Eszter Hegyi

1.4k total citations
45 papers, 758 citations indexed

About

Eszter Hegyi is a scholar working on Surgery, Oncology and Dermatology. According to data from OpenAlex, Eszter Hegyi has authored 45 papers receiving a total of 758 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Surgery, 14 papers in Oncology and 8 papers in Dermatology. Recurrent topics in Eszter Hegyi's work include Pancreatitis Pathology and Treatment (22 papers), Pancreatic and Hepatic Oncology Research (13 papers) and Gastrointestinal disorders and treatments (6 papers). Eszter Hegyi is often cited by papers focused on Pancreatitis Pathology and Treatment (22 papers), Pancreatic and Hepatic Oncology Research (13 papers) and Gastrointestinal disorders and treatments (6 papers). Eszter Hegyi collaborates with scholars based in United States, Hungary and Slovakia. Eszter Hegyi's co-authors include Miklós Sahin‐Tóth, Georg Beyer, Matthias Sendler, Julia Mayerle, Markus M. Lerch, Péter Hegyi, Balázs Csaba Németh, András Szabó, Andrea Geisz and S. Ferenčík and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and PLoS ONE.

In The Last Decade

Eszter Hegyi

40 papers receiving 737 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Eszter Hegyi United States 13 468 261 111 107 94 45 758
Jakob Axelsson Sweden 12 237 0.5× 88 0.3× 52 0.5× 34 0.3× 200 2.1× 23 631
Marni Wiseman Canada 20 156 0.3× 162 0.6× 127 1.1× 291 2.7× 70 0.7× 56 1.0k
Junji Tsuruta Japan 13 105 0.2× 205 0.8× 195 1.8× 45 0.4× 26 0.3× 34 613
Noriko Hashimoto Japan 13 182 0.4× 45 0.2× 356 3.2× 49 0.5× 50 0.5× 25 715
Mohamed H.M. El-Komy Egypt 16 124 0.3× 71 0.3× 166 1.5× 90 0.8× 29 0.3× 65 699
Axel Dignaß Germany 8 243 0.5× 111 0.4× 173 1.6× 78 0.7× 18 0.2× 24 605
Carlos Alberto de Carvalho Fraga Brazil 18 79 0.2× 176 0.7× 84 0.8× 98 0.9× 30 0.3× 58 796
Rachel L. Robson United Kingdom 12 111 0.2× 139 0.5× 307 2.8× 81 0.8× 26 0.3× 20 716
Luc Chouinard Canada 15 146 0.3× 186 0.7× 59 0.5× 71 0.7× 32 0.3× 28 733
Christina Stefanaki Greece 19 89 0.2× 111 0.4× 126 1.1× 165 1.5× 163 1.7× 47 901

Countries citing papers authored by Eszter Hegyi

Since Specialization
Citations

This map shows the geographic impact of Eszter Hegyi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eszter Hegyi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eszter Hegyi more than expected).

Fields of papers citing papers by Eszter Hegyi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eszter Hegyi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eszter Hegyi. The network helps show where Eszter Hegyi may publish in the future.

Co-authorship network of co-authors of Eszter Hegyi

This figure shows the co-authorship network connecting the top 25 collaborators of Eszter Hegyi. A scholar is included among the top collaborators of Eszter Hegyi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eszter Hegyi. Eszter Hegyi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiao, Xunjun, Mark E. Lowe, Bálint Erőss, et al.. (2024). Carboxyl ester lipase hybrid 1 (CEL-HYB1) haplotypes confer varying risk for chronic pancreatitis. Scientific Reports. 14(1). 30965–30965.
2.
Kiss, Bence, Gitta Schlosser, Andrea Geisz, et al.. (2023). Substrate specificity of human chymotrypsin-like protease (CTRL) characterized by phage display-selected small-protein inhibitors. Pancreatology. 23(6). 742–749. 6 indexed citations
3.
Beer, Sebastian, Noémi Gede, Andrea Szentesi, et al.. (2023). Risk of chronic pancreatitis in carriers of the c.180C>T (p.Gly60=) CTRC variant: case-control studies and meta-analysis. Pancreatology. 23(5). 481–490. 4 indexed citations
4.
Gede, Noémi, et al.. (2022). Bicarbonate defective CFTR variants increase risk for chronic pancreatitis: A meta-analysis. PLoS ONE. 17(10). e0276397–e0276397. 5 indexed citations
5.
Szentesi, Andrea, Gyula Farkas, F Izbéki, et al.. (2021). Common calcium-sensing receptor (CASR) gene variants do not modify risk for chronic pancreatitis in a Hungarian cohort. Pancreatology. 21(7). 1305–1310. 5 indexed citations
6.
Hanák, Lilla, Zsolt Szakács, Szabolcs Kiss, et al.. (2021). Oral Proton Pump Inhibitors May Be as Effective as Intravenous in Peptic Ulcer Bleeding: A Systematic Review and Meta-analysis. Clinical and Translational Gastroenterology. 12(4). e00341–e00341. 6 indexed citations
7.
Hegyi, Eszter, et al.. (2019). Chronic pancreatitis with polycystic kidney disease: A rare coincidence?. Nefrología. 40(3). 351–355.
8.
Geisz, Andrea, et al.. (2019). Natural single-nucleotide deletion in chymotrypsinogen C gene increases severity of secretagogue-induced pancreatitis in C57BL/6 mice. JCI Insight. 4(14). e129717–e129717. 15 indexed citations
9.
Hegyi, Eszter & Miklós Sahin‐Tóth. (2018). Trypsinogen isoforms in the ferret pancreas. Scientific Reports. 8(1). 15094–15094. 2 indexed citations
10.
Hegyi, Eszter, et al.. (2018). Chymotrypsin Reduces the Severity of Secretagogue-Induced Pancreatitis in Mice. Gastroenterology. 155(4). 1017–1021. 35 indexed citations
11.
Hegyi, Eszter & Miklós Sahin‐Tóth. (2018). Human CPA1 mutation causes digestive enzyme misfolding and chronic pancreatitis in mice. Gut. 68(2). 301–312. 58 indexed citations
12.
Hegyi, Eszter & Miklós Sahin‐Tóth. (2017). Genetic Risk in Chronic Pancreatitis: The Trypsin-Dependent Pathway. Digestive Diseases and Sciences. 62(7). 1692–1701. 124 indexed citations
13.
Hegyi, Eszter, Andrea Geisz, Miklós Sahin‐Tóth, et al.. (2015). SPINK1 Promoter Variants in Chronic Pancreatitis. Pancreas. 45(1). 148–153. 12 indexed citations
14.
Hegyi, Eszter, et al.. (1996). Vitiligo is associated with HLA-A2 and HLA-Dw7 in the Slovak populations.. PubMed. 42(1-2). 23–5. 24 indexed citations
15.
Hegyi, Eszter, et al.. (1993). Frequency of HLA antigens in persons sensitive to N‐isopropyl‐N'‐phenyl‐p‐ phenylenediamine (IPPD). Contact Dermatitis. 28(3). 194–195. 3 indexed citations
16.
Hegyi, Eszter, Václav Suchý, & Melinda Nagy. (1990). On the question of propolis allergy.. Der Hautarzt. 41(12). 675–679. 8 indexed citations
17.
Hegyi, Eszter, et al.. (1989). The nickel content of moulded bricks as building material. Contact Dermatitis. 21(1). 63–63. 5 indexed citations
18.
Hegyi, Eszter, et al.. (1984). [Overview of the allergens from several feeder regions of Czechoslovakia. Incidence and prevalence of contact hypersensitivity caused by the allergens spread by the population of the feeder areas of Bratislava, Trnava and Banská Bystrica 1978-1980].. PubMed. 32(2). 59–63. 1 indexed citations
19.
Hegyi, Eszter, et al.. (1969). [Usefulness of the electrodermatometric method (EDM) for the determination of procaine allergy].. PubMed. 51(4). 479–85.
20.
Hegyi, Eszter, et al.. (1968). [Verification of established procaine allergy by the method of cultured lymphoycte stimulation].. PubMed. 50(4). 507–15.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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