Jeffrey N. Keller
- Molecular Biology top 0.5%
- Physiology top 0.1%
- Neurology top 0.1%
- Cellular and Molecular Neuroscience top 0.5%
- Cell Biology top 0.5%
- Co-authors
- William R. MarkesberyAnnadora J. Bruce‐KellerMark P. MattsonQunxing DingKeith B. HanniEdgardo DimayugaSunita GuptaD. Allan Butterfield
- Topics
- Alzheimer's disease research and treatments (55 papers)Ubiquitin and proteasome pathways (40 papers)Endoplasmic Reticulum Stress and Disease (31 papers)
- Partner nations
- United StatesItalyArgentina
In The Last Decade
Jeffrey N. Keller
218 papers receiving 17.0k citations
Hit Papers
Peers
Comparison fields: 5 of 161
- Molecular Biology 7.6k
- Physiology 6.6k
- Neurology 2.7k
- Cellular and Molecular Neuroscience 2.5k
- Cell Biology 2.0k
Countries citing papers authored by Jeffrey N. Keller
This map shows the geographic impact of Jeffrey N. Keller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeffrey N. Keller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeffrey N. Keller more than expected).
Fields of papers citing papers by Jeffrey N. Keller
This network shows the impact of papers produced by Jeffrey N. Keller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeffrey N. Keller. The network helps show where Jeffrey N. Keller may publish in the future.
Co-authorship network of co-authors of Jeffrey N. Keller
This figure shows the co-authorship network connecting the top 25 collaborators of Jeffrey N. Keller. A scholar is included among the top collaborators of Jeffrey N. Keller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeffrey N. Keller. Jeffrey N. Keller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 1 | |
| 3 | 4 | |
| 4 | 8 | |
| 5 | 16 | |
| 6 | 15 | |
| 7 | 47 | |
| 8 | 83 | |
| 9 | 29 | |
| 10 | 44 | |
| 11 | 11 | |
| 12 | 10 | |
| 13 | 84 | |
| 14 | 117 | |
| 15 | 309 | |
| 16 | 103 | |
| 17 | 51 | |
| 18 | 11 | |
| 19 | 52 | |
| 20 | 155 |
About Jeffrey N. Keller
Jeffrey N. Keller is a scholar working on Neurology, Physiology and Physical Therapy, Sports Therapy and Rehabilitation, having authored 226 papers that have together received 17.3k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (55 papers), Ubiquitin and proteasome pathways (40 papers) and Endoplasmic Reticulum Stress and Disease (31 papers). The work is most often cited by research in Biological Psychiatry (882 citations), Neurology (2.7k citations) and Physiology (6.6k citations). Jeffrey N. Keller has collaborated with scholars based in United States, Italy and Argentina. Frequent co-authors include William R. Markesbery, Annadora J. Bruce‐Keller, Mark P. Mattson, Qunxing Ding, Keith B. Hanni, Edgardo Dimayuga, Sunita Gupta, D. Allan Butterfield, Le Zhang and Stephen W. Scheff. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.