Isaac Veinbergs

4.7k total citations · 1 hit paper
25 papers, 3.7k citations indexed

About

Isaac Veinbergs is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Isaac Veinbergs has authored 25 papers receiving a total of 3.7k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Physiology, 12 papers in Cellular and Molecular Neuroscience and 11 papers in Molecular Biology. Recurrent topics in Isaac Veinbergs's work include Alzheimer's disease research and treatments (16 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (6 papers). Isaac Veinbergs is often cited by papers focused on Alzheimer's disease research and treatments (16 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cholinesterase and Neurodegenerative Diseases (6 papers). Isaac Veinbergs collaborates with scholars based in United States, South Korea and Japan. Isaac Veinbergs's co-authors include Eliezer Masliah, Margaret Mallory, Yutaka Sagara, Edward Rockenstein, Makoto Hashimoto, Lennart Mucke, Ayako Takeda, Michael Alford, Nianfeng Ge and Allen D. Roses and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Neuroscience.

In The Last Decade

Isaac Veinbergs

25 papers receiving 3.6k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Dopaminergic Loss and Inclusion Body Formation in α-Synuclein Mice: Implications for Neurodegenerative Disorders

2000 1.4k citations Eliezer Masliah, Edward Rockenstein et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Isaac Veinbergs United States	21	1.8k	1.6k	1.6k	1.1k	696	25	3.7k
Michael Mante United States	42	2.0k 1.2×	1.8k 1.1×	1.7k 1.0×	1.1k 1.1×	1.3k 1.8×	69	4.3k
Susan Leight United States	19	1.5k 0.9×	2.2k 1.4×	953 0.6×	1.2k 1.1×	795 1.1×	21	3.9k
Heather L. Melrose United States	29	1.8k 1.0×	1.8k 1.1×	1.2k 0.8×	1.2k 1.2×	764 1.1×	38	3.7k
Reidun Torp Norway	30	902 0.5×	1.4k 0.8×	1.9k 1.2×	1.8k 1.7×	745 1.1×	56	4.0k
Anita Sidhu United States	40	2.0k 1.1×	1.0k 0.6×	1.9k 1.2×	1.4k 1.4×	381 0.5×	86	4.0k
Marie‐Christine Chartier‐Harlin France	35	2.7k 1.5×	2.8k 1.7×	1.8k 1.1×	2.3k 2.2×	938 1.3×	88	6.0k
Akihiko Iwai Japan	16	2.4k 1.4×	1.9k 1.1×	1.3k 0.8×	784 0.7×	529 0.8×	38	3.5k
Guilian Xu United States	25	921 0.5×	2.4k 1.5×	1.0k 0.6×	1.4k 1.4×	922 1.3×	49	4.2k
Victoria Gonzales United States	16	699 0.4×	2.0k 1.2×	957 0.6×	1.1k 1.0×	869 1.2×	19	3.4k
T Saitoh United States	32	1.6k 0.9×	3.1k 1.9×	1.7k 1.0×	2.1k 2.0×	697 1.0×	49	5.1k

Countries citing papers authored by Isaac Veinbergs

Since Specialization

Citations

This map shows the geographic impact of Isaac Veinbergs's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Isaac Veinbergs with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Isaac Veinbergs more than expected).

Fields of papers citing papers by Isaac Veinbergs

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Isaac Veinbergs. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Isaac Veinbergs. The network helps show where Isaac Veinbergs may publish in the future.

Co-authorship network of co-authors of Isaac Veinbergs

This figure shows the co-authorship network connecting the top 25 collaborators of Isaac Veinbergs. A scholar is included among the top collaborators of Isaac Veinbergs based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Isaac Veinbergs. Isaac Veinbergs is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Creed, Rose B., Liliana Menalled, Bradford Casey, et al.. (2019). Basal and Evoked Neurotransmitter Levels in Parkin, DJ-1, PINK1 and LRRK2 Knockout Rat Striatum. Neuroscience. 409. 169–179. 36 indexed citations

Vanover, Kimberly E., Isaac Veinbergs, & Robert E. Davis. (2008). Antipsychotic-like behavioral effects and cognitive enhancement by a potent and selective muscarinic M₁ receptor agonist, AC-260584.. Behavioral Neuroscience. 122(3). 570–575. 28 indexed citations

Gardell, Luis R., Kimberly E. Vanover, Robert W. Johnson, et al.. (2007). ACP-103, a 5-Hydroxytryptamine 2A Receptor Inverse Agonist, Improves the Antipsychotic Efficacy and Side-Effect Profile of Haloperidol and Risperidone in Experimental Models. Journal of Pharmacology and Experimental Therapeutics. 322(2). 862–870. 47 indexed citations

Vanover, Kimberly E., Scott C. Harvey, Thomas Son, et al.. (2004). Pharmacological Characterization of AC-90179 [2-(4-Methoxyphenyl)-N-(4-methyl-benzyl)-N-(1-methyl-piperidin-4-yl)-acetamide Hydrochloride]: A Selective Serotonin 2A Receptor Inverse Agonist. Journal of Pharmacology and Experimental Therapeutics. 310(3). 943–951. 18 indexed citations

Weiner, David M., Herbert Y. Meltzer, Isaac Veinbergs, et al.. (2004). The role of M1 muscarinic receptor agonism of N-desmethylclozapine in the unique clinical effects of clozapine. Psychopharmacology. 177(1-2). 207–216. 206 indexed citations

Veinbergs, Isaac, et al.. (2002). Neurotoxic effects of apolipoprotein E4 are mediated via dysregulation of calcium homeostasis. Journal of Neuroscience Research. 67(3). 379–387. 74 indexed citations

Uden, Emily Van, Margaret Mallory, Isaac Veinbergs, et al.. (2002). Increased Extracellular Amyloid Deposition and Neurodegeneration in Human Amyloid Precursor Protein Transgenic Mice Deficient in Receptor-Associated Protein. Journal of Neuroscience. 22(21). 9298–9304. 73 indexed citations

Veinbergs, Isaac, Emily Van Uden, Margaret Mallory, et al.. (2001). Role of Apolipoprotein E Receptors in Regulating the Differential in vivo Neurotrophic Effects of Apolipoprotein E. Experimental Neurology. 170(1). 15–26. 31 indexed citations

Masliah, Eliezer, Edward Rockenstein, Isaac Veinbergs, et al.. (2001). β-Amyloid peptides enhance α-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease. Proceedings of the National Academy of Sciences. 98(21). 12245–12250. 490 indexed citations

10.

Veinbergs, Isaac, Michael Mante, Margaret Mallory, & Eliezer Masliah. (2000). Neurotrophic effects of Cerebrolysin® in animal models of excitotoxicity. PubMed. 59. 273–280. 45 indexed citations

11.

Licastro, Federico, Iain L. Campbell, Carrie Kincaid, et al.. (1999). A Role for apoE in Regulating the Levels of α-1-Antichymotrypsin in the Aging Mouse Brain and in Alzheimer's Disease. American Journal Of Pathology. 155(3). 869–875. 22 indexed citations

12.

Veinbergs, Isaac, et al.. (1999). Synaptotagmin and synaptic transmission alterations in apolipoprotein E-deficient mice. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 23(3). 519–531. 28 indexed citations

13.

Veinbergs, Isaac, Margaret Mallory, Michael Mante, et al.. (1999). Differential neurotrophic effects of apolipoprotein E in aged transgenic mice. Neuroscience Letters. 265(3). 218–222. 41 indexed citations

14.

Masliah, Eliezer, et al.. (1999). Cerebrolysin Ameliorates Performance Deficits, and Neuronal Damage in Apolipoprotein E-Deficient Mice. Pharmacology Biochemistry and Behavior. 62(2). 239–245. 59 indexed citations

15.

Uden, Emily Van, Isaac Veinbergs, Margaret Mallory, Robert A. Orlando, & Eliezer Masliah. (1999). A novel role for receptor-associated protein in somatostatin modulation: implications for Alzheimer's disease. Neuroscience. 88(3). 687–700. 19 indexed citations

16.

Hsu, Leigh J., Margaret Mallory, Yu Xia, et al.. (1998). Expression Pattern of Synucleins (Non‐Aβ Component of Alzheimer's Disease Amyloid Precursor Protein/α‐Synuclein) During Murine Brain Development. Journal of Neurochemistry. 71(1). 338–344. 154 indexed citations

17.

Veinbergs, Isaac, Min Whan Jung, Steve Young, et al.. (1998). Altered long-term potentiation in the hippocampus of apolipoprotein E-deficient mice. Neuroscience Letters. 249(2-3). 71–74. 31 indexed citations

18.

Masliah, Eliezer, William Samuel, Isaac Veinbergs, et al.. (1997). Neurodegeneration and cognitive impairment in apoE-deficient mice is ameliorated by infusion of recombinant apoE. Brain Research. 751(2). 307–314. 120 indexed citations

19.

Masliah, Eliezer, et al.. (1996). ALTERATIONS IN APOLIPOPROTEIN E EXPRESSION DURING AGING AND NEURODEGENERATION. Progress in Neurobiology. 50(5-6). 493–503. 75 indexed citations

20.

Masliah, Eliezer, Margaret Mallory, Nianfeng Ge, et al.. (1995). Neurodegeneration in the Central Nervous System of apoE-Deficient Mice. Experimental Neurology. 136(2). 107–122. 327 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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