Hideyuki Miyazaki

474 total citations
9 papers, 354 citations indexed

About

Hideyuki Miyazaki is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Surgery. According to data from OpenAlex, Hideyuki Miyazaki has authored 9 papers receiving a total of 354 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Cellular and Molecular Neuroscience and 2 papers in Surgery. Recurrent topics in Hideyuki Miyazaki's work include Protein Kinase Regulation and GTPase Signaling (3 papers), Neuroscience and Neuropharmacology Research (2 papers) and Axon Guidance and Neuronal Signaling (2 papers). Hideyuki Miyazaki is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (3 papers), Neuroscience and Neuropharmacology Research (2 papers) and Axon Guidance and Neuronal Signaling (2 papers). Hideyuki Miyazaki collaborates with scholars based in Japan and United States. Hideyuki Miyazaki's co-authors include Yasunori Kanaho, Masakazu Yamazaki, Hiroshi Watanabe, Tomohiko Maehama, Michael A. Frohman, Takehiko Sasaki, Fumiaki Mori, Shinsuke Iida, Masaki Ri and Shigeru Kusumoto and has published in prestigious journals such as Journal of Biological Chemistry, FEBS Letters and Journal of Neurochemistry.

In The Last Decade

Hideyuki Miyazaki

9 papers receiving 353 citations

Peers

Hideyuki Miyazaki

MB

CB

HEMAT

CMN

ONCOL

Eszter Varga Hungary

M. Victoria Simón Argentina

Daniel K. Borger United States

Deanna Grant United States

José-Marı́a Trifaró Canada

Kristine J. Fernandez Australia

J P Steiner United States

Francesca Rocchio Italy

Ying Xia China

Raffaella Calligaris Italy

Eszter Varga Hungary

Hideyuki Miyazaki

316 ×1.2

MB

51 ×0.5

CB

49 ×0.7

HEMAT

39 ×0.6

CMN

13 ×0.3

ONCOL

Citations per year, relative to Hideyuki Miyazaki Hideyuki Miyazaki (= 1×) peers Eszter Varga

Countries citing papers authored by Hideyuki Miyazaki

Since Specialization

Citations

This map shows the geographic impact of Hideyuki Miyazaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hideyuki Miyazaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hideyuki Miyazaki more than expected).

Fields of papers citing papers by Hideyuki Miyazaki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hideyuki Miyazaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hideyuki Miyazaki. The network helps show where Hideyuki Miyazaki may publish in the future.

Co-authorship network of co-authors of Hideyuki Miyazaki

This figure shows the co-authorship network connecting the top 25 collaborators of Hideyuki Miyazaki. A scholar is included among the top collaborators of Hideyuki Miyazaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hideyuki Miyazaki. Hideyuki Miyazaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Miyazaki, Hideyuki. (2016). Material and Fabrication Technology of Sports Shoes. Seikei-Kakou. 28(5). 172–175. 1 indexed citations

2.

Ri, Masaki, Shinsuke Iida, Takayuki Nakashima, et al.. (2010). Bortezomib-resistant myeloma cell lines: a role for mutated PSMB5 in preventing the accumulation of unfolded proteins and fatal ER stress. Leukemia. 24(8). 1506–1512. 139 indexed citations

3.

Miyazaki, Hideyuki, Masakazu Yamazaki, Hiroshi Watanabe, et al.. (2005). The small GTPase ADP‐ribosylation factor 6 negatively regulates dendritic spine formation. FEBS Letters. 579(30). 6834–6838. 36 indexed citations

4.

Watanabe, Hiroshi, Takeaki Yokozeki, Masakazu Yamazaki, et al.. (2004). Essential Role for Phospholipase D2 Activation Downstream of ERK MAP Kinase in Nerve Growth Factor-stimulated Neurite Outgrowth from PC12 Cells. Journal of Biological Chemistry. 279(36). 37870–37877. 41 indexed citations

5.

Watanabe, Hiroshi, Masakazu Yamazaki, Hideyuki Miyazaki, et al.. (2004). Phospholipase D2 functions as a downstream signaling molecule of MAP kinase pathway in L1‐stimulated neurite outgrowth of cerebellar granule neurons. Journal of Neurochemistry. 89(1). 142–151. 50 indexed citations

6.

Kanaho, Yasunori, Hideyuki Miyazaki, & Masakazu Yamazaki. (2003). Activation of PI(4)P 5-kinase by small G proteins. Advances in Enzyme Regulation. 43(1). 107–119. 4 indexed citations

7.

Yamazaki, Masakazu, Hideyuki Miyazaki, Hiroshi Watanabe, et al.. (2002). Phosphatidylinositol 4-Phosphate 5-Kinase Is Essential for ROCK-mediated Neurite Remodeling. Journal of Biological Chemistry. 277(19). 17226–17230. 66 indexed citations

8.

Nishino, Takashi, Hideyuki Miyazaki, & Katsuhiko Nakamae. (2002). X-ray diffraction of polymer under load at cryogenic temperature. Review of Scientific Instruments. 73(4). 1809–1812. 8 indexed citations

9.

Post, Steven R., Hideyuki Miyazaki, & Howard S. Tager. (1992). Identification of a Mg(2+)- and guanyl nucleotide-dependent glucagon receptor cycle by use of permeabilized canine hepatocytes.. Journal of Biological Chemistry. 267(36). 25776–25785. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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