Fernando Vidal‐Vanaclocha

5.1k total citations
72 papers, 3.1k citations indexed

About

Fernando Vidal‐Vanaclocha is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Fernando Vidal‐Vanaclocha has authored 72 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Molecular Biology, 28 papers in Oncology and 20 papers in Immunology. Recurrent topics in Fernando Vidal‐Vanaclocha's work include Cell Adhesion Molecules Research (12 papers), Angiogenesis and VEGF in Cancer (11 papers) and Liver physiology and pathology (9 papers). Fernando Vidal‐Vanaclocha is often cited by papers focused on Cell Adhesion Molecules Research (12 papers), Angiogenesis and VEGF in Cancer (11 papers) and Liver physiology and pathology (9 papers). Fernando Vidal‐Vanaclocha collaborates with scholars based in Spain, United States and Italy. Fernando Vidal‐Vanaclocha's co-authors include Lorea Mendoza, Miren J. Anasagasti, Elvira Olaso, Charles A. Dinarello, Clarisa Salado, Aintzane Asumendi, Emilio Barberá‐Guillem, Teresa Carrascal, Javier Martı́n and María Valcárcel and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Angewandte Chemie International Edition and PLoS ONE.

In The Last Decade

Fernando Vidal‐Vanaclocha

71 papers receiving 3.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fernando Vidal‐Vanaclocha Spain	34	1.6k	965	942	487	447	72	3.1k
Antonio Mazzocca Italy	30	1.4k 0.9×	815 0.8×	343 0.4×	691 1.4×	768 1.7×	86	3.0k
Olivier Dormond Switzerland	33	1.7k 1.1×	1.1k 1.2×	422 0.4×	584 1.2×	146 0.3×	72	3.3k
Hitoshi Akedo Japan	31	2.1k 1.4×	793 0.8×	549 0.6×	582 1.2×	117 0.3×	96	3.5k
Masuo Hosokawa Japan	30	1.2k 0.7×	854 0.9×	779 0.8×	657 1.3×	89 0.2×	88	2.7k
Douglas C. Hixson United States	34	1.6k 1.0×	800 0.8×	261 0.3×	350 0.7×	793 1.8×	110	3.2k
Oliver Stoeltzing United States	43	3.0k 1.9×	1.6k 1.6×	383 0.4×	1.4k 2.8×	323 0.7×	73	4.6k
Yoichi Matsuo Japan	32	1.3k 0.8×	1.3k 1.4×	552 0.6×	563 1.2×	101 0.2×	153	3.0k
Patricia Sancho Spain	33	2.5k 1.6×	1.4k 1.5×	608 0.6×	1.3k 2.6×	362 0.8×	61	4.0k
Stefania Scala Italy	42	2.0k 1.3×	2.8k 2.9×	1.5k 1.6×	930 1.9×	192 0.4×	130	5.1k
Manfred Jücker Germany	30	2.0k 1.3×	904 0.9×	518 0.5×	553 1.1×	111 0.2×	104	3.2k

Countries citing papers authored by Fernando Vidal‐Vanaclocha

Since Specialization

Citations

This map shows the geographic impact of Fernando Vidal‐Vanaclocha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fernando Vidal‐Vanaclocha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fernando Vidal‐Vanaclocha more than expected).

Fields of papers citing papers by Fernando Vidal‐Vanaclocha

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fernando Vidal‐Vanaclocha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fernando Vidal‐Vanaclocha. The network helps show where Fernando Vidal‐Vanaclocha may publish in the future.

Co-authorship network of co-authors of Fernando Vidal‐Vanaclocha

This figure shows the co-authorship network connecting the top 25 collaborators of Fernando Vidal‐Vanaclocha. A scholar is included among the top collaborators of Fernando Vidal‐Vanaclocha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fernando Vidal‐Vanaclocha. Fernando Vidal‐Vanaclocha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Vidal‐Vanaclocha, Fernando, Olatz Crende, Alejandro Herreros‐Pomares, et al.. (2020). Liver prometastatic reaction: Stimulating factors and responsive cancer phenotypes. Seminars in Cancer Biology. 71. 122–133. 9 indexed citations

Crende, Olatz, Marianna Sabatino, María Valcárcel, et al.. (2013). Metastatic Lesions with and without Interleukin-18–Dependent Genes in Advanced-Stage Melanoma Patients. American Journal Of Pathology. 183(1). 69–82. 11 indexed citations

Valcárcel, María, Teresa Carrascal, Olatz Crende, & Fernando Vidal‐Vanaclocha. (2013). IL-18 Regulates Melanoma VLA-4 Integrin Activation through a Hierarchized Sequence of Inflammatory Factors. Journal of Investigative Dermatology. 134(2). 470–480. 25 indexed citations

Gril, Brunilde, Diane Palmieri, Yongzhen Qian, et al.. (2013). Pazopanib Inhibits the Activation of PDGFRβ-Expressing Astrocytes in the Brain Metastatic Microenvironment of Breast Cancer Cells. American Journal Of Pathology. 182(6). 2368–2379. 48 indexed citations

Pérez-Garay, Marta, Beatriz Arteta, Esther Llop, et al.. (2013). α2,3-Sialyltransferase ST3Gal IV promotes migration and metastasis in pancreatic adenocarcinoma cells and tends to be highly expressed in pancreatic adenocarcinoma tissues. The International Journal of Biochemistry & Cell Biology. 45(8). 1748–1757. 74 indexed citations

Vidal‐Vanaclocha, Fernando. (2011). The Liver Prometastatic Reaction of Cancer Patients: Implications for Microenvironment-Dependent Colon Cancer Gene Regulation. Cancer Microenvironment. 4(2). 163–180. 23 indexed citations

Valcárcel, María, Lorea Mendoza, Teresa Carrascal, et al.. (2011). Vascular endothelial growth factor regulates melanoma cell adhesion and growth in the bone marrow microenvironment via tumor cyclooxygenase-2. Journal of Translational Medicine. 9(1). 142–142. 18 indexed citations

Salado, Clarisa, Elvira Olaso, María Valcárcel, et al.. (2011). Resveratrol prevents inflammation-dependent hepatic melanoma metastasis by inhibiting the secretion and effects of interleukin-18. Journal of Translational Medicine. 9(1). 59–59. 47 indexed citations

Arteta, Beatriz, et al.. (2010). Colon Carcinoma Cell Interaction With Liver Sinusoidal Endothelium Inhibits Organ-Specific Antitumor Immunity Through Interleukin-1-Induced Mannose Receptor in Mice. Hepatology. 51(6). 2172–2182. 64 indexed citations

10.

Pérez-Garay, Marta, Beatriz Arteta, Lluı́s Pagès, et al.. (2010). α2,3-Sialyltransferase ST3Gal III Modulates Pancreatic Cancer Cell Motility and Adhesion In Vitro and Enhances Its Metastatic Potential In Vivo. PLoS ONE. 5(9). e12524–e12524. 73 indexed citations

11.

Badiola, Iker, et al.. (2007). Implication of discoidin domain receptors in the activation of hepatic stellate cells during liver metastasis. Cancer Research. 67. 2457–2457. 1 indexed citations

12.

Badiola, Iker, Yves A. DeClerck, Steven J. Wall, Fernando Vidal‐Vanaclocha, & Elvira Olaso. (2006). The role of DDR1 and DDR2 collagen receptors in tumor progression. Cancer Research. 66. 394–394. 1 indexed citations

13.

Rodríguez‐Cuesta, Juan, et al.. (2005). Effect of Asymptomatic Natural Infections due to Common Mouse Pathogens on the Metastatic Progression of B16 Murine Melanoma in C57BL/6 Mice. Clinical & Experimental Metastasis. 22(7). 549–558. 14 indexed citations

14.

Mendoza, Lorea & Fernando Vidal‐Vanaclocha. (2003). Infl ammatory Response of Tumor-Activated Hepatic Sinusoidal Endothelium as a Target for the Screening of Metastasis Chemopreventive Drugs. Humana Press eBooks. 85. 107–116. 1 indexed citations

15.

Carretero, Julián, Elena Obrador, Miren J. Anasagasti, et al.. (1999). Growth-associated changes in glutathione content correlate with liver metastatic activity of B16 melanoma cells. Clinical & Experimental Metastasis. 17(7). 567–574. 101 indexed citations

16.

Anasagasti, Miren J., Javier Martı́n, Lorea Mendoza, et al.. (1998). Glutathione protects metastatic melanoma cells against oxidative stress in the murine hepatic microvasculature. Hepatology. 27(5). 1249–1256. 54 indexed citations

17.

Olaso, Elvira, et al.. (1997). Tumor-dependent activation of rodent hepatic stellate cells during experimental melanoma metastasis. Hepatology. 26(3). 634–642. 118 indexed citations

18.

Anasagasti, Miren J., A Alvarez, Camila Avivi, & Fernando Vidal‐Vanaclocha. (1996). Interleukin-1-mediated H2O2 production by hepatic sinusoidal endothelium in response to B16 melanoma cell adhesion. Journal of Cellular Physiology. 167(2). 314–323. 31 indexed citations

19.

Vidal‐Vanaclocha, Fernando, D Glaves, Emilio Barberá‐Guillem, & L. Weiss. (1991). Quantitative microscopy of mouse colon 26 cells growing in different metastatic sites. British Journal of Cancer. 63(5). 748–752. 7 indexed citations

20.

Barberá‐Guillem, Emilio, et al.. (1989). Differential location of hemopoietic colonies within liver acini of postnatal and phenylhydrazine-treated adult mice. Hepatology. 9(1). 29–36. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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