Ilker Öztop
Impact in
- Virology top 2%
- HIV Research and Treatment
- Infectious Diseases top 10%
- HIV/AIDS drug development and treatment
Papers in
-
- Signaling Pathways in Disease 2
- Nuclear Structure and Function 2
- RNA Research and Splicing 1
- Cell death mechanisms and regulation 1
- Virology 5
- HIV Research and Treatment 5
- Co-authors
- Alan Engelman (6 shared papers)Kyeongeun Lee (3 shared papers)Vineet N. KewalRamani (3 shared papers)Lavanya Krishnan (2 shared papers)Kenneth A. Matreyek (2 shared papers)Mohd Jamal Dar (1 shared paper)Christopher Tipper (1 shared paper)Xiang Li (1 shared paper)
- Journals
- Journal of Virology (3 papers)Methods (1 paper)The Journal of Cell Biology (1 paper)Proceedings of the National Academy of Sciences (1 paper)Expert Review of Anti-infective Therapy (1 paper)
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Ilker Öztop
7 papers receiving 520 citations
Peers
Comparison fields: 5 of 44
- Virology 357
- Infectious Diseases 224
- Molecular Biology 341
- Immunology 67
- Epidemiology 81
Countries citing papers authored by Ilker Öztop
This map shows the geographic impact of Ilker Öztop's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ilker Öztop with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ilker Öztop more than expected).
Fields of papers citing papers by Ilker Öztop
This network shows the impact of papers produced by Ilker Öztop. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ilker Öztop. The network helps show where Ilker Öztop may publish in the future.
Co-authors
The 25 scholars most cited alongside Ilker Öztop, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 152 | |
| 2 | 2012 | 112 | |
| 3 | 2014 | 111 | |
| 4 | 2012 | 76 | |
| 5 | 2007 | 42 | |
| 6 | 2009 | 23 | |
| 7 | 2012 | 6 |
About Ilker Öztop
Ilker Öztop is a scholar working on Molecular Biology, Virology, Infectious Diseases, Oncology and Epidemiology, having authored 7 papers that have together received 522 indexed citations. Recurring topics across this work include HIV Research and Treatment (5 papers), HIV/AIDS drug development and treatment (3 papers), Signaling Pathways in Disease (2 papers), Nuclear Structure and Function (2 papers), Cytomegalovirus and herpesvirus research (1 paper), RNA Research and Splicing (1 paper), Cell death mechanisms and regulation (1 paper) and Cancer-related Molecular Pathways (1 paper). The work is most often cited by research in Virology (357 citations), Infectious Diseases (224 citations), Molecular Biology (341 citations), Immunology (67 citations) and Epidemiology (81 citations). Ilker Öztop has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Alan Engelman, Kyeongeun Lee, Vineet N. KewalRamani, Lavanya Krishnan, Kenneth A. Matreyek, Mohd Jamal Dar, Christopher Tipper, Xiang Li, Jiong Shi and David R. Hout. Their work appears in journals such as Journal of Virology, Methods, The Journal of Cell Biology, Proceedings of the National Academy of Sciences and Expert Review of Anti-infective Therapy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.