Il‐mi Okazaki

5.0k total citations · 3 hit papers
37 papers, 3.9k citations indexed

About

Il‐mi Okazaki is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Il‐mi Okazaki has authored 37 papers receiving a total of 3.9k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Immunology, 20 papers in Oncology and 11 papers in Molecular Biology. Recurrent topics in Il‐mi Okazaki's work include Immune Cell Function and Interaction (20 papers), Cancer Immunotherapy and Biomarkers (17 papers) and T-cell and B-cell Immunology (14 papers). Il‐mi Okazaki is often cited by papers focused on Immune Cell Function and Interaction (20 papers), Cancer Immunotherapy and Biomarkers (17 papers) and T-cell and B-cell Immunology (14 papers). Il‐mi Okazaki collaborates with scholars based in Japan and United States. Il‐mi Okazaki's co-authors include Tasuku Honjo, Taku Okazaki, Daisuke Sugiura, Kazuo Kinoshita, Takumi Maruhashi, Masamichi Muramatsu, Kenji Shimizu, Kiyotsugu Yoshikawa, Hitoshi Nagaoka and Hiroshi Hiai and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Il‐mi Okazaki

37 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Helicobacter pylori infection triggers aberrant expression of activation-induced cytidine deaminase in gastric epithelium

2007 380 citations Hiroyuki Marusawa, Kazuo Kinoshita et al. profile →
Constitutive Expression of AID Leads to Tumorigenesis

2003 360 citations Il‐mi Okazaki, Hiroshi Hiai et al. profile →
PD-1 and LAG-3 inhibitory co-receptors act synergistically to prevent autoimmunity in mice

2011 255 citations Taku Okazaki, Il‐mi Okazaki et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Il‐mi Okazaki Japan	27	2.3k	1.7k	1.1k	376	303	37	3.9k
Régis Costello France	33	2.0k 0.9×	1.3k 0.8×	872 0.8×	229 0.6×	384 1.3×	153	3.9k
Ryan Emerson United States	32	2.6k 1.1×	2.2k 1.3×	1.0k 0.9×	291 0.8×	172 0.6×	64	4.5k
Andrew J. Long United States	8	2.8k 1.2×	2.9k 1.7×	688 0.6×	263 0.7×	255 0.8×	11	4.6k
Arko Gorter Netherlands	40	2.2k 1.0×	1.5k 0.8×	1.4k 1.2×	429 1.1×	210 0.7×	82	4.1k
Michael Bowman United States	8	2.8k 1.2×	2.7k 1.6×	785 0.7×	270 0.7×	263 0.9×	13	4.4k
Katsuto Takenaka Japan	32	1.7k 0.7×	1.4k 0.8×	1.5k 1.3×	572 1.5×	272 0.9×	153	4.8k
Angelo A. Cardoso United States	38	2.5k 1.1×	1.2k 0.7×	2.0k 1.8×	276 0.7×	135 0.4×	86	5.3k
Cecilia Sgadari Italy	29	1.6k 0.7×	2.2k 1.3×	1.3k 1.1×	562 1.5×	149 0.5×	68	4.2k
Lydie Cassard France	25	2.5k 1.1×	2.8k 1.6×	1.5k 1.3×	329 0.9×	177 0.6×	49	4.6k
Sophie Lucas Belgium	38	4.5k 1.9×	2.0k 1.1×	2.1k 1.9×	257 0.7×	182 0.6×	71	5.8k

Countries citing papers authored by Il‐mi Okazaki

Since Specialization

Citations

This map shows the geographic impact of Il‐mi Okazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Il‐mi Okazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Il‐mi Okazaki more than expected).

Fields of papers citing papers by Il‐mi Okazaki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Il‐mi Okazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Il‐mi Okazaki. The network helps show where Il‐mi Okazaki may publish in the future.

Co-authorship network of co-authors of Il‐mi Okazaki

This figure shows the co-authorship network connecting the top 25 collaborators of Il‐mi Okazaki. A scholar is included among the top collaborators of Il‐mi Okazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Il‐mi Okazaki. Il‐mi Okazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Maruhashi, Takumi, Daisuke Sugiura, Il‐mi Okazaki, et al.. (2022). Binding of LAG-3 to stable peptide-MHC class II limits T cell function and suppresses autoimmunity and anti-cancer immunity. Immunity. 55(5). 912–924.e8. 129 indexed citations

Sugiura, Daisuke, Il‐mi Okazaki, Takumi Maruhashi, et al.. (2022). PD-1 agonism by anti-CD80 inhibits T cell activation and alleviates autoimmunity. Nature Immunology. 23(3). 399–410. 57 indexed citations

Shimizu, Kenji, Daisuke Sugiura, Il‐mi Okazaki, et al.. (2021). PD-1 preferentially inhibits the activation of low-affinity T cells. Proceedings of the National Academy of Sciences. 118(35). 14 indexed citations

Shimizu, Kenji, Daisuke Sugiura, Il‐mi Okazaki, et al.. (2020). PD-1 Imposes Qualitative Control of Cellular Transcriptomes in Response to T Cell Activation. Molecular Cell. 77(5). 937–950.e6. 43 indexed citations

Sugiura, Daisuke, Takumi Maruhashi, Il‐mi Okazaki, et al.. (2019). Restriction of PD-1 function by cis -PD-L1/CD80 interactions is required for optimal T cell responses. Science. 364(6440). 558–566. 285 indexed citations

Okazaki, Taku & Il‐mi Okazaki. (2019). Stimulatory and Inhibitory Co-signals in Autoimmunity. Advances in experimental medicine and biology. 1189. 213–232. 8 indexed citations

Okazaki, Il‐mi, et al.. (2019). PD-1 aborts the activation trajectory of autoreactive CD8+ T cells to prohibit their acquisition of effector functions. Journal of Autoimmunity. 105. 102296–102296. 12 indexed citations

Maruhashi, Takumi, et al.. (2019). Glucocorticoids potentiate the inhibitory capacity of programmed cell death 1 by up-regulating its expression on T cells. Journal of Biological Chemistry. 294(52). 19896–19906. 35 indexed citations

Sugiura, Daisuke, et al.. (2019). PD-1 Primarily Targets TCR Signal in the Inhibition of Functional T Cell Activation. Frontiers in Immunology. 10. 630–630. 123 indexed citations

10.

Sugiura, Daisuke, et al.. (2019). Atypical motifs in the cytoplasmic region of the inhibitory immune co-receptor LAG-3 inhibit T cell activation. Journal of Biological Chemistry. 294(15). 6017–6026. 79 indexed citations

11.

Maruhashi, Takumi, et al.. (2018). LAG-3 inhibits the activation of CD4+ T cells that recognize stable pMHCII through its conformation-dependent recognition of pMHCII. Nature Immunology. 19(12). 1415–1426. 202 indexed citations

12.

Nishijima, Hitoshi, Yoshiki Matsuoka, Yasuhiro Mouri, et al.. (2017). Paradoxical development of polymyositis-like autoimmunity through augmented expression of autoimmune regulator (AIRE). Journal of Autoimmunity. 86. 75–92. 14 indexed citations

13.

Wang, Jian, Il‐mi Okazaki, Taku Yoshida, et al.. (2010). PD-1 deficiency results in the development of fatal myocarditis in MRL mice. International Immunology. 22(6). 443–452. 220 indexed citations

14.

Morisawa, Toshiyuki, Hiroyuki Marusawa, Yoshihide Ueda, et al.. (2008). Organ‐specific profiles of genetic changes in cancers caused by activation‐induced cytidine deaminase expression. International Journal of Cancer. 123(12). 2735–2740. 68 indexed citations

15.

Okazaki, Il‐mi, Ai Kotani, & Tasuku Honjo. (2007). Role of AID in Tumorigenesis. Advances in immunology. 94. 245–273. 111 indexed citations

16.

Shinkura, Reiko, Il‐mi Okazaki, Taro Muto, Nasim A. Begum, & Tasuku Honjo. (2007). Regulation of AID Function In Vivo. Advances in experimental medicine and biology. 596. 71–81. 11 indexed citations

17.

Muto, Taro, Il‐mi Okazaki, Shuichi Yamada, et al.. (2006). Negative regulation of activation-induced cytidine deaminase in B cells. Proceedings of the National Academy of Sciences. 103(8). 2752–2757. 76 indexed citations

18.

Kotani, Ai, Il‐mi Okazaki, Masamichi Muramatsu, et al.. (2005). A target selection of somatic hypermutations is regulated similarly between T and B cells upon activation-induced cytidine deaminase expression. Proceedings of the National Academy of Sciences. 102(12). 4506–4511. 60 indexed citations

19.

Yoshikawa, Kiyotsugu, Il‐mi Okazaki, Kazuo Kinoshita, et al.. (2002). AID Enzyme-Induced Hypermutation in an Actively Transcribed Gene in Fibroblasts. Science. 296(5575). 2033–2036. 312 indexed citations

20.

Okazaki, Il‐mi, Kazuo Kinoshita, Masamichi Muramatsu, Kiyotsugu Yoshikawa, & Tasuku Honjo. (2002). The AID enzyme induces class switch recombination in fibroblasts. Nature. 416(6878). 340–345. 221 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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