Benjamin D. Philpot

10.1k total citations · 1 hit paper
107 papers, 7.1k citations indexed

About

Benjamin D. Philpot is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Benjamin D. Philpot has authored 107 papers receiving a total of 7.1k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Molecular Biology, 49 papers in Genetics and 47 papers in Cellular and Molecular Neuroscience. Recurrent topics in Benjamin D. Philpot's work include Genetic Syndromes and Imprinting (37 papers), Neuroscience and Neuropharmacology Research (37 papers) and Epigenetics and DNA Methylation (26 papers). Benjamin D. Philpot is often cited by papers focused on Genetic Syndromes and Imprinting (37 papers), Neuroscience and Neuropharmacology Research (37 papers) and Epigenetics and DNA Methylation (26 papers). Benjamin D. Philpot collaborates with scholars based in United States, Netherlands and Spain. Benjamin D. Philpot's co-authors include Mark F. Bear, Koji Yashiro, Angela M. Mabb, Mark J. Zylka, Matthew C. Judson, Elizabeth M. Quinlan, Richard L. Huganir, Rebekah Corlew, Ken D. McCarthy and Kristen B. Casper and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Benjamin D. Philpot

96 papers receiving 7.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Regulation of NMDA receptor subunit expression and its implications for LTD, LTP, and metaplasticity

2008 510 citations Koji Yashiro, Benjamin D. Philpot profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Benjamin D. Philpot United States	43	3.8k	3.8k	2.1k	2.1k	660	107	7.1k
Anne E. West United States	32	2.6k 0.7×	4.5k 1.2×	1.1k 0.5×	1.9k 0.9×	886 1.3×	62	7.2k
Gavin Rumbaugh United States	40	3.4k 0.9×	3.6k 1.0×	1.5k 0.7×	1.3k 0.6×	477 0.7×	71	6.4k
Lucas Pozzo‐Miller United States	45	4.6k 1.2×	2.6k 0.7×	1.8k 0.8×	1.7k 0.8×	2.1k 3.2×	85	7.6k
Joseph J. LoTurco United States	41	2.5k 0.6×	2.9k 0.8×	1.0k 0.5×	1.1k 0.5×	1.7k 2.6×	91	6.3k
Yingxi Lin United States	23	2.9k 0.8×	4.3k 1.1×	1.9k 0.9×	1.6k 0.8×	520 0.8×	33	8.4k
Craig M. Powell United States	37	2.5k 0.6×	2.9k 0.8×	2.5k 1.2×	2.4k 1.2×	915 1.4×	75	6.6k
Claudio Rivera Finland	40	6.2k 1.6×	4.3k 1.2×	1.6k 0.7×	537 0.3×	1000 1.5×	85	8.6k
Gaël Malleret France	25	3.1k 0.8×	2.3k 0.6×	1.8k 0.8×	779 0.4×	1.1k 1.6×	45	5.5k
Courtney A. Miller United States	32	2.0k 0.5×	3.5k 0.9×	1.4k 0.7×	1.9k 0.9×	507 0.8×	70	5.8k
Jean‐Claude Lacaille Canada	53	6.0k 1.6×	4.0k 1.1×	3.5k 1.6×	909 0.4×	665 1.0×	140	8.7k

Countries citing papers authored by Benjamin D. Philpot

Since Specialization

Citations

This map shows the geographic impact of Benjamin D. Philpot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin D. Philpot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin D. Philpot more than expected).

Fields of papers citing papers by Benjamin D. Philpot

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin D. Philpot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin D. Philpot. The network helps show where Benjamin D. Philpot may publish in the future.

Co-authorship network of co-authors of Benjamin D. Philpot

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin D. Philpot. A scholar is included among the top collaborators of Benjamin D. Philpot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin D. Philpot. Benjamin D. Philpot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Styner, Martin, Li‐Ming Hsu, Yen‐Yu Ian Shih, et al.. (2024). Comparative profiling of white matter development in the human and mouse brain reveals volumetric deficits and delayed myelination in Angelman syndrome. Molecular Autism. 15(1). 54–54. 1 indexed citations

Judson, Matthew C., et al.. (2024). Neuronal UBE3A substrates hold therapeutic potential for Angelman syndrome. Current Opinion in Neurobiology. 88. 102899–102899. 5 indexed citations

Bettadapur, Kiran R., Justin Cotney, Jon L. Collins, et al.. (2024). Ube3a unsilencer for the potential treatment of Angelman syndrome. Nature Communications. 15(1). 5558–5558. 9 indexed citations

Judson, Matthew C., Michael S. Sidorov, Brittany Williams, et al.. (2022). Molecular and behavioral consequences of Ube3a gene overdosage in mice. JCI Insight. 7(18). 15 indexed citations

Kim, Hyojin, et al.. (2022). Rescue of behavioral and electrophysiological phenotypes in a Pitt-Hopkins syndrome mouse model by genetic restoration of Tcf4 expression. eLife. 11. 14 indexed citations

Philpot, Benjamin D., et al.. (2021). The Impact of SST and PV Interneurons on Nonlinear Synaptic Integration in the Neocortex. eNeuro. 8(5). ENEURO.0235–21.2021. 6 indexed citations

Sidorov, Michael S., Hyojin Kim, Marie Rougié, et al.. (2020). Visual Sequences Drive Experience-Dependent Plasticity in Mouse Anterior Cingulate Cortex. Cell Reports. 32(11). 108152–108152. 16 indexed citations

Wallace, Michael L., Geeske M. van Woerden, Ype Elgersma, Spencer L. Smith, & Benjamin D. Philpot. (2017). Ube3a loss increases excitability and blunts orientation tuning in the visual cortex of Angelman syndrome model mice. Journal of Neurophysiology. 118(1). 634–646. 21 indexed citations

Larsen, Rylan S., Rebecca L. Bigler, Shawn B. Frost, et al.. (2017). Distal axotomy enhances retrograde presynaptic excitability onto injured pyramidal neurons via trans-synaptic signaling. Nature Communications. 8(1). 625–625. 38 indexed citations

10.

Thaxton, Courtney, et al.. (2017). Common Pathophysiology in Multiple Mouse Models of Pitt–Hopkins Syndrome. Journal of Neuroscience. 38(4). 918–936. 35 indexed citations

11.

Judson, Matthew C., A Burette, Courtney Thaxton, et al.. (2017). Decreased Axon Caliber Underlies Loss of Fiber Tract Integrity, Disproportional Reductions in White Matter Volume, and Microcephaly in Angelman Syndrome Model Mice. Journal of Neuroscience. 37(31). 7347–7361. 29 indexed citations

12.

Berrios, Janet, Alice M. Stamatakis, Pranish A. Kantak, et al.. (2016). Loss of UBE3A from TH-expressing neurons suppresses GABA co-release and enhances VTA-NAc optical self-stimulation. Nature Communications. 7(1). 10702–10702. 52 indexed citations

13.

Bruinsma, Caroline F., Martijn Schonewille, Zhenyu Gao, et al.. (2015). Dissociation of locomotor and cerebellar deficits in a murine Angelman syndrome model. Journal of Clinical Investigation. 125(11). 4305–4315. 39 indexed citations

14.

Buttermore, Elizabeth D., Claire Piochon, Michael L. Wallace, et al.. (2012). Pinceau Organization in the Cerebellum Requires Distinct Functions of Neurofascin in Purkinje and Basket Neurons during Postnatal Development. Journal of Neuroscience. 32(14). 4724–4742. 46 indexed citations

15.

Wallace, Michael L., A Burette, Richard J. Weinberg, & Benjamin D. Philpot. (2012). Maternal Loss of Ube3a Produces an Excitatory/Inhibitory Imbalance through Neuron Type-Specific Synaptic Defects. Neuron. 75(3). 542–542. 4 indexed citations

16.

Huang, Hsien‐Sung, John A. Allen, Angela M. Mabb, et al.. (2011). Topoisomerase inhibitors unsilence the dormant allele of Ube3a in neurons. Nature. 481(7380). 185–189. 287 indexed citations

17.

Roberts, Adam C., Javier Díez‐García, Ramona M. Rodriguiz, et al.. (2009). Downregulation of NR3A-Containing NMDARs Is Required for Synapse Maturation and Memory Consolidation. Neuron. 63(3). 342–356. 121 indexed citations

18.

Henson, Maile A., Adam C. Roberts, Kayvon Salimi, et al.. (2008). Developmental Regulation of the NMDA Receptor Subunits, NR3A and NR1, in Human Prefrontal Cortex. Cerebral Cortex. 18(11). 2560–2573. 86 indexed citations

19.

Corlew, Rebekah, Yun Wang, Haben Ghermazien, Alev Erişir, & Benjamin D. Philpot. (2007). Developmental Switch in the Contribution of Presynaptic and Postsynaptic NMDA Receptors to Long-Term Depression. Journal of Neuroscience. 27(37). 9835–9845. 149 indexed citations

20.

Yashiro, Koji, Rebekah Corlew, & Benjamin D. Philpot. (2005). Visual Deprivation Modifies Both Presynaptic Glutamate Release and the Composition of Perisynaptic/Extrasynaptic NMDA Receptors in Adult Visual Cortex. Journal of Neuroscience. 25(50). 11684–11692. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact