Horia Vais
- Molecular Biology top 5%
- Insect Science top 1%
- Cell Biology top 5%
- Cellular and Molecular Neuroscience top 5%
- Epidemiology top 10%
- Co-authors
- J. Kevin FoskettP.N.R. UsherwoodMartin S. WilliamsonKing‐Ho CheungA. L. DevonshireJun YangCraig B. ThompsonDon‐On Daniel Mak
- Topics
- Ion channel regulation and function (23 papers)Neuroscience and Neuropharmacology Research (11 papers)Cardiac electrophysiology and arrhythmias (8 papers)
- Partner nations
- United StatesUnited KingdomRomania
In The Last Decade
Horia Vais
43 papers receiving 2.5k citations
Hit Papers
Peers
Comparison fields: 5 of 119
- Molecular Biology 1.8k
- Insect Science 452
- Cell Biology 412
- Cellular and Molecular Neuroscience 374
- Epidemiology 330
Countries citing papers authored by Horia Vais
This map shows the geographic impact of Horia Vais's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Horia Vais with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Horia Vais more than expected).
Fields of papers citing papers by Horia Vais
This network shows the impact of papers produced by Horia Vais. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Horia Vais. The network helps show where Horia Vais may publish in the future.
Co-authorship network of co-authors of Horia Vais
This figure shows the co-authorship network connecting the top 25 collaborators of Horia Vais. A scholar is included among the top collaborators of Horia Vais based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Horia Vais. Horia Vais is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 43 | |
| 2 | 26 | |
| 3 | 12 | |
| 4 | 102 | |
| 5 | 116 | |
| 6 | 16 | |
| 7 | 22 | |
| 8 | Essential Regulation of Cell Bioenergetics by Constitutive InsP3 Receptor Ca2+ Transfer to Mitochondriabreakdown → | 851 |
| 9 | 30 | |
| 10 | 8 | |
| 11 | 106 | |
| 12 | 55 | |
| 13 | 24 | |
| 14 | 33 | |
| 15 | 14 | |
| 16 | 157 | |
| 17 | 6 | |
| 18 | 22 | |
| 19 | 58 | |
| 20 | 14 |
About Horia Vais
Horia Vais is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Insect Science, having authored 44 papers that have together received 2.5k indexed citations. Recurring topics across this work include Ion channel regulation and function (23 papers), Neuroscience and Neuropharmacology Research (11 papers) and Cardiac electrophysiology and arrhythmias (8 papers). The work is most often cited by research in Physiology (211 citations), Insect Science (452 citations) and Molecular Biology (1.8k citations). Horia Vais has collaborated with scholars based in United States, United Kingdom and Romania. Frequent co-authors include J. Kevin Foskett, P.N.R. Usherwood, Martin S. Williamson, King‐Ho Cheung, A. L. Devonshire, Jun Yang, Craig B. Thompson, Don‐On Daniel Mak, Ian Parker and César Cárdenas. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.