I. E. Smith

1.1k citations
27 papers · 848 indexed · h-index 12

I. E. Smith

26 papers receiving 816 citations

Peers

I. E. Smith
Comparison fields: 5 of 70
  • Cancer Research 403
  • Oncology 503
  • Pathology and Forensic Medicine 137
  • Genetics 200
  • Dermatology 45
Replace E. Bourstyn with:
E. Bourstyn France
S. Ashley United Kingdom
J. Carmichael United Kingdom
Takaomi Suzuma Japan
Masafumi Inokuchi Japan
A McKinna United Kingdom
A. Michael Bilous Australia
R Bettelheim United Kingdom
R. Knight United Kingdom
K. Mehta Germany
I. E. Smith relative to E. Bourstyn France E. Bourstyn's profile →
Citations per field
00.5×1.6×
E. Bourstyn · 1×
Citations per year

Countries citing papers authored by I. E. Smith

Since Specialization
Citations

This map shows the geographic impact of I. E. Smith's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. E. Smith with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. E. Smith more than expected).

Fields of papers citing papers by I. E. Smith

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by I. E. Smith. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. E. Smith. The network helps show where I. E. Smith may publish in the future.

Co-authorship network

The 25 scholars most cited alongside I. E. Smith, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with I. E. Smith Line = papers co-authored together I. E. Smith links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 202510
2 20184
3 201153
4 20118
5 20111
6 20096
7 20081
8 2006234
9 200533
10 20041
11
HER-2 amplification impedes the antiproliferative effects of hormone therapy in estrogen receptor-positive primary breast cancer.
2001150
12 200170
13 200166
14 199611
15 199022
16 198758
17 19852
18 197718
19 19772
20 197647

About I. E. Smith

I. E. Smith is a scholar working on Cancer Research, Oncology, Genetics, Internal Medicine and Dermatology, having authored 27 papers that have together received 848 indexed citations. Recurring topics across this work include Breast Cancer Treatment Studies (12 papers), Estrogen and related hormone effects (9 papers), Cancer Treatment and Pharmacology (7 papers), Advanced Breast Cancer Therapies (5 papers), HER2/EGFR in Cancer Research (4 papers), Neuroendocrine Tumor Research Advances (2 papers), Neuroblastoma Research and Treatments (2 papers) and Monoclonal and Polyclonal Antibodies Research (2 papers). The work is most often cited by research in Cancer Research (403 citations), Oncology (503 citations), Pathology and Forensic Medicine (137 citations), Genetics (200 citations) and Dermatology (45 citations). I. E. Smith has collaborated with scholars based in United Kingdom, United States and Canada. Frequent co-authors include Sunil R. Lakhani, Jorge S. Reis‐Filho, Susana Banerjee, S. Ashley, Lara Lipton, D Steele, A. Ashworth, Janine Salter, Martin Gore and Mitch Dowsett. Their work appears in journals such as Journal of Clinical Oncology, Cancer Research, Postgraduate Medical Journal, Cancer and BJS Open.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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