Hitoshi Ishibashi

2.0k total citations
76 papers, 1.7k citations indexed

About

Hitoshi Ishibashi is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Hitoshi Ishibashi has authored 76 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Cellular and Molecular Neuroscience, 45 papers in Molecular Biology and 16 papers in Physiology. Recurrent topics in Hitoshi Ishibashi's work include Neuroscience and Neuropharmacology Research (43 papers), Ion channel regulation and function (33 papers) and Pain Mechanisms and Treatments (12 papers). Hitoshi Ishibashi is often cited by papers focused on Neuroscience and Neuropharmacology Research (43 papers), Ion channel regulation and function (33 papers) and Pain Mechanisms and Treatments (12 papers). Hitoshi Ishibashi collaborates with scholars based in Japan, Australia and United States. Hitoshi Ishibashi's co-authors include Norio Akaike, Junichi Nabekura, Kei Eto, JeongSeop Rhee, Shutaro Katsurabayashi, Yoshinaka Murai, Hiroaki Wake, Yoshito Mizoguchi, Yushi Ito and Yuchio Yanagawa and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and Nature Neuroscience.

In The Last Decade

Hitoshi Ishibashi

76 papers receiving 1.7k citations

Peers

Hitoshi Ishibashi
Edward C. Burgard United States
Cheng Long China
Il‐Sung Jang South Korea
Christophe Drieu La Rochelle France
Geraint Price United Kingdom
Steven R. Glaum United States
Masumi Inoue Japan
Karen Maubach United Kingdom
Patrick L. Sheets United States
Javad Mirnajafi‐Zadeh Iran
Edward C. Burgard United States
Hitoshi Ishibashi
Citations per year, relative to Hitoshi Ishibashi Hitoshi Ishibashi (= 1×) peers Edward C. Burgard

Countries citing papers authored by Hitoshi Ishibashi

Since Specialization
Citations

This map shows the geographic impact of Hitoshi Ishibashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hitoshi Ishibashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hitoshi Ishibashi more than expected).

Fields of papers citing papers by Hitoshi Ishibashi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hitoshi Ishibashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hitoshi Ishibashi. The network helps show where Hitoshi Ishibashi may publish in the future.

Co-authorship network of co-authors of Hitoshi Ishibashi

This figure shows the co-authorship network connecting the top 25 collaborators of Hitoshi Ishibashi. A scholar is included among the top collaborators of Hitoshi Ishibashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hitoshi Ishibashi. Hitoshi Ishibashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Eto, Kei, et al.. (2024). Ketogenic Diet Alleviates Mechanical Allodynia in the Models of Inflammatory and Neuropathic Pain in Male Mice. Biological and Pharmaceutical Bulletin. 47(3). 629–634. 1 indexed citations
2.
Sasaki‐Hamada, Sachie, et al.. (2019). Excitatory effect of bradykinin on intrinsic neurons of the rat heart. Neuropeptides. 75. 65–74. 7 indexed citations
3.
Ogata, Masanori, et al.. (2019). Behavioral responses to anxiogenic tasks in young adult rats with neonatal dopamine depletion. Physiology & Behavior. 204. 10–19. 2 indexed citations
4.
Sasaki‐Hamada, Sachie, et al.. (2018). Effects of centrally administered glucagon-like peptide-2 on blood pressure and barosensitive neurons in spontaneously hypertensive rats. Neuropeptides. 69. 66–75. 3 indexed citations
5.
Nakahata, Yoshihisa, Kei Eto, Hideji Murakoshi, et al.. (2017). Activation-Dependent Rapid Postsynaptic Clustering of Glycine Receptors in Mature Spinal Cord Neurons. eNeuro. 4(1). ENEURO.0194–16.2017. 7 indexed citations
6.
Ishibashi, Hitoshi, Junya Yamaguchi, Yoshihisa Nakahata, & Junichi Nabekura. (2013). Dynamic regulation of glycine–GABA co‐transmission at spinal inhibitory synapses by neuronal glutamate transporter. The Journal of Physiology. 591(16). 3821–3832. 26 indexed citations
7.
Eto, Kei, Hitoshi Ishibashi, Takeshi Yoshimura, et al.. (2012). Enhanced GABAergic Activity in the Mouse Primary Somatosensory Cortex Is Insufficient to Alleviate Chronic Pain Behavior with Reduced Expression of Neuronal Potassium–Chloride Cotransporter. Journal of Neuroscience. 32(47). 16552–16559. 41 indexed citations
8.
Eto, Kei, Hiroaki Wake, Miho Watanabe, et al.. (2011). Inter-regional Contribution of Enhanced Activity of the Primary Somatosensory Cortex to the Anterior Cingulate Cortex Accelerates Chronic Pain Behavior. Journal of Neuroscience. 31(21). 7631–7636. 103 indexed citations
9.
Mizokami, Akiko, Takashi Kanematsu, Hitoshi Ishibashi, et al.. (2007). Phosholipase C-Related Inactive Protein Is Involved in Trafficking of γ2 Subunit-Containing GABAAReceptors to the Cell Surface. Journal of Neuroscience. 27(7). 1692–1701. 65 indexed citations
10.
Eto, Kei, et al.. (2007). The effect of zinc on glycinergic inhibitory postsynaptic currents in rat spinal dorsal horn neurons. Brain Research. 1161. 11–20. 7 indexed citations
11.
Ishibashi, Hitoshi, et al.. (2005). Activation of presynaptic GABAA receptors increases spontaneous glutamate release onto noradrenergic neurons of the rat locus coeruleus. Brain Research. 1046(1-2). 24–31. 27 indexed citations
12.
Taketomi, Takaharu, Daigo Yoshiga, Koji Taniguchi, et al.. (2005). Loss of mammalian Sprouty2 leads to enteric neuronal hyperplasia and esophageal achalasia. Nature Neuroscience. 8(7). 855–857. 113 indexed citations
13.
Nabekura, Junichi, Shutaro Katsurabayashi, Yasuhiro Kakazu, et al.. (2003). Developmental switch from GABA to glycine release in single central synaptic terminals. Nature Neuroscience. 7(1). 17–23. 176 indexed citations
14.
15.
Ishibashi, Hitoshi, et al.. (2001). Activation of potassium conductance by ophiopogonin‐D in acutely dissociated rat paratracheal neurones. British Journal of Pharmacology. 132(2). 461–466. 33 indexed citations
16.
Murai, Yoshinaka, et al.. (2000). Preferential inhibition of L- and N-type calcium channels in the rat hippocampal neurons by cilnidipine. Brain Research. 854(1-2). 6–10. 17 indexed citations
17.
18.
Murai, Yoshinaka, Hitoshi Ishibashi, Norio Akaike, & Yushi Ito. (1998). Acetylcholine modulation of high‐voltage‐activated calcium channels in the neurones acutely dissociated from rat paratracheal ganglia. British Journal of Pharmacology. 123(7). 1441–1449. 22 indexed citations
19.
Ishibashi, Hitoshi, Jeong Seop Rhee, & Norio Akaike. (1997). Effect of nilvadipine on high-voltage activated Ca2+ channels in rat CNS neurons. Neuroreport. 8(4). 853–857. 11 indexed citations
20.
Ueno, Shinya, Hitoshi Ishibashi, & Norio Akaike. (1992). Perforated-patch method reveals extracellular ATP-induced K+ conductance in dissociated rat nucleus solitarii neurons. Brain Research. 597(1). 176–179. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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