J William L Brown
- Physiology top 10%
- Pathology and Forensic Medicine top 10%
- Endocrine and Autonomic Systems top 5%
- Pulmonary and Respiratory Medicine
- Neurology
- Co-authors
- Alagaratnam NirubanPhyo Kyaw MyintYoon K. LokeChun Shing KwokAlasdair ColesDeclan ChardMatteo PardiniNicholas Lawn
- Topics
- Multiple Sclerosis Research Studies (17 papers)Retinoids in leukemia and cellular processes (5 papers)Peripheral Neuropathies and Disorders (4 papers)
- Partner nations
- United KingdomAustraliaItaly
In The Last Decade
J William L Brown
30 papers receiving 658 citations
Peers
Comparison fields: 5 of 82
- Physiology 242
- Pathology and Forensic Medicine 180
- Endocrine and Autonomic Systems 166
- Pulmonary and Respiratory Medicine 138
- Neurology 78
Countries citing papers authored by J William L Brown
This map shows the geographic impact of J William L Brown's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J William L Brown with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J William L Brown more than expected).
Fields of papers citing papers by J William L Brown
This network shows the impact of papers produced by J William L Brown. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J William L Brown. The network helps show where J William L Brown may publish in the future.
Co-authorship network of co-authors of J William L Brown
This figure shows the co-authorship network connecting the top 25 collaborators of J William L Brown. A scholar is included among the top collaborators of J William L Brown based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J William L Brown. J William L Brown is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 0 | |
| 5 | 3 | |
| 6 | 4 | |
| 7 | 14 | |
| 8 | 4 | |
| 9 | 16 | |
| 10 | 9 | |
| 11 | 15 | |
| 12 | 59 | |
| 13 | 4 | |
| 14 | 28 | |
| 15 | 26 | |
| 16 | 25 | |
| 17 | 10 | |
| 18 | 2 | |
| 19 | 20 | |
| 20 | 2 |
About J William L Brown
J William L Brown is a scholar working on Pathology and Forensic Medicine, Endocrine and Autonomic Systems and Neurology, having authored 35 papers that have together received 676 indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (17 papers), Retinoids in leukemia and cellular processes (5 papers) and Peripheral Neuropathies and Disorders (4 papers). The work is most often cited by research in Endocrine and Autonomic Systems (166 citations), Developmental Neuroscience (42 citations) and Pathology and Forensic Medicine (180 citations). J William L Brown has collaborated with scholars based in United Kingdom, Australia and Italy. Frequent co-authors include Alagaratnam Niruban, Phyo Kyaw Myint, Yoon K. Loke, Chun Shing Kwok, Alasdair Coles, Declan Chard, Matteo Pardini, Nicholas Lawn, Richard Reynolds and Roberta Magliozzi. Their work appears in journals such as The Lancet, American Journal of Psychiatry and Brain.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.