Hirataka Ito
- Molecular Biology top 5%
- Physiology top 2%
- Immunology top 10%
- Cellular and Molecular Neuroscience top 5%
- Genetics top 10%
- Co-authors
- Satoshi ShiojiriNobuya KitaguchiYasuyuki TakahashiYasuo TokushimaKeiichi ItakuraTakashi ShiraiR. Bruce WallaceC. W. Todd
- Topics
- Alzheimer's disease research and treatments (6 papers)Prion Diseases and Protein Misfolding (4 papers)Bacterial Genetics and Biotechnology (4 papers)
- Partner nations
- JapanUnited StatesUnited Kingdom
In The Last Decade
Hirataka Ito
22 papers receiving 2.3k citations
Hit Papers
Peers
Comparison fields: 5 of 102
- Molecular Biology 1.6k
- Physiology 1.1k
- Immunology 360
- Cellular and Molecular Neuroscience 276
- Genetics 242
Countries citing papers authored by Hirataka Ito
This map shows the geographic impact of Hirataka Ito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hirataka Ito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hirataka Ito more than expected).
Fields of papers citing papers by Hirataka Ito
This network shows the impact of papers produced by Hirataka Ito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hirataka Ito. The network helps show where Hirataka Ito may publish in the future.
Co-authorship network of co-authors of Hirataka Ito
This figure shows the co-authorship network connecting the top 25 collaborators of Hirataka Ito. A scholar is included among the top collaborators of Hirataka Ito based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hirataka Ito. Hirataka Ito is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 22 | |
| 2 | 36 | |
| 3 | 48 | |
| 4 | 124 | |
| 5 | 56 | |
| 6 | 6 | |
| 7 | 4 | |
| 8 | 26 | |
| 9 | Novel precursor of Alzheimer's disease amyloid protein shows protease inhibitory activitybreakdown → | 905 |
| 10 | 114 | |
| 11 | 46 | |
| 12 | 40 | |
| 13 | 34 | |
| 14 | 396 | |
| 15 | CLONING AND EXPRESSION IN E.COLI OF THE GENE FOR HUMAN TUMOR NECROSIS FACTOR | 9 |
| 16 | 189 | |
| 17 | 48 | |
| 18 | 204 | |
| 19 | 20 | |
| 20 | 38 |
About Hirataka Ito
Hirataka Ito is a scholar working on Microbiology, Molecular Biology and Physiology, having authored 22 papers that have together received 2.4k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (6 papers), Prion Diseases and Protein Misfolding (4 papers) and Bacterial Genetics and Biotechnology (4 papers). The work is most often cited by research in Physiology (1.1k citations), Molecular Biology (1.6k citations) and Immunology (360 citations). Hirataka Ito has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Satoshi Shiojiri, Nobuya Kitaguchi, Yasuyuki Takahashi, Yasuo Tokushima, Keiichi Itakura, Takashi Shirai, R. Bruce Wallace, C. W. Todd, Hiroshi Yamaguchi and Satoshi Ikuta. Their work appears in journals such as Nature, Nucleic Acids Research and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.