Huadong Pei

1.4k total citations
23 papers, 1.0k citations indexed

About

Huadong Pei is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Huadong Pei has authored 23 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 6 papers in Oncology and 4 papers in Cell Biology. Recurrent topics in Huadong Pei's work include DNA Repair Mechanisms (14 papers), Microtubule and mitosis dynamics (4 papers) and PARP inhibition in cancer therapy (4 papers). Huadong Pei is often cited by papers focused on DNA Repair Mechanisms (14 papers), Microtubule and mitosis dynamics (4 papers) and PARP inhibition in cancer therapy (4 papers). Huadong Pei collaborates with scholars based in China, United States and Hong Kong. Huadong Pei's co-authors include Zhenkun Lou, Liewei Wang, Yuxin Qin, Marta Chesi, Yueyang Fei, Lindsey Zhang, P. Leif Bergsagel, Zhongsheng You, Kuntian Luo and Yan Yao and has published in prestigious journals such as Nature, Nucleic Acids Research and Nature Communications.

In The Last Decade

Huadong Pei

23 papers receiving 1.0k citations

Peers

Huadong Pei

ONCOL

IMMUN

GENET

Kyoko Kojima United States

Hai Zheng China

Guy J. Leclerc United States

Katelyn O’Neill United States

Cong Liang China

Linda Fong United States

Damien J. Downes United Kingdom

Julia Zinngrebe Germany

Pam Tangvoranuntakul United States

Kyoko Kojima United States

Huadong Pei

523 ×0.6

162 ×0.9

ONCOL

124 ×1.3

152 ×2.0

IMMUN

28 ×0.4

GENET

Citations per year, relative to Huadong Pei Huadong Pei (= 1×) peers Kyoko Kojima

Countries citing papers authored by Huadong Pei

Since Specialization

Citations

This map shows the geographic impact of Huadong Pei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huadong Pei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huadong Pei more than expected).

Fields of papers citing papers by Huadong Pei

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huadong Pei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huadong Pei. The network helps show where Huadong Pei may publish in the future.

Co-authorship network of co-authors of Huadong Pei

This figure shows the co-authorship network connecting the top 25 collaborators of Huadong Pei. A scholar is included among the top collaborators of Huadong Pei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huadong Pei. Huadong Pei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Chen, Lulu, Mengxue Hu, Yihan Peng, et al.. (2024). Targeting O-GlcNAcylation in cancer therapeutic resistance: The sugar Saga continues. Cancer Letters. 588. 216742–216742. 15 indexed citations

Zhang, Yi, Jing Li, Yuan Zhou, et al.. (2022). And-1 Coordinates with the FANCM Complex to Regulate Fanconi Anemia Signaling and Cisplatin Resistance. Cancer Research. 82(18). 3249–3262. 10 indexed citations

Zhang, Simin, Yihan Peng, Xin Zhao, et al.. (2021). Yeast Bromodomain Factor 1 and Its Human Homolog TAF1 Play Conserved Roles in Promoting Homologous Recombination. Advanced Science. 8(15). e2100753–e2100753. 9 indexed citations

Zhang, Yi, Zhuqing Li, Qiang Hao, et al.. (2019). The Cdk2–c-Myc–miR-571 Axis Regulates DNA Replication and Genomic Stability by Targeting Geminin. Cancer Research. 79(19). 4896–4910. 17 indexed citations

Xue, Renyu, et al.. (2019). Metastasis suppressor NME1 promotes non-homologous end joining of DNA double-strand breaks. DNA repair. 77. 27–35. 11 indexed citations

Ha, Kyungsoo, Lin Han, Lichun Tang, et al.. (2017). The anaphase promoting complex impacts repair choice by protecting ubiquitin signalling at DNA damage sites. Nature Communications. 8(1). 15751–15751. 19 indexed citations

Tan, Wei, Hua Guan, Yu Wang, et al.. (2017). Overexpression of TNKS1BP1 in lung cancers and its involvement in homologous recombination pathway of DNA double‐strand breaks. Cancer Medicine. 6(2). 483–493. 14 indexed citations

Chen, Yali, Hailong Liu, Haoxing Zhang, et al.. (2016). And-1 coordinates with CtIP for efficient homologous recombination and DNA damage checkpoint maintenance. Nucleic Acids Research. 45(5). 2516–2530. 25 indexed citations

Guo, Xuewu, Yazhou Wang, Lina Li, et al.. (2015). Reduction of biogenic amines production by eliminating the PEP4 gene in Saccharomyces cerevisiae during fermentation of Chinese rice wine. Food Chemistry. 178. 208–211. 19 indexed citations

10.

Yang, Yeran, Zhenbo Liu, Fengli Wang, et al.. (2015). FANCD2 and REV1 cooperate in the protection of nascent DNA strands in response to replication stress. Nucleic Acids Research. 43(17). 8325–8339. 40 indexed citations

11.

Liu, Hailong, Haoxing Zhang, Xiaohui Wang, et al.. (2015). The Deubiquitylating Enzyme USP4 Cooperates with CtIP in DNA Double-Strand Break End Resection. Cell Reports. 13(1). 93–107. 52 indexed citations

12.

Peng, Changmin, Xia Liu, Hailong Liu, et al.. (2015). OGT Mediated Histone H2B S112 GlcNAcylation Regulates DNA Damage Response. Journal of genetics and genomics. 42(9). 467–475. 27 indexed citations

13.

Lin, Yi–Hui, Jian Yuan, Huadong Pei, et al.. (2015). KAP1 Deacetylation by SIRT1 Promotes Non-Homologous End-Joining Repair. PLoS ONE. 10(4). e0123935–e0123935. 46 indexed citations

14.

Xu, Qiuran, Caihong Yang, Yu Du, et al.. (2014). AMPK regulates histone H2B O-GlcNAcylation. Nucleic Acids Research. 42(9). 5594–5604. 76 indexed citations

15.

Guo, Xuewu, Yazhou Wang, Chaoqun Li, et al.. (2014). Effect of the inactivation of lactate dehydrogenase, ethanol dehydrogenase, and phosphotransacetylase on 2,3-butanediol production in Klebsiella pneumoniae strain. Biotechnology for Biofuels. 7(1). 44–44. 67 indexed citations

16.

Liu, Jingfang, Jie Li, Zhenfang Wu, et al.. (2012). Identification and Characterization of the Cognate Anti-Sigma Factor and Specific Promoter Elements of a T. tengcongensis ECF Sigma Factor. PLoS ONE. 7(7). e40885–e40885. 4 indexed citations

17.

Pei, Huadong, Lindsey Zhang, Kuntian Luo, et al.. (2011). MMSET regulates histone H4K20 methylation and 53BP1 accumulation at DNA damage sites. Nature. 470(7332). 124–128. 334 indexed citations

18.

Pei, Huadong, et al.. (2010). Krüppel-like factor KLF9 regulates PPARγ transactivation at the middle stage of adipogenesis. Cell Death and Differentiation. 18(2). 315–327. 114 indexed citations

19.

Liu, Jingfang, Huadong Pei, Shuangshuang Mei, et al.. (2008). Replication initiator DnaA interacts with an anti-terminator NusG in T. tengcongensis. Biochemical and Biophysical Research Communications. 371(3). 573–577. 11 indexed citations

20.

Pei, Huadong, Jingfang Liu, Yun Cheng, et al.. (2005). Expression of SARS-coronavirus nucleocapsid protein in Escherichia coli and Lactococcus lactis for serodiagnosis and mucosal vaccination. Applied Microbiology and Biotechnology. 68(2). 220–227. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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