Holger Schneider
- Co-authors
- Max SchneiderFriedrich RosenBernhard SallerGünter K. StallaB HusemannManfred UhrStephan PetersennMartín Reincke
- Topics
- Hormonal Regulation and Hypertension (18 papers)Adrenal and Paraganglionic Tumors (9 papers)Adrenal Hormones and Disorders (9 papers)
- Journals
- Proceedings of the National Academy of SciencesCirculation ResearchThe Journal of Clinical Endocrinology & Metabolism
- Partner nations
- GermanySwitzerlandUnited Kingdom
In The Last Decade
Holger Schneider
47 papers receiving 984 citations
Peers
Comparison fields: 5 of 100
- Endocrinology, Diabetes and Metabolism 562
- Surgery 208
- Molecular Biology 208
- Neurology 166
- Physiology 127
Countries citing papers authored by Holger Schneider
This map shows the geographic impact of Holger Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Holger Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Holger Schneider more than expected).
Fields of papers citing papers by Holger Schneider
This network shows the impact of papers produced by Holger Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Holger Schneider. The network helps show where Holger Schneider may publish in the future.
Co-authorship network of co-authors of Holger Schneider
This figure shows the co-authorship network connecting the top 25 collaborators of Holger Schneider. A scholar is included among the top collaborators of Holger Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Holger Schneider. Holger Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 3 | |
| 4 | 2 | |
| 5 | 3 | |
| 6 | 15 | |
| 7 | 1 | |
| 8 | 3 | |
| 9 | 12 | |
| 10 | 16 | |
| 11 | 3 | |
| 12 | 80 | |
| 13 | 177 | |
| 14 | 16 | |
| 15 | 2 | |
| 16 | 65 | |
| 17 | 70 | |
| 18 | 1 | |
| 19 | 12 | |
| 20 | Enzyme response to thyrotoxicosis and hypothyroidism in human liver and muscle | 1 |
About Holger Schneider
Holger Schneider is a scholar working on Endocrinology, Diabetes and Metabolism, Behavioral Neuroscience and Neurology, having authored 50 papers that have together received 1.0k indexed citations. Recurring topics across this work include Hormonal Regulation and Hypertension (18 papers), Adrenal and Paraganglionic Tumors (9 papers) and Adrenal Hormones and Disorders (9 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (562 citations), Neurology (166 citations) and Bioengineering (29 citations). Holger Schneider has collaborated with scholars based in Germany, Switzerland and United Kingdom. Frequent co-authors include Max Schneider, Friedrich Rosen, Bernhard Saller, Günter K. Stalla, B Husemann, Manfred Uhr, Stephan Petersenn, Martín Reincke, Daniel Heinrich and Christian Adolf. Their work appears in journals such as Proceedings of the National Academy of Sciences, Circulation Research and The Journal of Clinical Endocrinology & Metabolism.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.