Iker Badiola

747 citations
36 papers · 559 · h-index 14

Impact in

Papers in

    • Circular RNAs in diseases 5
    • RNA Interference and Gene Delivery 4
    • RNA modifications and cancer 3
    • MicroRNA in disease regulation 8

Iker Badiola

34 papers receiving 551 citations

Peers

Iker Badiola
Comparison fields: 5 of 99
  • Cancer Research 131
  • Immunology and Allergy 43
  • Oncology 126
  • Molecular Biology 307
  • Food Science 62
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Ha Na Choi South Korea
Mario Alberto Ramírez-Camacho Mexico
Gina Sennello United States
Clement Penny South Africa
Margherita Cortini Italy
Shinichi Asada Japan
Yan Lin China
Cheolhee Won South Korea
Marzia Pucci Italy
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Citations per field
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Citations per year

Countries citing papers authored by Iker Badiola

Since Specialization
Citations

This map shows the geographic impact of Iker Badiola's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Iker Badiola with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Iker Badiola more than expected).

Fields of papers citing papers by Iker Badiola

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Iker Badiola. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Iker Badiola. The network helps show where Iker Badiola may publish in the future.

Co-authors

The 25 scholars most cited alongside Iker Badiola, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Iker Badiola Line = papers co-authored together Iker Badiola links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.

#Work
1 201796
2 200674
3 201160
4 201841
5 202031
6 201226
7 201822
8 201621
9 202019
10 201519
11 201717
12 202217
13 202015
14 202113
15 201813
16 202012
17 201811
18 20228
19 20217
20 20206

About Iker Badiola

Iker Badiola is a scholar working on Molecular Biology, Cancer Research, Oncology, Cell Biology and Immunology and Allergy, having authored 36 papers that have together received 559 indexed citations. Recurring topics across this work include MicroRNA in disease regulation (8 papers), Cell Adhesion Molecules Research (5 papers), Circular RNAs in diseases (5 papers), RNA Interference and Gene Delivery (4 papers), Cancer Cells and Metastasis (3 papers), RNA modifications and cancer (3 papers), Proteoglycans and glycosaminoglycans research (3 papers) and HER2/EGFR in Cancer Research (2 papers). The work is most often cited by research in Cancer Research (131 citations), Immunology and Allergy (43 citations), Oncology (126 citations), Molecular Biology (307 citations) and Food Science (62 citations). Iker Badiola has collaborated with scholars based in Spain, France and United States. Frequent co-authors include Alejandro Sánchez, Elvira Olaso, Olatz Crende, Joana Márquez, Abdel‐Majid Khatib, Gaskon Ibarretxe, Scott L. Friedman, Fernando Vidal‐Vanaclocha, Fernando Unda and Patricia García-Gallastegui. Their work appears in journals such as Cancers, Cancer Research, Biology, Scientific Reports and Cell and Tissue Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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