Itsuo Tokunaga
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Neurology top 10%
- Physiology
- Toxicology top 5%
- Co-authors
- Shin‐ichi KuboTakako GotohdaOsamu KitamuraAkiko IshigamiIchiro NakasonoToshiaki TakeichiSanae TakeichiElaine Lu Wang
- Topics
- Neuroscience and Neuropharmacology Research (7 papers)Neuroinflammation and Neurodegeneration Mechanisms (4 papers)Hormonal and reproductive studies (4 papers)
- Partner nations
- JapanUnited Kingdom
In The Last Decade
Itsuo Tokunaga
46 papers receiving 557 citations
Peers
Comparison fields: 5 of 95
- Molecular Biology 143
- Cellular and Molecular Neuroscience 142
- Neurology 68
- Physiology 54
- Toxicology 53
Countries citing papers authored by Itsuo Tokunaga
This map shows the geographic impact of Itsuo Tokunaga's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Itsuo Tokunaga with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Itsuo Tokunaga more than expected).
Fields of papers citing papers by Itsuo Tokunaga
This network shows the impact of papers produced by Itsuo Tokunaga. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Itsuo Tokunaga. The network helps show where Itsuo Tokunaga may publish in the future.
Co-authorship network of co-authors of Itsuo Tokunaga
This figure shows the co-authorship network connecting the top 25 collaborators of Itsuo Tokunaga. A scholar is included among the top collaborators of Itsuo Tokunaga based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Itsuo Tokunaga. Itsuo Tokunaga is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 9 | |
| 3 | 3 | |
| 4 | 63 | |
| 5 | 3 | |
| 6 | 5 | |
| 7 | 4 | |
| 8 | 48 | |
| 9 | 20 | |
| 10 | 9 | |
| 11 | 30 | |
| 12 | 11 | |
| 13 | 7 | |
| 14 | 17 | |
| 15 | 44 | |
| 16 | 18 | |
| 17 | 13 | |
| 18 | 3 | |
| 19 | ANCIENT HUMAN BONES WITH MERCURIC COSMETICS EXCAVATED FROM THE BURIAL MOUND OF KANZO-YAMA IN TOKUSHIMA | 1 |
| 20 | Electroencephalographical analysis of acute toluene poisoning. | 5 |
About Itsuo Tokunaga
Itsuo Tokunaga is a scholar working on Developmental Neuroscience, Toxicology and Emergency Medicine, having authored 49 papers that have together received 573 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (7 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers) and Hormonal and reproductive studies (4 papers). The work is most often cited by research in Toxicology (53 citations), Biological Psychiatry (37 citations) and Developmental Neuroscience (46 citations). Itsuo Tokunaga has collaborated with scholars based in Japan and United Kingdom. Frequent co-authors include Shin‐ichi Kubo, Takako Gotohda, Osamu Kitamura, Akiko Ishigami, Ichiro Nakasono, Toshiaki Takeichi, Sanae Takeichi, Elaine Lu Wang, Akiko Eguchi and Kazuya Ikematsu. Their work appears in journals such as Life Sciences, Forensic Science International and IUBMB Life.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.