Jacob G. Jansen
- Molecular Biology top 5%
- Cancer Research top 5%
- Oncology
- Plant Science top 10%
- Pathology and Forensic Medicine top 10%
- Co-authors
- Niels de WindA.A. van ZeelandHarry VrielingAnastasia Tsaalbi‐ShtylikHeinz JacobsLeon H.F. MullendersG.R. MohnPetra Langerak
- Topics
- DNA Repair Mechanisms (36 papers)Carcinogens and Genotoxicity Assessment (24 papers)CRISPR and Genetic Engineering (15 papers)
- Journals
- Proceedings of the National Academy of SciencesNucleic Acids ResearchThe Journal of Experimental Medicine
- Partner nations
- NetherlandsFranceJapan
In The Last Decade
Jacob G. Jansen
42 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 94
- Molecular Biology 1.4k
- Cancer Research 606
- Oncology 227
- Plant Science 172
- Pathology and Forensic Medicine 122
Countries citing papers authored by Jacob G. Jansen
This map shows the geographic impact of Jacob G. Jansen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jacob G. Jansen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jacob G. Jansen more than expected).
Fields of papers citing papers by Jacob G. Jansen
This network shows the impact of papers produced by Jacob G. Jansen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jacob G. Jansen. The network helps show where Jacob G. Jansen may publish in the future.
Co-authorship network of co-authors of Jacob G. Jansen
This figure shows the co-authorship network connecting the top 25 collaborators of Jacob G. Jansen. A scholar is included among the top collaborators of Jacob G. Jansen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jacob G. Jansen. Jacob G. Jansen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 52 | |
| 2 | 35 | |
| 3 | 40 | |
| 4 | 29 | |
| 5 | 17 | |
| 6 | 33 | |
| 7 | 54 | |
| 8 | 17 | |
| 9 | 44 | |
| 10 | 171 | |
| 11 | 71 | |
| 12 | 23 | |
| 13 | 43 | |
| 14 | 38 | |
| 15 | 83 | |
| 16 | 44 | |
| 17 | 3 | |
| 18 | 21 | |
| 19 | 31 | |
| 20 | 49 |
About Jacob G. Jansen
Jacob G. Jansen is a scholar working on Cancer Research, Molecular Biology and Pathology and Forensic Medicine, having authored 42 papers that have together received 1.6k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (36 papers), Carcinogens and Genotoxicity Assessment (24 papers) and CRISPR and Genetic Engineering (15 papers). The work is most often cited by research in Cancer Research (606 citations), Molecular Biology (1.4k citations) and Aging (18 citations). Jacob G. Jansen has collaborated with scholars based in Netherlands, France and Japan. Frequent co-authors include Niels de Wind, A.A. van Zeeland, Harry Vrieling, Anastasia Tsaalbi‐Shtylik, Heinz Jacobs, Leon H.F. Mullenders, G.R. Mohn, Petra Langerak, Paul van den Berk and Giel Hendriks. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and The Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.