Hussein Al‐Mossawi

2.2k total citations
51 papers, 1.2k citations indexed

About

Hussein Al‐Mossawi is a scholar working on Rheumatology, Immunology and Hematology. According to data from OpenAlex, Hussein Al‐Mossawi has authored 51 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Rheumatology, 25 papers in Immunology and 13 papers in Hematology. Recurrent topics in Hussein Al‐Mossawi's work include Spondyloarthritis Studies and Treatments (17 papers), Psoriasis: Treatment and Pathogenesis (14 papers) and Systemic Lupus Erythematosus Research (13 papers). Hussein Al‐Mossawi is often cited by papers focused on Spondyloarthritis Studies and Treatments (17 papers), Psoriasis: Treatment and Pathogenesis (14 papers) and Systemic Lupus Erythematosus Research (13 papers). Hussein Al‐Mossawi collaborates with scholars based in United Kingdom, United States and Sweden. Hussein Al‐Mossawi's co-authors include Paul Bowness, Ariane Hammitzsch, Jelle de Wit, Julian C. Knight, Anna Ridley, Fernando O. Martínez, Benjamin P. Fairfax, Davide Simone, Hai Fang and Andrew Carr and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Nature Medicine.

In The Last Decade

Hussein Al‐Mossawi

43 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hussein Al‐Mossawi United Kingdom 17 594 455 341 278 234 51 1.2k
Masahiko Tsuboi Japan 18 333 0.6× 212 0.5× 445 1.3× 190 0.7× 69 0.3× 31 973
Kei Sakata Japan 15 432 0.7× 373 0.8× 216 0.6× 128 0.5× 55 0.2× 21 905
Theodore J. Yun United States 15 723 1.2× 123 0.3× 1.0k 2.9× 602 2.2× 113 0.5× 22 1.7k
Motoharu Kaneko Japan 11 283 0.5× 229 0.5× 258 0.8× 177 0.6× 40 0.2× 14 750
Hélder Trindade Portugal 17 421 0.7× 156 0.3× 353 1.0× 161 0.6× 88 0.4× 42 985
Peter Gribling United States 12 595 1.0× 190 0.4× 215 0.6× 174 0.6× 85 0.4× 12 1.0k
Eva Emmell United States 12 371 0.6× 136 0.3× 297 0.9× 237 0.9× 139 0.6× 18 1.0k
Katsuhiro Nishioka Japan 13 318 0.5× 452 1.0× 411 1.2× 283 1.0× 92 0.4× 14 1.0k
Sook Kyung Chang United States 11 642 1.1× 175 0.4× 319 0.9× 271 1.0× 67 0.3× 13 1.1k
Begoña Santiago Spain 11 242 0.4× 294 0.6× 214 0.6× 177 0.6× 77 0.3× 13 619

Countries citing papers authored by Hussein Al‐Mossawi

Since Specialization
Citations

This map shows the geographic impact of Hussein Al‐Mossawi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hussein Al‐Mossawi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hussein Al‐Mossawi more than expected).

Fields of papers citing papers by Hussein Al‐Mossawi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hussein Al‐Mossawi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hussein Al‐Mossawi. The network helps show where Hussein Al‐Mossawi may publish in the future.

Co-authorship network of co-authors of Hussein Al‐Mossawi

This figure shows the co-authorship network connecting the top 25 collaborators of Hussein Al‐Mossawi. A scholar is included among the top collaborators of Hussein Al‐Mossawi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hussein Al‐Mossawi. Hussein Al‐Mossawi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Newey, Alice, Guillaume Rieunier, Tara Mahon, et al.. (2025). Cell-targeted PD-1 agonists are potent NK-cell inhibitors. Frontiers in Immunology. 16. 1640509–1640509.
2.
Vollenhoven, Ronald van, Eric F. Morand, Richard Furie, et al.. (2024). LBO1 DORIS remission in patients with SLE treated with anifrolumab or placebo during the 4-year TULIP-LTE trial: post hoc analysis. SHILAP Revista de lepidopterología. A169–A170. 2 indexed citations
4.
Gilchrist, James J., Hai Fang, Sara Danielli, et al.. (2024). Characterization of the genetic determinants of context-specific DNA methylation in primary monocytes. Cell Genomics. 4(5). 100541–100541. 1 indexed citations
5.
Morand, Eric F., Ronald van Vollenhoven, Richard Furie, et al.. (2023). OP0051 LUPUS LOW DISEASE ACTIVITY STATE ATTAINMENT IN THE PHASE 3 PLACEBO-CONTROLLED TULIP LONG-TERM EXTENSION TRIAL OF ANIFROLUMAB. Annals of the Rheumatic Diseases. 82. 33–34. 5 indexed citations
6.
Jayne, David, Patrick J. Gavin, Erik L. Allman, et al.. (2023). OP0136 METABOLOMIC SERUM PROFILING IDENTIFIES METABOLITES LINKED TO KIDNEY DAMAGE WHICH ARE MODULATED BY ANIFROLUMAB IN A PHASE 2 TRIAL IN LUPUS NEPHRITIS. Annals of the Rheumatic Diseases. 82. 90–90. 2 indexed citations
8.
Vollenhoven, Ronald van, Eric F. Morand, Ian N Bruce, et al.. (2023). Targeting DORIS Remission and LLDAS in SLE: A Review. Rheumatology and Therapy. 10(6). 1459–1477. 14 indexed citations
9.
11.
Al‐Mossawi, Hussein, Chris Worth, Patrícia Maria Abreu Machado, et al.. (2021). A first in disease phase 2A trial of granulocyte monocyte colony stimulating factor neutralisation for axial spondyloarthritis (NAMASTE study). Clinical and Experimental Rheumatology. 39(5). 1161–1161.
12.
Yager, Nicole, Suzanne Cole, Alicia Lledó Lara, et al.. (2021). Ex vivo mass cytometry analysis reveals a profound myeloid proinflammatory signature in psoriatic arthritis synovial fluid. Annals of the Rheumatic Diseases. 80(12). 1559–1567. 29 indexed citations
13.
Simone, Davide, Frank Penkava, Anna Ridley, et al.. (2021). Single cell analysis of spondyloarthritis regulatory T cells identifies distinct synovial gene expression patterns and clonal fates. Communications Biology. 4(1). 1395–1395. 33 indexed citations
14.
Fairfax, Benjamin P., Chelsea Taylor, Robert Watson, et al.. (2020). Peripheral CD8+ T cell characteristics associated with durable responses to immune checkpoint blockade in patients with metastatic melanoma. Nature Medicine. 26(2). 193–199. 198 indexed citations
15.
Kendal, Adrian, T. B. Layton, Hussein Al‐Mossawi, et al.. (2020). Multi-omic single cell analysis resolves novel stromal cell populations in healthy and diseased human tendon. Scientific Reports. 10(1). 13939–13939. 89 indexed citations
16.
Al‐Mossawi, Hussein & Laura C. Coates. (2018). Personalized medicine — a new reality in psoriatic arthritis?. Nature Reviews Rheumatology. 14(8). 449–451. 4 indexed citations
17.
Bowness, Paul, et al.. (2018). Novel Therapeutic Targets in Axial Spondyloarthritis. PubMed. 4(2). 174–182. 8 indexed citations
18.
Dakin, Stephanie G., Christopher D. Buckley, Hussein Al‐Mossawi, et al.. (2017). Persistent stromal fibroblast activation is present in chronic tendinopathy. Arthritis Research & Therapy. 19(1). 16–16. 76 indexed citations
19.
Hammitzsch, Ariane, C. Tallant, O. Fedorov, et al.. (2015). CBP30, a selective CBP/p300 bromodomain inhibitor, suppresses human Th17 responses. Proceedings of the National Academy of Sciences. 112(34). 10768–10773. 195 indexed citations
20.
Al‐Mossawi, Hussein, et al.. (2013). CHARACTERIZING TYPE 17 IMMUNE RESPONSES IN ANKYLOSING SPONDYLITIS. Lara D. Veeken. 52. 28–29. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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