Richard P. Harvey

29.5k total citations · 5 hit papers
240 papers, 18.6k citations indexed

About

Richard P. Harvey is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Richard P. Harvey has authored 240 papers receiving a total of 18.6k indexed citations (citations by other indexed papers that have themselves been cited), including 186 papers in Molecular Biology, 49 papers in Cardiology and Cardiovascular Medicine and 43 papers in Surgery. Recurrent topics in Richard P. Harvey's work include Congenital heart defects research (110 papers), Congenital Heart Disease Studies (28 papers) and Cardiac Fibrosis and Remodeling (28 papers). Richard P. Harvey is often cited by papers focused on Congenital heart defects research (110 papers), Congenital Heart Disease Studies (28 papers) and Cardiac Fibrosis and Remodeling (28 papers). Richard P. Harvey collaborates with scholars based in Australia, United States and United Kingdom. Richard P. Harvey's co-authors include Christine Biben, Lynne Hartley, Ian Lyons, R Li, Lorraine Robb, Thierry Lints, Douglas A. Melton, Linda M. Parsons, Owen W.J. Prall and David A. Elliott and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Richard P. Harvey

238 papers receiving 18.3k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Myogenic and morphogenetic defects in the heart tubes of murine embryos lacking the homeo box gene Nkx2-5.

1995 894 citations Lynne Hartley, Lorraine Robb et al. profile →
Disrupted cardiac development but normal hematopoiesis in mice deficient in the second CXCL12/SDF-1 receptor, CXCR7

2007 501 citations Christine Biben, Richard P. Harvey et al. profile →
ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation

2015 497 citations Gabriele D’Uva, Alla Aharonov et al. Nature Cell Biology profile →
Endothelial to Mesenchymal Transition in Cardiovascular Disease

2019 409 citations Richard P. Harvey et al. Journal of the American College of Cardiology profile →
Single-cell expression profiling reveals dynamic flux of cardiac stromal, vascular and immune cells in health and injury

2019 370 citations Ralph Patrick, Vaibhao Janbandhu et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Richard P. Harvey Australia	76	13.8k	3.4k	3.0k	2.6k	2.6k	240	18.6k
Thomas Braun Germany	82	21.9k 1.6×	3.1k 0.9×	4.1k 1.4×	3.1k 1.2×	2.0k 0.8×	483	31.1k
James F. Martin United States	80	15.8k 1.1×	3.1k 0.9×	3.1k 1.1×	3.5k 1.3×	1.5k 0.6×	267	21.8k
John M. Shelton United States	75	15.7k 1.1×	3.3k 1.0×	2.2k 0.7×	2.8k 1.1×	2.5k 1.0×	152	22.8k
Nadia Rosenthal United States	74	13.5k 1.0×	3.7k 1.1×	3.5k 1.2×	2.1k 0.8×	1.0k 0.4×	239	20.7k
Jonathan A. Epstein United States	92	17.1k 1.2×	2.1k 0.6×	3.7k 1.2×	3.1k 1.2×	2.2k 0.8×	263	23.9k
Michael Schneider United States	79	15.0k 1.1×	5.8k 1.7×	4.0k 1.4×	1.4k 0.5×	3.5k 1.4×	233	22.5k
Min Lü United States	81	13.2k 1.0×	1.7k 0.5×	4.2k 1.4×	2.6k 1.0×	2.9k 1.1×	227	21.4k
William T. Pu United States	77	13.7k 1.0×	4.5k 1.3×	4.2k 1.4×	1.8k 0.7×	2.0k 0.8×	203	18.5k
Thomas Doetschman United States	63	13.8k 1.0×	2.9k 0.9×	2.4k 0.8×	3.3k 1.3×	709 0.3×	134	19.5k
Ju Chen United States	72	13.3k 1.0×	6.5k 1.9×	2.3k 0.8×	1.2k 0.4×	1.5k 0.6×	270	19.6k

Countries citing papers authored by Richard P. Harvey

Since Specialization

Citations

This map shows the geographic impact of Richard P. Harvey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard P. Harvey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard P. Harvey more than expected).

Fields of papers citing papers by Richard P. Harvey

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard P. Harvey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard P. Harvey. The network helps show where Richard P. Harvey may publish in the future.

Co-authorship network of co-authors of Richard P. Harvey

This figure shows the co-authorship network connecting the top 25 collaborators of Richard P. Harvey. A scholar is included among the top collaborators of Richard P. Harvey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard P. Harvey. Richard P. Harvey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kesteven, Scott, Delfine Cheng, Vaibhao Janbandhu, et al.. (2025). BPS2025 - Piezo1 channel-mediated mechanotransduction as a driver of cardiac fibrosis. Biophysical Journal. 124(3). 269a–269a. 1 indexed citations

Patrick, Ralph, Vaibhao Janbandhu, Vikram J. Tallapragada, et al.. (2024). Integration mapping of cardiac fibroblast single-cell transcriptomes elucidates cellular principles of fibrosis in diverse pathologies. Science Advances. 10(25). eadk8501–eadk8501. 22 indexed citations

Davies, Elizabeth M., Rajendra Gurung, Richard P. Harvey, et al.. (2023). PI(4,5)P ₂ -dependent regulation of endothelial tip cell specification contributes to angiogenesis. Science Advances. 9(13). eadd6911–eadd6911. 10 indexed citations

Cheng, Delfine, Navid Bavi, Genevieve A. Secker, et al.. (2023). MyoD-family inhibitor proteins act as auxiliary subunits of Piezo channels. Science. 381(6659). 799–804. 52 indexed citations

Moreau, Julie, Scott Kesteven, Ella MMA Martin, et al.. (2019). Gene-environment interaction impacts on heart development and embryo survival. Development. 146(4). 49 indexed citations

Wang, Yidong, Bingruo Wu, Pengfei Lu, et al.. (2017). Uncontrolled angiogenic precursor expansion causes coronary artery anomalies in mice lacking Pofut1. Nature Communications. 8(1). 578–578. 30 indexed citations

D’Uva, Gabriele, Alla Aharonov, Mattia Lauriola, et al.. (2015). ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation. Nature Cell Biology. 17(5). 627–638. 497 indexed citations breakdown →

Zhou, Qifeng, Scott Kesteven, Jianxin Wu, et al.. (2015). Pressure Overload by Transverse Aortic Constriction Induces Maladaptive Hypertrophy in a Titin-Truncated Mouse Model. BioMed Research International. 2015. 1–6. 16 indexed citations

Cannon, L. Edward, Ze‐Yan Yu, Tadeusz Marciniec, et al.. (2015). Irreversible Triggers for Hypertrophic Cardiomyopathy Are Established in the Early Postnatal Period. Journal of the American College of Cardiology. 65(6). 560–569. 24 indexed citations

10.

Lee, Lawrence K., Katherine J. Bryant, Romaric Bouveret, et al.. (2013). Selective Inhibition of Human Group IIA-secreted Phospholipase A2 (hGIIA) Signaling Reveals Arachidonic Acid Metabolism Is Associated with Colocalization of hGIIA to Vimentin in Rheumatoid Synoviocytes. Journal of Biological Chemistry. 288(21). 15269–15279. 24 indexed citations

11.

Iacovino, Michelina, Jamie L. Lohr, Claudia Zierold, et al.. (2013). Nkx2-5 Mediates Differential Cardiac Differentiation Through Interaction with Hoxa10. Stem Cells and Development. 22(15). 2211–2220. 25 indexed citations

12.

Costa, Mauro W., Stella Lee, Milena B. Furtado, et al.. (2011). Complex SUMO-1 Regulation of Cardiac Transcription Factor Nkx2-5. PLoS ONE. 6(9). e24812–e24812. 33 indexed citations

13.

Granados-Riveron, Javier T, Mark Pope, Frances Bu’Lock, et al.. (2011). Combined Mutation Screening of NKX2-5, GATA4, and TBX5 in Congenital Heart Disease: Multiple Heterozygosity and Novel Mutations. Congenital Heart Disease. 7(2). 151–159. 71 indexed citations

14.

Butler, Tanya L., Giorgia Esposito, Gillian M. Blue, et al.. (2010). GATA4 Mutations in 357 Unrelated Patients with Congenital Heart Malformation. Genetic Testing and Molecular Biomarkers. 14(6). 797–802. 48 indexed citations

15.

Artap, Stanley T, Jost I. Preis, Diane Fatkin, et al.. (2010). Loss of Cited2 causes congenital heart disease by perturbing left–right patterning of the body axis. Human Molecular Genetics. 20(6). 1097–1110. 49 indexed citations

16.

Stanley, Edouard G., Christine Biben, Andrew G. Elefanty, et al.. (2002). Efficient Cre-mediated deletion in cardiac progenitor cells conferred by a 3'UTR-ires-Cre allele of the homeobox gene Nkx2-5. The International Journal of Developmental Biology. 46(4). 431–439. 241 indexed citations

17.

Christoffels, Vincent M., Petra E.M.H. Habets, Diego Franco, et al.. (2000). Chamber Formation and Morphogenesis in the Developing Mammalian Heart. Developmental Biology. 223(2). 266–278. 390 indexed citations

18.

Harvey, Richard P.. (1999). Seeking a regulatory roadmap for heart morphogenesis. Seminars in Cell and Developmental Biology. 10(1). 99–107. 31 indexed citations

19.

Robb, Lorraine, et al.. (1998). musculin: a murine basic helix-loop-helix transcription factor gene expressed in embryonic skeletal muscle. Mechanisms of Development. 76(1-2). 197–201. 40 indexed citations

20.

Harvey, Richard P., et al.. (1978). Mouse model of pulmonary blastomycosis: utility, simplicity, and quantitative parameters.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 117(4). 695–703. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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