Kodai Kume

576 total citations
34 papers, 347 citations indexed

About

Kodai Kume is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Kodai Kume has authored 34 papers receiving a total of 347 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Cellular and Molecular Neuroscience, 13 papers in Molecular Biology and 13 papers in Neurology. Recurrent topics in Kodai Kume's work include Genetic Neurodegenerative Diseases (8 papers), Mitochondrial Function and Pathology (6 papers) and Parkinson's Disease Mechanisms and Treatments (5 papers). Kodai Kume is often cited by papers focused on Genetic Neurodegenerative Diseases (8 papers), Mitochondrial Function and Pathology (6 papers) and Parkinson's Disease Mechanisms and Treatments (5 papers). Kodai Kume collaborates with scholars based in Japan, China and United Kingdom. Kodai Kume's co-authors include Kazushi Deguchi, Masaki Kamada, Tsutomu Masaki, Tetsuo Touge, Kazuyo Ikeda, Hideshi Kawakami, Masaaki Tokuda, Fuminori Yamaguchi, Naoya Hatano and Tao Song and has published in prestigious journals such as SHILAP Revista de lepidopterología, Biochemical Journal and FEBS Letters.

In The Last Decade

Kodai Kume

33 papers receiving 341 citations

Peers

Kodai Kume

CMN

NEURO

PHYSI

PFM

Seema Shroff United States

Satoru Tada Japan

Jinsong Jiao China

Mahima Kapoor United Kingdom

Madhurima Chatterjee Netherlands

A. Al‐Memar United Kingdom

Kirsty Greenwood United Kingdom

Anthony Fernández-Castañeda United States

Cathy Jensen Belgium

Stefania Stenirri Italy

Seema Shroff United States

Kodai Kume

206 ×1.2

137 ×1.3

CMN

54 ×0.6

NEURO

81 ×1.3

PHYSI

31 ×0.7

PFM

Citations per year, relative to Kodai Kume Kodai Kume (= 1×) peers Seema Shroff

Countries citing papers authored by Kodai Kume

Since Specialization

Citations

This map shows the geographic impact of Kodai Kume's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kodai Kume with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kodai Kume more than expected).

Fields of papers citing papers by Kodai Kume

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kodai Kume. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kodai Kume. The network helps show where Kodai Kume may publish in the future.

Co-authorship network of co-authors of Kodai Kume

This figure shows the co-authorship network connecting the top 25 collaborators of Kodai Kume. A scholar is included among the top collaborators of Kodai Kume based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kodai Kume. Kodai Kume is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kurashige, Takashi, Kodai Kume, Yusuke Sotomaru, et al.. (2025). Optineurin knock-out forms TDP-43 aggregates to regulate TDP-43 protein levels despite autophagic up-regulation and aberrant TDP-43 expression. Neuroscience Research. 216. 104893–104893. 2 indexed citations

Itabashi, Takeshi, Tomoka Morita, Hiroko Kishi, et al.. (2025). Cholesterol ensures ciliary polycystin-2 localization to prevent polycystic kidney disease. Life Science Alliance. 8(4). e202403063–e202403063. 2 indexed citations

Kume, Kodai, et al.. (2024). C9orf72 repeat expansions in Wakayama: One potential cause of amyotrophic lateral sclerosis in the Kii Peninsula, Japan. Journal of the Neurological Sciences. 466. 123209–123209.

Kurashige, Takashi, Tomohiko Ohshita, Kodai Kume, et al.. (2023). ‘Raisin bread sign’ feature of pontine autosomal dominant microangiopathy and leukoencephalopathy. Brain Communications. 5(6). fcad281–fcad281. 2 indexed citations

Kume, Kodai, Tomoko Sekiya, Kazuto Nishinaka, et al.. (2022). Comparison of two families with and without ataxia harboring novel variants in PRKCG. Journal of Human Genetics. 67(10). 595–599. 1 indexed citations

Saito, Rie, Daisuke Oikawa, Yusuke Sato, et al.. (2022). Spinocerebellar ataxia type 17-digenic TBP/STUB1 disease: neuropathologic features of an autopsied patient. Acta Neuropathologica Communications. 10(1). 177–177. 5 indexed citations

Ohshita, Tomohiko, Takashi Kurashige, Hiroyuki Morino, et al.. (2021). FXTAS is difficult to differentiate from neuronal intranuclear inclusion disease through skin biopsy: a case report. BMC Neurology. 21(1). 396–396. 21 indexed citations

Kume, Kodai, et al.. (2021). Novel monoallelic variant in ERLIN2 causes spastic paraplegia converted to amyotrophic lateral sclerosis. Journal of the Neurological Sciences. 430. 119984–119984. 4 indexed citations

Kume, Kodai, Hiroyuki Morino, Ryosuke Ohsawa, et al.. (2021). Analysis of genetic risk factors in Japanese patients with Parkinson’s disease. Journal of Human Genetics. 66(10). 957–964. 4 indexed citations

10.

Kume, Kodai, Jun Sone, Hideki Kobara, et al.. (2020). Long-term MRI findings of adult-onset neuronal intranuclear inclusion disease. Clinical Neurology and Neurosurgery. 201. 106456–106456. 8 indexed citations

11.

Kume, Kodai, Yukiko Matsuda, Takashi Kurashige, et al.. (2020). Genetic screening for potassium channel mutations in Japanese autosomal dominant spinocerebellar ataxia. Journal of Human Genetics. 65(4). 363–369. 9 indexed citations

12.

Kume, Kodai, Hiroyuki Morino, Yukiko Matsuda, et al.. (2020). The first Japanese case of primary familial brain calcification caused by an MYORG variant. Journal of Human Genetics. 65(10). 917–920. 7 indexed citations

13.

Kume, Kodai, Hiroyuki Morino, Ryosuke Miyamoto, et al.. (2020). Middle-age-onset cerebellar ataxia caused by a homozygous TWNK variant: a case report. BMC Medical Genetics. 21(1). 68–68. 8 indexed citations

14.

Mizuno, Noriyoshi, Kodai Kume, Shinji Matsuda, et al.. (2020). Aggressive periodontitis and NOD2 variants. Journal of Human Genetics. 65(10). 841–846. 8 indexed citations

15.

Kume, Kodai, Tetsuo Touge, Kimihiko Kaneko, et al.. (2018). Clinical significance of assaying anti-MOG antibody in cerebrospinal fluid in MOG-antibody-associated diseases: A case report. Multiple Sclerosis and Related Disorders. 28. 165–166. 10 indexed citations

16.

Kume, Kodai, Hisakazu Iwama, Kazushi Deguchi, et al.. (2017). Serum microRNA expression profiling in patients with multiple system atrophy. Molecular Medicine Reports. 17(1). 852–860. 32 indexed citations

17.

Kume, Kodai, Kazuyo Ikeda, Masaki Kamada, et al.. (2016). Dialysis-induced Subdural Hematoma in an Arachnoid Cyst Associated with Autosomal Dominant Polycystic Kidney Disease. Internal Medicine. 55(15). 2065–2067. 2 indexed citations

18.

Deguchi, Kazushi, Kazuyo Ikeda, Kodai Kume, et al.. (2015). Significance of the hot-cross bun sign on T2*-weighted MRI for the diagnosis of multiple system atrophy. Journal of Neurology. 262(6). 1433–1439. 32 indexed citations

19.

Kume, Kodai, Kazuyo Ikeda, Masaki Kamada, et al.. (2013). Successful treatment of HIV-associated chronic inflammatory demyelinating polyneuropathy by early initiation of highly active anti-retroviral therapy. Rinsho Shinkeigaku. 53(5). 362–366. 6 indexed citations

20.

Song, Tao, Naoya Hatano, Kodai Kume, et al.. (2005). Inhibition of neuronal nitric‐oxide synthase by phosphorylation at Threonine1296 in NG108‐15 neuronal cells. FEBS Letters. 579(25). 5658–5662. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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