James G. Alb

1.7k total citations
19 papers, 1.4k citations indexed

About

James G. Alb is a scholar working on Molecular Biology, Cell Biology and Surgery. According to data from OpenAlex, James G. Alb has authored 19 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 14 papers in Cell Biology and 7 papers in Surgery. Recurrent topics in James G. Alb's work include Cellular transport and secretion (10 papers), Pancreatic function and diabetes (7 papers) and Endoplasmic Reticulum Stress and Disease (5 papers). James G. Alb is often cited by papers focused on Cellular transport and secretion (10 papers), Pancreatic function and diabetes (7 papers) and Endoplasmic Reticulum Stress and Disease (5 papers). James G. Alb collaborates with scholars based in United States, United Kingdom and Italy. James G. Alb's co-authors include Vytas A. Bankaitis, Scott E. Phillips, Brian G. Kearns, Jaclyn R. Stonebraker, Terry Magnuson, Elizabeth Morin‐Kensicki, Sharon L. Milgram, Wanda K. O’Neal, Michael Howell and H B Skinner and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

James G. Alb

19 papers receiving 1.4k citations

Peers

James G. Alb
Igor V. Boronenkov United States
Gerry T. Snoek Netherlands
Vicki A. Sciorra United States
Emer Cunningham United Kingdom
Joanne Buxton United States
Brian G. Kearns United States
Amanda Carozzi Australia
Igor V. Boronenkov United States
James G. Alb
Citations per year, relative to James G. Alb James G. Alb (= 1×) peers Igor V. Boronenkov

Countries citing papers authored by James G. Alb

Since Specialization
Citations

This map shows the geographic impact of James G. Alb's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Alb with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Alb more than expected).

Fields of papers citing papers by James G. Alb

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Alb. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Alb. The network helps show where James G. Alb may publish in the future.

Co-authorship network of co-authors of James G. Alb

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Alb. A scholar is included among the top collaborators of James G. Alb based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Alb. James G. Alb is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Tribble, Emily K., Pavlina T. Ivanova, James G. Alb, et al.. (2016). Quantitative profiling of the endonuclear glycerophospholipidome of murine embryonic fibroblasts. Journal of Lipid Research. 57(8). 1492–1506. 13 indexed citations
2.
King, Samantha J., Sreeja B. Asokan, Elizabeth M. Haynes, et al.. (2016). Lamellipodia are crucial for haptotactic sensing and response. Journal of Cell Science. 129(12). 2329–2342. 58 indexed citations
3.
Sorrentino, Jessica A., Janakiraman Krishnamurthy, Stephen L. Tilley, et al.. (2013). p16INK4a reporter mice reveal age-promoting effects of environmental toxicants. Journal of Clinical Investigation. 124(1). 169–173. 66 indexed citations
4.
Ile, Kristina E., Sean C. Kassen, Canhong Cao, et al.. (2010). Zebrafish Class 1 Phosphatidylinositol Transfer Proteins: PITPβ and Double Cone Cell Outer Segment Integrity in Retina. Traffic. 11(9). 1151–1167. 48 indexed citations
5.
Alb, James G., et al.. (2007). The pathologies associated with functional titration of phosphatidylinositol transfer protein α activity in mice. Journal of Lipid Research. 48(8). 1857–1872. 29 indexed citations
6.
Morin‐Kensicki, Elizabeth, Michael Howell, Jaclyn R. Stonebraker, et al.. (2005). Defects in Yolk Sac Vasculogenesis, Chorioallantoic Fusion, and Embryonic Axis Elongation in Mice with Targeted Disruption of Yap65. Molecular and Cellular Biology. 26(1). 77–87. 330 indexed citations
7.
Bankaitis, Vytas A., Jorge D. Cortese, Scott E. Phillips, & James G. Alb. (2004). Phosphatidylinositol transfer protein function in the mouse. Advances in Enzyme Regulation. 44(1). 201–218. 4 indexed citations
8.
Hunt, Alan N., James G. Alb, Grielof Koster, Anthony D. Postle, & Vytas A. Bankaitis. (2004). Use of mass spectrometry-based lipidomics to probe PITPα (phosphatidylinositol transfer protein α) function inside the nuclei of PITPα+/+ and PITPα−/− cells. Biochemical Society Transactions. 32(6). 1063–1065. 11 indexed citations
9.
Alb, James G., Jorge D. Cortese, Scott E. Phillips, et al.. (2003). Mice Lacking Phosphatidylinositol Transfer Protein-α Exhibit Spinocerebellar Degeneration, Intestinal and Hepatic Steatosis, and Hypoglycemia. Journal of Biological Chemistry. 278(35). 33501–33518. 96 indexed citations
10.
Alb, James G., Scott E. Phillips, Xiaoxia Cui, et al.. (2002). Genetic Ablation of Phosphatidylinositol Transfer Protein Function in Murine Embryonic Stem Cells. Molecular Biology of the Cell. 13(3). 739–754. 67 indexed citations
11.
Nemoto, Yasuo, Brian G. Kearns, Markus R. Wenk, et al.. (2000). Functional Characterization of a Mammalian Sac1 and Mutants Exhibiting Substrate-specific Defects in Phosphoinositide Phosphatase Activity. Journal of Biological Chemistry. 275(44). 34293–34305. 106 indexed citations
12.
Phillips, Scott E., Bingdong Sha, Zhigang Xie, et al.. (1999). Yeast Sec14p Deficient in Phosphatidylinositol Transfer Activity Is Functional In Vivo. Molecular Cell. 4(2). 187–197. 125 indexed citations
13.
Jones, Steven M., James G. Alb, Scott E. Phillips, Vytas A. Bankaitis, & Kathryn E. Howell. (1998). A Phosphatidylinositol 3-Kinase and Phosphatidylinositol Transfer Protein Act Synergistically in Formation of Constitutive Transport Vesicles from the Trans-Golgi Network. Journal of Biological Chemistry. 273(17). 10349–10354. 79 indexed citations
14.
Kearns, Brian G., James G. Alb, & Vytas A. Bankaitis. (1998). Phosphatidylinositol transfer proteins: the long and winding road to physiological function. Trends in Cell Biology. 8(7). 276–282. 76 indexed citations
16.
Alb, James G., et al.. (1996). Phospholipid metabolism and membrane dynamics. Current Opinion in Cell Biology. 8(4). 534–541. 44 indexed citations
17.
Alb, James G., et al.. (1996). Phospholipid metabolism and membrane dynamics. Current Opinion in Cell Biology. 8(6). 890–890. 2 indexed citations
18.
Alb, James G., Alma Gedvilaitė, Robert T. Cartee, H B Skinner, & Vytas A. Bankaitis. (1995). Mutant rat phosphatidylinositol/phosphatidylcholine transfer proteins specifically defective in phosphatidylinositol transfer: implications for the regulation of phospholipid transfer activity.. Proceedings of the National Academy of Sciences. 92(19). 8826–8830. 53 indexed citations
19.
Skinner, H B, et al.. (1993). Phospholipid transfer activity is relevant to but not sufficient for the essential function of the yeast SEC14 gene product.. The EMBO Journal. 12(12). 4775–4784. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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