Heiko Witt

9.0k total citations · 1 hit paper
100 papers, 3.5k citations indexed

About

Heiko Witt is a scholar working on Surgery, Oncology and Epidemiology. According to data from OpenAlex, Heiko Witt has authored 100 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Surgery, 34 papers in Oncology and 20 papers in Epidemiology. Recurrent topics in Heiko Witt's work include Pancreatitis Pathology and Treatment (50 papers), Pancreatic and Hepatic Oncology Research (30 papers) and Gastrointestinal disorders and treatments (21 papers). Heiko Witt is often cited by papers focused on Pancreatitis Pathology and Treatment (50 papers), Pancreatic and Hepatic Oncology Research (30 papers) and Gastrointestinal disorders and treatments (21 papers). Heiko Witt collaborates with scholars based in Germany, United States and Netherlands. Heiko Witt's co-authors include Michael Becker, W. A. P. Luck, Olfert Landt, Andreas Kage, Volker Keim, Martin Claßen, Ulrich Laß, Hans Christian Hennies, Jeremy S. Wilson and Minoti V. Apte and has published in prestigious journals such as Journal of Biological Chemistry, Nature Genetics and The Journal of Immunology.

In The Last Decade

Heiko Witt

99 papers receiving 3.5k citations

Hit Papers

Mutations in the gene encoding the serine protease inhibi... 2000 2026 2008 2017 2000 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Mutations in the gene encoding the serine protease inhibitor, Kazal type 1 are associated with chronic pancreatitis
    2000 722 citations Heiko Witt, W. A. P. Luck et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Heiko Witt Germany 30 2.3k 1.6k 582 456 374 100 3.5k
Yasunobu Abe Japan 30 908 0.4× 922 0.6× 724 1.2× 608 1.3× 738 2.0× 163 3.5k
Takeshi Kawabe Japan 29 1.4k 0.6× 665 0.4× 520 0.9× 929 2.0× 527 1.4× 82 3.0k
Frank A. Mitros United States 30 1.4k 0.6× 817 0.5× 666 1.1× 161 0.4× 903 2.4× 92 3.8k
Shogo Tanaka Japan 30 1.5k 0.7× 1.0k 0.6× 675 1.2× 191 0.4× 406 1.1× 220 3.5k
Françoise Smets Belgium 28 1.2k 0.5× 828 0.5× 806 1.4× 207 0.5× 428 1.1× 73 3.0k
Violet Esquenazi United States 32 1.6k 0.7× 263 0.2× 542 0.9× 735 1.6× 272 0.7× 153 3.5k
Abed M. Zaitoun United Kingdom 26 807 0.4× 492 0.3× 300 0.5× 236 0.5× 499 1.3× 77 1.9k
Elie‐Serge Zafrani France 26 586 0.3× 531 0.3× 1.0k 1.8× 386 0.8× 342 0.9× 55 2.8k
Sook Hee Hong South Korea 25 404 0.2× 1.0k 0.6× 342 0.6× 232 0.5× 648 1.7× 113 2.2k
Jochen Schmitz Germany 23 1.8k 0.8× 1.0k 0.6× 652 1.1× 3.8k 8.4× 852 2.3× 44 5.6k

Countries citing papers authored by Heiko Witt

Since Specialization
Citations

This map shows the geographic impact of Heiko Witt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heiko Witt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heiko Witt more than expected).

Fields of papers citing papers by Heiko Witt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Heiko Witt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heiko Witt. The network helps show where Heiko Witt may publish in the future.

Co-authorship network of co-authors of Heiko Witt

This figure shows the co-authorship network connecting the top 25 collaborators of Heiko Witt. A scholar is included among the top collaborators of Heiko Witt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Heiko Witt. Heiko Witt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiao, Xunjun, Mark E. Lowe, Bálint Erőss, et al.. (2024). Carboxyl ester lipase hybrid 1 (CEL-HYB1) haplotypes confer varying risk for chronic pancreatitis. Scientific Reports. 14(1). 30965–30965.
2.
Masson, Emmanuelle, Jian‐Min Chen, Claude Férec, et al.. (2023). Genetic and functional analysis of chymotrypsin-like protease (CTRL) in chronic pancreatitis. Pancreatology. 23(8). 957–963. 2 indexed citations
3.
Canaff, Lucie, Emmanuelle Masson, Jian‐Min Chen, et al.. (2021). The three common polymorphisms p.A986S, p.R990G and p.Q1011E in the calcium sensing receptor (CASR) are not associated with chronic pancreatitis. Pancreatology. 21(7). 1299–1304. 3 indexed citations
4.
Rosendahl, Jonas & Heiko Witt. (2021). Pathogenese der chronischen Pankreatitis. Der Internist. 62(10). 1007–1014. 3 indexed citations
5.
Párniczky, Andrea, Maisam Abu‐El‐Haija, Sohail Z. Husain, et al.. (2018). EPC/HPSG evidence-based guidelines for the management of pediatric pancreatitis. Pancreatology. 18(2). 146–160. 68 indexed citations
6.
Woerd, Wendy L. van der, Stan F.J. van de Graaf, Claude Férec, et al.. (2013). Mutational Analysis of ATP8B1 in Patients with Chronic Pancreatitis. PLoS ONE. 8(11). e80553–e80553. 8 indexed citations
7.
Beer, Sebastian, András Szabó, Steven Keiles, et al.. (2012). Comprehensive functional analysis of chymotrypsin C ( CTRC ) variants reveals distinct loss-of-function mechanisms associated with pancreatitis risk. Gut. 62(11). 1616–1624. 72 indexed citations
8.
Boulling, Arnaud, Heiko Witt, Giriraj R. Chandak, et al.. (2011). Assessing the pathological relevance of SPINK1 promoter variants. European Journal of Human Genetics. 19(10). 1066–1073. 16 indexed citations
9.
Király, Orsolya, Arnaud Boulling, Heiko Witt, et al.. (2007). Signal peptide variants that impair secretion of pancreatic secretory trypsin inhibitor (SPINK1) cause autosomal dominant hereditary pancreatitis. Human Mutation. 28(5). 469–476. 66 indexed citations
10.
Morsche, René H. M. te, Joost P.H. Drenth, Kaspar Truninger, et al.. (2007). UGT1A7 polymorphisms in chronic pancreatitis: an example of genotyping pitfalls. The Pharmacogenomics Journal. 8(1). 34–41. 11 indexed citations
11.
Ruppert, Clemens, Małgorzata Wygrecka, Reinhold E. Schmidt, et al.. (2006). Surfactant protein C mutations in sporadic forms of idiopathic interstitial pneumonias. European Respiratory Journal. 29(1). 134–137. 39 indexed citations
12.
Sengler, Claudia, Andreas Günther, Hans‐Dieter Walmrath, et al.. (2005). Evaluation of the −26G>A CC16 polymorphism in acute respiratory distress syndrome*. Critical Care Medicine. 33(10). 2404–2406. 9 indexed citations
13.
Mueller, Tobias, Andreas Mas Marques, Christoph Sarrazin, et al.. (2004). Influence of interleukin 12B (IL12B) polymorphisms on spontaneous and treatment-induced recovery from hepatitis C virus infection. Journal of Hepatology. 41(4). 652–658. 48 indexed citations
14.
Büning, Carsten, Juliane Halangk, Axel Dignaß, et al.. (2004). Keratin 8 Y54H and G62C mutations are not associated with inflammatory bowel disease. Digestive and Liver Disease. 36(6). 388–391. 16 indexed citations
15.
Teich, Niels, et al.. (2003). Analysis of tumour necrosis factor alpha and interleukin 10 promotor variants in patients with chronic pancreatitis. European Journal of Gastroenterology & Hepatology. 15(11). 1223–1227. 18 indexed citations
16.
Witt, Heiko, et al.. (2003). Prenatal Diagnosis of Alagille Syndrome. Journal of Pediatric Gastroenterology and Nutrition. 38(1). 105–106. 6 indexed citations
17.
Witt, Heiko & Michael Becker. (2002). Genetics of Chronic Pancreatitis. Journal of Pediatric Gastroenterology and Nutrition. 34(2). 125–136. 3 indexed citations
18.
Bhatia, Eesh, Gourdas Choudhuri, Sadiq S. Sikora, et al.. (2002). Tropical calcific pancreatitis: Strong association with SPINK1 trypsin inhibitor mutations. Gastroenterology. 123(4). 1020–1025. 148 indexed citations
19.
Witt, Heiko. (2001). Gene Mutations in Children with Chronic Pancreatitis. Pancreatology. 1(5). 432–438. 35 indexed citations
20.
Witt, Heiko, W. A. P. Luck, Hans Christian Hennies, et al.. (2000). Mutations in the gene encoding the serine protease inhibitor, Kazal type 1 are associated with chronic pancreatitis. Nature Genetics. 25(2). 213–216. 722 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Breakdown of academic impact, for papers by Yasunobu Abe Breakdown of academic impact, for papers by Takeshi Kawabe Breakdown of academic impact, for papers by Frank A. Mitros Breakdown of academic impact, for papers by Shogo Tanaka Breakdown of academic impact, for papers by Françoise Smets Breakdown of academic impact, for papers by Violet Esquenazi Breakdown of academic impact, for papers by Abed M. Zaitoun Breakdown of academic impact, for papers by Elie‐Serge Zafrani Breakdown of academic impact, for papers by Sook Hee Hong Breakdown of academic impact, for papers by Jochen Schmitz
Rankless by CCL
2026