Ariane Hammitzsch

1.1k total citations
18 papers, 816 citations indexed

About

Ariane Hammitzsch is a scholar working on Rheumatology, Immunology and Hematology. According to data from OpenAlex, Ariane Hammitzsch has authored 18 papers receiving a total of 816 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Rheumatology, 7 papers in Immunology and 5 papers in Hematology. Recurrent topics in Ariane Hammitzsch's work include Spondyloarthritis Studies and Treatments (7 papers), Psoriasis: Treatment and Pathogenesis (5 papers) and Autoimmune and Inflammatory Disorders Research (3 papers). Ariane Hammitzsch is often cited by papers focused on Spondyloarthritis Studies and Treatments (7 papers), Psoriasis: Treatment and Pathogenesis (5 papers) and Autoimmune and Inflammatory Disorders Research (3 papers). Ariane Hammitzsch collaborates with scholars based in Germany, United Kingdom and Netherlands. Ariane Hammitzsch's co-authors include Hussein Al‐Mossawi, Paul Bowness, Hendrik Schulze‐Koops, M. Witt, Fabian Proft, Jelle de Wit, Jan Leipe, Alla Skapenko, Qihui Zhou and Johannes Kreuzer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Scientific Reports.

In The Last Decade

Ariane Hammitzsch

16 papers receiving 806 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ariane Hammitzsch Germany 11 371 316 288 212 126 18 816
Anette Knedla Germany 4 305 0.8× 244 0.8× 162 0.6× 95 0.4× 100 0.8× 5 650
Priscilla Wong China 16 300 0.8× 492 1.6× 201 0.7× 68 0.3× 91 0.7× 32 893
Begoña Santiago Spain 11 294 0.8× 214 0.7× 242 0.8× 77 0.4× 78 0.6× 13 619
Delphine Lemeiter France 14 220 0.6× 202 0.6× 304 1.1× 65 0.3× 42 0.3× 22 658
B N Franklin Germany 4 385 1.0× 224 0.7× 184 0.6× 95 0.4× 102 0.8× 5 711
Kurisu Tada Japan 12 283 0.8× 231 0.7× 447 1.6× 68 0.3× 306 2.4× 47 868
Antonia Puchner Austria 10 137 0.4× 238 0.8× 196 0.7× 42 0.2× 179 1.4× 26 539
B. Vitolo Italy 12 396 1.1× 111 0.4× 281 1.0× 90 0.4× 26 0.2× 20 727
GS Firestein United States 10 243 0.7× 187 0.6× 125 0.4× 51 0.2× 108 0.9× 16 534
L. Ballantine United Kingdom 6 131 0.4× 328 1.0× 236 0.8× 50 0.2× 288 2.3× 8 628

Countries citing papers authored by Ariane Hammitzsch

Since Specialization
Citations

This map shows the geographic impact of Ariane Hammitzsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ariane Hammitzsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ariane Hammitzsch more than expected).

Fields of papers citing papers by Ariane Hammitzsch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ariane Hammitzsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ariane Hammitzsch. The network helps show where Ariane Hammitzsch may publish in the future.

Co-authorship network of co-authors of Ariane Hammitzsch

This figure shows the co-authorship network connecting the top 25 collaborators of Ariane Hammitzsch. A scholar is included among the top collaborators of Ariane Hammitzsch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ariane Hammitzsch. Ariane Hammitzsch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
2.
Hammitzsch, Ariane, Georg Lorenz, M. Witt, et al.. (2024). Increased interleukin-26 in the peripheral joints of patients with axial spondyloarthritis and psoriatic arthritis, co-localizing with CD68-positive synoviocytes. Frontiers in Immunology. 15. 1355824–1355824. 3 indexed citations
3.
Schmaderer, Christoph, Ariane Hammitzsch, Matthias C. Braunisch, et al.. (2023). Immunophenotypic Characterization of Citrate-Containing A Concentrates in Maintenance Hemodialysis: A Pre-Post Study. International Journal of Nephrology. 2023. 1–10.
4.
Cordts, Isabell, Judith S. Hecker, Joohyun Park, et al.. (2021). Successful treatment with azacitidine in VEXAS syndrome with prominent myofasciitis. Lara D. Veeken. 61(5). e117–e119. 21 indexed citations
5.
Hammitzsch, Ariane, Georg Lorenz, & Philipp Moog. (2020). Impact of Janus Kinase Inhibition on the Treatment of Axial Spondyloarthropathies. Frontiers in Immunology. 11. 591176–591176. 15 indexed citations
6.
Hammitzsch, Ariane, Liye Chen, Jelle de Wit, et al.. (2018). Inhibiting ex-vivo Th17 responses in Ankylosing Spondylitis by targeting Janus kinases. Scientific Reports. 8(1). 15645–15645. 33 indexed citations
7.
Al‐Mossawi, Hussein, L. Chen, Hai Fang, et al.. (2017). Unique transcriptome signatures and GM-CSF expression in lymphocytes from patients with spondyloarthritis. Nature Communications. 8(1). 1510–1510. 110 indexed citations
8.
Chen, L, Hussein Al‐Mossawi, Anne J. Ridley, et al.. (2016). miR-10b-5p is a novel Th17 regulator present in Th17 cells from ankylosing spondylitis. Annals of the Rheumatic Diseases. 76(3). 620–624. 60 indexed citations
9.
Hammitzsch, Ariane, L Chen, Hussein Al‐Mossawi, et al.. (2016). OP0156 Deciphering The in Vitro Therapeutic Potential of JAK Inhibitors in Ankylosing Spondylitis. Annals of the Rheumatic Diseases. 75. 115–115. 1 indexed citations
10.
Hammitzsch, Ariane, C. Tallant, O. Fedorov, et al.. (2015). CBP30, a selective CBP/p300 bromodomain inhibitor, suppresses human Th17 responses. Proceedings of the National Academy of Sciences. 112(34). 10768–10773. 195 indexed citations
11.
Chen, Liye, Anna Ridley, Ariane Hammitzsch, et al.. (2015). Silencing or inhibition of endoplasmic reticulum aminopeptidase 1 (ERAP1) suppresses free heavy chain expression and Th17 responses in ankylosing spondylitis. Annals of the Rheumatic Diseases. 75(5). 916–923. 72 indexed citations
12.
Zhou, Qihui, Johannes Kreuzer, Ariane Hammitzsch, et al.. (2014). Decreased expression of miR-146a and miR-155 contributes to an abnormal Treg phenotype in patients with rheumatoid arthritis. Annals of the Rheumatic Diseases. 74(6). 1265–1274. 154 indexed citations
13.
Hammitzsch, Ariane, Jelle de Wit, Anne J. Ridley, Hussein Al‐Mossawi, & Paul Bowness. (2014). AB0022 Comparison of in Vitro Effects of Kinase and Epigenetic Inhibitors on TH17 Responses in Inflammatory Arthritis. Annals of the Rheumatic Diseases. 73. 811–811. 4 indexed citations
14.
Witt, M., et al.. (2013). Disease burden, disease manifestations and current treatment regimen of the SAPHO syndrome in Germany: Results from a nationwide patient survey. Seminars in Arthritis and Rheumatism. 43(6). 745–750. 40 indexed citations
15.
Neumann, Christian, et al.. (2013). THU0515 Hypogammaglobulinemia is a Frequent Finding in Patients Under Immunosuppressive Therapy but Does not Correlate with Susceptibility to Infections. Annals of the Rheumatic Diseases. 72. A338–A338. 1 indexed citations
16.
Witt, M., Axel Nigg, Christiane Reindl, et al.. (2013). Relevance of Grade 1 Gray‐Scale Ultrasound Findings in Wrists and Small Joints to the Assessment of Subclinical Synovitis in Rheumatoid Arthritis. Arthritis & Rheumatism. 65(7). 1694–1701. 75 indexed citations
17.
18.
Mannell, Hanna, et al.. (2009). Suppression of DNA-PKcs enhances FGF-2 dependent human endothelial cell proliferation via negative regulation of Akt. Cellular Signalling. 22(1). 88–96. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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