J. David Brook
- Cellular and Molecular Neuroscience top 0.5%
- Genetic Neurodegenerative Diseases 41
- Genetics top 0.5%
- Genomic variations and chromosomal abnormalities 9
- Molecular Biology top 1%
- Mitochondrial Function and Pathology 21
- Congenital heart defects research 19
- Ubiquitin and proteasome pathways 8
- Neurology top 2%
- Parkinson's Disease Mechanisms and Treatments 8
- Reproductive Medicine top 2%
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- Cardiomyopathy and Myosin Studies 12
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- Chromosomal and Genetic Variations 9
- Co-authors
- David E. HousmanDuncan J. ShawI D YoungSilvana GuioliJamie W. FosterM. Dominguez-SteglichSahar MansourAlan Buckler
- Partner nations
- United KingdomUnited StatesFrance
In The Last Decade
J. David Brook
104 papers receiving 7.2k citations
Hit Papers
Peers
Comparison fields: 5 of 131
- Cellular and Molecular Neuroscience 2.1k
- Genetics 2.7k
- Molecular Biology 5.6k
- Neurology 749
- Reproductive Medicine 343
Countries citing papers authored by J. David Brook
This map shows the geographic impact of J. David Brook's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. David Brook with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. David Brook more than expected).
Fields of papers citing papers by J. David Brook
This network shows the impact of papers produced by J. David Brook. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. David Brook. The network helps show where J. David Brook may publish in the future.
Co-authorship network
The 25 scholars most cited alongside J. David Brook, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 1 | |
| 2 | 2018 | 27 | |
| 3 | 2017 | 39 | |
| 4 | 2016 | 14 | |
| 5 | 2013 | 11 | |
| 6 | 2011 | 71 | |
| 7 | 2008 | 59 | |
| 8 | 2007 | 73 | |
| 9 | 2006 | 33 | |
| 10 | 2005 | 195 | |
| 11 | High levels of allele loss at the FHIT and ATM genes in non-comedo ductal carcinoma in situ and grade I tubular invasive breast cancers. | 1996 | 63 |
| 12 | 1993 | 41 | |
| 13 | Expansion of an unstable DNA region and phenotypic variation in myotonic dystrophybreakdown → | 1992 | 587 |
| 14 | 1992 | 143 | |
| 15 | 1992 | 7 | |
| 16 | 1992 | 8 | |
| 17 | 1991 | 23 | |
| 18 | 1991 | 12 | |
| 19 | 1990 | 5 | |
| 20 | 1986 | 26 |
About J. David Brook
J. David Brook is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Genetics, Neurology and Cardiology and Cardiovascular Medicine, having authored 105 papers that have together received 7.3k indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (41 papers), Mitochondrial Function and Pathology (21 papers), Congenital heart defects research (19 papers), Cardiomyopathy and Myosin Studies (12 papers), Genomic variations and chromosomal abnormalities (9 papers), Chromosomal and Genetic Variations (9 papers), Parkinson's Disease Mechanisms and Treatments (8 papers) and Ubiquitin and proteasome pathways (8 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (2.1k citations), Genetics (2.7k citations), Molecular Biology (5.6k citations), Neurology (749 citations) and Reproductive Medicine (343 citations). J. David Brook has collaborated with scholars based in United Kingdom, United States and France. Frequent co-authors include David E. Housman, Duncan J. Shaw, I D Young, Silvana Guioli, Jamie W. Foster, M. Dominguez-Steglich, Sahar Mansour, Alan Buckler, Cheni Kwok and William Reardon. Their work appears in journals such as Human Genetics, Genomics, Human Molecular Genetics, Nucleic Acids Research and Journal of Medical Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.