Helen E. Jolin

7.2k total citations · 2 hit papers
36 papers, 5.7k citations indexed

About

Helen E. Jolin is a scholar working on Immunology, Surgery and Molecular Biology. According to data from OpenAlex, Helen E. Jolin has authored 36 papers receiving a total of 5.7k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Immunology, 10 papers in Surgery and 8 papers in Molecular Biology. Recurrent topics in Helen E. Jolin's work include IL-33, ST2, and ILC Pathways (24 papers), Immune Cell Function and Interaction (19 papers) and Eosinophilic Esophagitis (10 papers). Helen E. Jolin is often cited by papers focused on IL-33, ST2, and ILC Pathways (24 papers), Immune Cell Function and Interaction (19 papers) and Eosinophilic Esophagitis (10 papers). Helen E. Jolin collaborates with scholars based in United Kingdom, Ireland and Germany. Helen E. Jolin's co-authors include Andrew N. J. McKenzie, Padraic G. Fallon, See Heng Wong, Jillian L. Barlow, Daniel R. Neill, Richard Pannell, Maria Daly, Christine M. Bucks, Colleen Kane and Robin J. Flynn and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Helen E. Jolin

36 papers receiving 5.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity

2010 1.6k citations Daniel R. Neill, See Heng Wong et al. Nature profile →
Identification of an interleukin (IL)-25–dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion

2006 596 citations Padraic G. Fallon, Sarah J. Ballantyne et al. The Journal of Experimental Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Helen E. Jolin United Kingdom	26	4.2k	2.3k	1.2k	732	322	36	5.7k
Jillian L. Barlow United Kingdom	29	4.7k 1.1×	2.7k 1.2×	1.5k 1.3×	491 0.7×	208 0.6×	39	5.9k
Kazuyo Moro Japan	29	3.9k 0.9×	2.3k 1.0×	1.1k 0.9×	403 0.6×	182 0.6×	61	4.9k
Steven J. Van Dyken United States	24	3.2k 0.8×	1.7k 0.7×	1.2k 1.0×	652 0.9×	170 0.5×	42	4.5k
Koubun Yasuda Japan	28	3.1k 0.7×	1.0k 0.4×	961 0.8×	1.1k 1.6×	274 0.9×	57	4.6k
Ariel Munitz Israel	36	2.6k 0.6×	789 0.3×	1.2k 1.0×	908 1.2×	442 1.4×	93	4.6k
Brandon M. Sullivan United States	21	4.6k 1.1×	1.0k 0.4×	793 0.7×	844 1.2×	775 2.4×	26	5.8k
Terri M. Laufer United States	34	4.2k 1.0×	635 0.3×	582 0.5×	911 1.2×	503 1.6×	58	5.6k
Jennifer K. Bando United States	21	2.4k 0.6×	944 0.4×	812 0.7×	428 0.6×	288 0.9×	29	3.4k
Colleen Kane United States	20	2.1k 0.5×	1.2k 0.5×	622 0.5×	482 0.7×	154 0.5×	46	3.4k
Alma J. Nauta Netherlands	26	2.1k 0.5×	1.2k 0.5×	478 0.4×	1.3k 1.8×	424 1.3×	35	5.4k

Countries citing papers authored by Helen E. Jolin

Since Specialization

Citations

This map shows the geographic impact of Helen E. Jolin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Helen E. Jolin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Helen E. Jolin more than expected).

Fields of papers citing papers by Helen E. Jolin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Helen E. Jolin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Helen E. Jolin. The network helps show where Helen E. Jolin may publish in the future.

Co-authorship network of co-authors of Helen E. Jolin

This figure shows the co-authorship network connecting the top 25 collaborators of Helen E. Jolin. A scholar is included among the top collaborators of Helen E. Jolin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Helen E. Jolin. Helen E. Jolin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Clark, Paula A., Noé Rodríguez‐Rodríguez, Ana C. F. Ferreira, et al.. (2024). Recipient tissue microenvironment determines developmental path of intestinal innate lymphoid progenitors. Nature Communications. 15(1). 7809–7809. 5 indexed citations

Szeto, Aydan C. H., Ana C. F. Ferreira, Paula A. Clark, et al.. (2022). An αvβ3 integrin checkpoint is critical for efficient TH2 cell cytokine polarization and potentiation of antigen-specific immunity. Nature Immunology. 24(1). 123–135. 26 indexed citations

Damgaard, Rune Busk, Helen E. Jolin, Michael Allison, et al.. (2020). OTULIN protects the liver against cell death, inflammation, fibrosis, and cancer. Cell Death and Differentiation. 27(5). 1457–1474. 59 indexed citations

Panova, Veera, Mayuri Gogoi, Noé Rodríguez‐Rodríguez, et al.. (2020). Group-2 innate lymphoid cell-dependent regulation of tissue neutrophil migration by alternatively activated macrophage-secreted Ear11. Mucosal Immunology. 14(1). 26–37. 11 indexed citations

Lu, Yuning, Gemma Basatemur, Ian C. Scott, et al.. (2020). Interleukin-33 Signaling Controls the Development of Iron-Recycling Macrophages. Immunity. 52(5). 782–793.e5. 35 indexed citations

Kerscher, Bernhard, Jillian L. Barlow, Batika M.J. Rana, et al.. (2019). BET Bromodomain Inhibitor iBET151 Impedes Human ILC2 Activation and Prevents Experimental Allergic Lung Inflammation. Frontiers in Immunology. 10. 678–678. 18 indexed citations

Halim, Timotheus Y.F., Batika M.J. Rana, Jennifer A. Walker, et al.. (2018). Tissue-Restricted Adaptive Type 2 Immunity Is Orchestrated by Expression of the Costimulatory Molecule OX40L on Group 2 Innate Lymphoid Cells. Immunity. 48(6). 1195–1207.e6. 198 indexed citations

Salimi, Maryam, Luzheng Xue, Helen E. Jolin, et al.. (2015). Group 2 Innate Lymphoid Cells Express Functional NKp30 Receptor Inducing Type 2 Cytokine Production. The Journal of Immunology. 196(1). 45–54. 76 indexed citations

Lucken‐Ardjomande, Safa, Yvonne Vallis, Helen E. Jolin, Andrew N. J. McKenzie, & Harvey T. McMahon. (2014). GRAF1a is a brain-specific protein promoting lipid droplet clustering and growth and enriched at lipid droplet junctions. Journal of Cell Science. 127(Pt 21). 4602–19. 35 indexed citations

10.

Barlow, Jillian L., See Heng Wong, Sarah J. Ballantyne, Helen E. Jolin, & Andrew N. J. McKenzie. (2011). Tim1 and Tim3 are not essential for experimental allergic asthma. Clinical & Experimental Allergy. 41(7). 1012–1021. 12 indexed citations

11.

Neill, Daniel R., See Heng Wong, Robin J. Flynn, et al.. (2010). Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity. Nature. 464(7293). 1367–1370. 1632 indexed citations breakdown →

12.

Barlow, Jillian L., Lesley Drynan, D. Hewett, et al.. (2009). A p53-dependent mechanism underlies macrocytic anemia in a mouse model of human 5q– syndrome. Nature Medicine. 16(1). 59–66. 254 indexed citations

13.

Fallon, Padraic G., Sarah J. Ballantyne, Niamh E. Mangan, et al.. (2006). Identification of an interleukin (IL)-25–dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion. The Journal of Experimental Medicine. 203(4). 1105–1116. 596 indexed citations breakdown →

14.

Cope, David W., Peer Wulff, Alessandra Oberto, et al.. (2004). Abolition of zolpidem sensitivity in mice with a point mutation in the GABAA receptor γ2 subunit. Neuropharmacology. 47(1). 17–34. 60 indexed citations

15.

Jolin, Helen E., P. H. SMITH, José G. van den Berg, et al.. (2004). Lymphomagenesis, Hydronephrosis, and Autoantibodies Result from Dysregulation of IL-9 and Are Differentially Dependent on Th2 Cytokines. The Journal of Immunology. 173(1). 113–122. 14 indexed citations

16.

Lanoue, Astrid, Menna R. Clatworthy, P. H. SMITH, et al.. (2004). SIGN-R1 Contributes to Protection against Lethal Pneumococcal Infection in Mice. The Journal of Experimental Medicine. 200(11). 1383–1393. 115 indexed citations

17.

Fallon, Padraic G., Helen E. Jolin, P. H. SMITH, et al.. (2002). IL-4 Induces Characteristic Th2 Responses Even in the Combined Absence of IL-5, IL-9, and IL-13. Immunity. 17(1). 7–17. 280 indexed citations

18.

Matthews, David J., Claire Emson, Grahame J. McKenzie, et al.. (2000). IL-13 Is a Susceptibility Factor for Leishmania major Infection. The Journal of Immunology. 164(3). 1458–1462. 129 indexed citations

19.

Kamalati, Tahereh, Helen E. Jolin, Michael Fry, & M. Rufus Crompton. (2000). Expression of the BRK tyrosine kinase in mammary epithelial cells enhances the coupling of EGF signalling to PI 3-kinase and Akt, via erbB3 phosphorylation. Oncogene. 19(48). 5471–5476. 90 indexed citations

20.

Kamalati, Tahereh, Helen E. Jolin, Philip J. Mitchell, et al.. (1996). Brk, a Breast Tumor-derived Non-receptor Protein-tyrosine Kinase, Sensitizes Mammary Epithelial Cells to Epidermal Growth Factor. Journal of Biological Chemistry. 271(48). 30956–30963. 98 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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