Jaana Tolvanen

973 total citations · 1 hit paper
7 papers, 685 citations indexed

About

Jaana Tolvanen is a scholar working on Obstetrics and Gynecology, Reproductive Medicine and Molecular Biology. According to data from OpenAlex, Jaana Tolvanen has authored 7 papers receiving a total of 685 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Obstetrics and Gynecology, 5 papers in Reproductive Medicine and 1 paper in Molecular Biology. Recurrent topics in Jaana Tolvanen's work include Uterine Myomas and Treatments (6 papers), Endometriosis Research and Treatment (5 papers) and Gynecological conditions and treatments (2 papers). Jaana Tolvanen is often cited by papers focused on Uterine Myomas and Treatments (6 papers), Endometriosis Research and Treatment (5 papers) and Gynecological conditions and treatments (2 papers). Jaana Tolvanen collaborates with scholars based in Finland, Sweden and United States. Jaana Tolvanen's co-authors include Lauri A. Aaltonen, Pia Vahteristo, Jari Sjöberg, Netta Mäkinen, Miika Mehine, Eevi Kaasinen, Tom Böhling, Massimiliano Gentile, Elina Virolainen and Riku Katainen and has published in prestigious journals such as Science, Journal of Biological Chemistry and Scientific Reports.

In The Last Decade

Jaana Tolvanen

7 papers receiving 677 citations

Hit Papers

MED12 , the Mediator Complex Subunit 12 Gene, Is Mutated ... 2011 2026 2016 2021 2011 100 200 300 400

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jaana Tolvanen Finland 7 487 421 151 81 65 7 685
Hanna-Riikka Heinonen Finland 12 784 1.6× 672 1.6× 143 0.9× 129 1.6× 83 1.3× 14 991
Elina Virolainen Finland 4 346 0.7× 305 0.7× 121 0.8× 51 0.6× 60 0.9× 5 528
Heikki Ristolainen Finland 4 190 0.4× 166 0.4× 133 0.9× 54 0.7× 99 1.5× 6 396
Ayaka Orii Japan 9 240 0.5× 156 0.4× 99 0.7× 118 1.5× 42 0.6× 9 414
Joana Sederias Canada 6 193 0.4× 136 0.3× 211 1.4× 105 1.3× 80 1.2× 9 433
Helena Lagarda Spain 7 240 0.5× 185 0.4× 162 1.1× 47 0.6× 117 1.8× 8 460
Alison D. Clark United States 5 114 0.2× 105 0.2× 196 1.3× 34 0.4× 38 0.6× 5 370
Ahmed El‐Balat Germany 10 81 0.2× 166 0.4× 118 0.8× 72 0.9× 28 0.4× 34 426
Keizo Tokumo Japan 8 105 0.2× 106 0.3× 209 1.4× 40 0.5× 21 0.3× 12 369

Countries citing papers authored by Jaana Tolvanen

Since Specialization
Citations

This map shows the geographic impact of Jaana Tolvanen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jaana Tolvanen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jaana Tolvanen more than expected).

Fields of papers citing papers by Jaana Tolvanen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jaana Tolvanen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jaana Tolvanen. The network helps show where Jaana Tolvanen may publish in the future.

Co-authorship network of co-authors of Jaana Tolvanen

This figure shows the co-authorship network connecting the top 25 collaborators of Jaana Tolvanen. A scholar is included among the top collaborators of Jaana Tolvanen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jaana Tolvanen. Jaana Tolvanen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Shen, Hailian, Jason M. Spaeth, Jaana Tolvanen, et al.. (2018). Oncogenic exon 2 mutations in Mediator subunit MED12 disrupt allosteric activation of cyclin C-CDK8/19. Journal of Biological Chemistry. 293(13). 4870–4882. 48 indexed citations
2.
Välimäki, Niko, Annukka Pasanen, Oskari Heikinheimo, et al.. (2018). Genetic predisposition to uterine leiomyoma is determined by loci for genitourinary development and genome stability. eLife. 7. 53 indexed citations
3.
Heinonen, Hanna-Riikka, Annukka Pasanen, Oskari Heikinheimo, et al.. (2017). Multiple clinical characteristics separate MED12-mutation-positive and -negative uterine leiomyomas. Scientific Reports. 7(1). 1015–1015. 43 indexed citations
4.
Kämpjärvi, Kati, Netta Mäkinen, Miika Mehine, et al.. (2016). MED12 mutations and FH inactivation are mutually exclusive in uterine leiomyomas. British Journal of Cancer. 114(12). 1405–1411. 36 indexed citations
5.
Tolvanen, Jaana, Outi Uimari, Markku Ryynänen, Lauri A. Aaltonen, & Pia Vahteristo. (2012). Strong family history of uterine leiomyomatosis warrants fumarate hydratase mutation screening. Human Reproduction. 27(6). 1865–1869. 22 indexed citations
6.
Mäkinen, Netta, Miika Mehine, Jaana Tolvanen, et al.. (2011). MED12 , the Mediator Complex Subunit 12 Gene, Is Mutated at High Frequency in Uterine Leiomyomas. Science. 334(6053). 252–255. 470 indexed citations breakdown →
7.
Niittymäki, Iina, Alexandra E. Gylfe, Marko Laakso, et al.. (2010). High frequency of TTK mutations in microsatellite-unstable colorectal cancer and evaluation of their effect on spindle assembly checkpoint. Carcinogenesis. 32(3). 305–311. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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