Cells. Microorganisms. 11(5). 1205–1205. 3 indexed citations
3.
Sastre, Isabel, Marı́a Recuero, Ana Frank, et al.. (2021). LAMP2 deficiency attenuates the neurodegeneration markers induced by HSV-1 infection. Neurochemistry International. 146. 105032–105032. 7 indexed citations
4.
Sastre, Isabel, et al.. (2021). Matrix metalloproteinase 14 regulates HSV-1 infection in neuroblastoma cells. Antiviral Research. 192. 105116–105116. 6 indexed citations
5.
Sastre, Isabel, et al.. (2018). The lysosome system is severely impaired in a cellular model of neurodegeneration induced by HSV-1 and oxidative stress. Neurobiology of Aging. 68. 5–17. 24 indexed citations
6.
Santana, Soraya, et al.. (2015). Herpes simplex virus type 2 infection induces AD-like neurodegeneration markers in human neuroblastoma cells. Neurobiology of Aging. 36(10). 2737–2747. 50 indexed citations
7.
Santana, Soraya, Isabel Sastre, Marı́a Recuero, María J. Bullido, & Jesús Aldudo. (2013). Oxidative Stress Enhances Neurodegeneration Markers Induced by Herpes Simplex Virus Type 1 Infection in Human Neuroblastoma Cells. PLoS ONE. 8(10). e75842–e75842. 49 indexed citations
8.
Bolós, Marta, et al.. (2013). Choroid plexus implants rescue Alzheimer’s disease-like pathologies by modulating amyloid-β degradation. Cellular and Molecular Life Sciences. 71(15). 2947–2955. 27 indexed citations
9.
Recuero, Marı́a, et al.. (2013). A Free Radical-Generating System Regulates AβPP Metabolism/Processing: Involvement of the Ubiquitin/Proteasome and Autophagy/Lysosome Pathways. Journal of Alzheimer s Disease. 34(3). 637–647. 8 indexed citations
10.
Santana, Soraya, Marı́a Recuero, María J. Bullido, Fernando Valdivieso, & Jesús Aldudo. (2011). Herpes simplex virus type I induces the accumulation of intracellular β-amyloid in autophagic compartments and the inhibition of the non-amyloidogenic pathway in human neuroblastoma cells. Neurobiology of Aging. 33(2). 430.e19–430.e33. 106 indexed citations
11.
Recuero, Marı́a, Teresa Muñoz‐Galdeano, Jesús Aldudo, et al.. (2010). A free radical‐generating system regulates APP metabolism/processing. FEBS Letters. 584(22). 4611–4618. 18 indexed citations
12.
Aldudo, Jesús, Marı́a Recuero, Isabel Sastre, et al.. (2008). Presenilin 1 Polymorphism Associated with Alzheimer’s Disease in Apolipoprotein E4Carriers. Dementia and Geriatric Cognitive Disorders. 26(5). 440–444. 7 indexed citations
13.
Bullido, María J., María J. Artiga, Jesús Aldudo, et al.. (2006). A TAP2 genotype associated with Alzheimer's disease in APOE4 carriers. Neurobiology of Aging. 28(4). 519–523. 24 indexed citations
14.
Bullido, María J., Jesús Aldudo, Ana Frank, et al.. (2000). A polymorphism in the tau gene associated with risk for Alzheimer's disease. Neuroscience Letters. 278(1-2). 49–52. 60 indexed citations
15.
Bullido, María J., Pilar Guallar‐Castillón, María J. Artiga, et al.. (2000). Alzheimer's risk associated with human apolipoprotein E, alpha-2 macroglobulin and lipoprotein receptor related protein polymorphisms: absence of genetic interactions, and modulation by gender. Neuroscience Letters. 289(3). 213–216. 37 indexed citations
16.
Aldudo, Jesús, María J. Bullido, & Fernando Valdivieso. (1999). DGGE method for the mutational analysis of the coding and proximal promoter regions of the Alzheimer's disease presenilin-1 gene: Two novel mutations. Human Mutation. 14(5). 433–439. 24 indexed citations
17.
Bullido, María J., María J. Artiga, Marı́a Recuero, et al.. (1998). A polymorphism in the regulatory region of APOE associated with risk for Alzheimer's dementia. Nature Genetics. 18(1). 69–71. 241 indexed citations
18.
Artiga, María J., María J. Bullido, Isabel Sastre, et al.. (1998). Allelic polymorphisms in the transcriptional regulatory region of apolipoprotein E gene. FEBS Letters. 421(2). 105–108. 200 indexed citations
19.
Aldudo, Jesús, María J. Bullido, Txomin Arbizu, Rafael Oliva, & Fernando Valdivieso. (1998). Identification of a novel mutation (Leu282Arg) of the human presenilin 1 gene in Alzheimer's disease. Neuroscience Letters. 240(3). 174–176. 23 indexed citations
20.
Cazorla, Pilar, et al.. (1995). Location of an epitope shared by Alzheimer's amyloid peptide and brain creatine kinase using a newly developed monoclonal antibody. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1270(2-3). 149–156. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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