Juliane Halangk

982 total citations
20 papers, 530 citations indexed

About

Juliane Halangk is a scholar working on Hepatology, Epidemiology and Immunology. According to data from OpenAlex, Juliane Halangk has authored 20 papers receiving a total of 530 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Hepatology, 10 papers in Epidemiology and 6 papers in Immunology. Recurrent topics in Juliane Halangk's work include Hepatitis C virus research (10 papers), Liver Disease Diagnosis and Treatment (9 papers) and Hepatitis B Virus Studies (4 papers). Juliane Halangk is often cited by papers focused on Hepatitis C virus research (10 papers), Liver Disease Diagnosis and Treatment (9 papers) and Hepatitis B Virus Studies (4 papers). Juliane Halangk collaborates with scholars based in Germany, United States and Switzerland. Juliane Halangk's co-authors include Thomas Berg, V Weich, Heiko Witt, Bertram Wiedenmann, Gero Puhl, A Bergk, Konrad Neumann, Tobias Müller, Eckart Schott and Ulrich Spengler and has published in prestigious journals such as Hepatology, Clinical Cancer Research and Journal of Hepatology.

In The Last Decade

Juliane Halangk

20 papers receiving 520 citations

Peers

Juliane Halangk
Juliane Halangk
Citations per year, relative to Juliane Halangk Juliane Halangk (= 1×) peers José‐María Sánchez‐Tapias

Countries citing papers authored by Juliane Halangk

Since Specialization
Citations

This map shows the geographic impact of Juliane Halangk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Juliane Halangk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Juliane Halangk more than expected).

Fields of papers citing papers by Juliane Halangk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Juliane Halangk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Juliane Halangk. The network helps show where Juliane Halangk may publish in the future.

Co-authorship network of co-authors of Juliane Halangk

This figure shows the co-authorship network connecting the top 25 collaborators of Juliane Halangk. A scholar is included among the top collaborators of Juliane Halangk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Juliane Halangk. Juliane Halangk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Herwaarden, Antonius E. van, Martijn G.H. van Oijen, Greet J. Boland, et al.. (2010). Influence of alpha-1 antitrypsin heterozygosity on treatment efficacy of HCV combination therapy. European Journal of Gastroenterology & Hepatology. 22(7). 808–812. 2 indexed citations
2.
Marques, Andreas Mas, Tobias Mueller, Justus Welke, et al.. (2009). Low-density lipoprotein receptor variants are associated with spontaneous and treatment-induced recovery from hepatitis C virus infection. Infection Genetics and Evolution. 9(5). 847–852. 11 indexed citations
3.
Halangk, Juliane, Thomas Berg, Konrad Neumann, et al.. (2009). Evaluation of Angiotensinogen c.1-44G>A and p.M268T Variants as Risk Factors for Fibrosis Progression in Chronic Hepatitis C and Liver Diseases of Various Etiologies. Genetic Testing and Molecular Biomarkers. 13(3). 407–414. 2 indexed citations
4.
Ockenga, Johann, Sebastian Strunck, Hans‐Ulrich Schulz, et al.. (2009). The Role of Epoxide Hydrolase Y113H Gene Variant in Pancreatic Diseases. Pancreas. 38(4). e97–e101. 7 indexed citations
5.
Halangk, Juliane, Heiko Witt, Gero Puhl, et al.. (2009). 431 HETEROZYGOUS ALPHA-1 ANTITRYPSIN DEFICIENCY AS AN INHERITED RISK FACTOR IN THE DEVELOPMENT OF CHRONIC LIVER DISEASE. Journal of Hepatology. 50. S162–S162. 5 indexed citations
6.
Österreicher, Christoph H., Juliane Halangk, Thomas Berg, et al.. (2008). Evaluation of the transforming growth factor β1 codon 25 (Arg→Pro) polymorphism in alcoholic liver disease. Cytokine. 42(1). 18–23. 12 indexed citations
7.
Halangk, Juliane, Christoph Sarrazin, Konrad Neumann, et al.. (2008). Evaluation of complement factor 5 variants as genetic risk factors for the development of advanced fibrosis in chronic hepatitis C infection. Journal of Hepatology. 49(3). 339–345. 8 indexed citations
8.
Stickel, Felix, Christoph H. Österreicher, Juliane Halangk, et al.. (2008). No Role of Matrixmetalloproteinase‐3 Genetic Promoter Polymorphism 1171 as a Risk Factor for Cirrhosis in Alcoholic Liver Disease. Alcoholism Clinical and Experimental Research. 32(6). 959–965. 9 indexed citations
9.
Hahn, Thomas von, Juliane Halangk, Heiko Witt, et al.. (2008). Relevance of endotoxin receptorCD14andTLR4gene variants in chronic liver disease. Scandinavian Journal of Gastroenterology. 43(5). 584–592. 11 indexed citations
10.
Schaefer, Martin, Axel Hinzpeter, M.G.L. Pich, et al.. (2007). Hepatitis C treatment in “difficult-to-treat” psychiatric patients with pegylated interferon-alpha and ribavirin. Hepatology. 46(4). 991–998. 113 indexed citations
11.
Schott, Eckart, Heiko Witt, Florian van Bömmel, et al.. (2007). Association of CTLA4 single nucleotide polymorphisms with viral but not autoimmune liver disease. European Journal of Gastroenterology & Hepatology. 19(11). 947–951. 41 indexed citations
12.
Schott, E., Heiko Witt, Konrad Neumann, et al.. (2007). Association of TLR7 single nucleotide polymorphisms with chronic HCV‐infection and response to interferon‐a‐based therapy. Journal of Viral Hepatitis. 15(1). 71–78. 65 indexed citations
13.
Schott, Eckart, Heiko Witt, Konrad Neumann, et al.. (2007). A Toll-like receptor 7 single nucleotide polymorphism protects from advanced inflammation and fibrosis in male patients with chronic HCV-infection. Journal of Hepatology. 47(2). 203–211. 74 indexed citations
14.
Müller, Tobias, Reinhard Geßner, C. Sarrazin, et al.. (2007). Protektiver Effekt des Apolipoprotein E4 Allels bei Hepatitis C Virus (HCV) Infektion. Zeitschrift für Gastroenterologie. 45(8). 1 indexed citations
15.
Feldmann, Georg, Hans Dieter Nischalke, Jacob Nattermann, et al.. (2006). Induction of Interleukin-6 by Hepatitis C Virus Core Protein in Hepatitis C–Associated Mixed Cryoglobulinemia and B-Cell Non–Hodgkin's Lymphoma. Clinical Cancer Research. 12(15). 4491–4498. 64 indexed citations
16.
Schott, Eckart, Heiko Witt, Holger Hinrichsen, et al.. (2006). Gender-dependent association of CTLA4 polymorphisms with resolution of hepatitis C virus infection. Journal of Hepatology. 46(3). 372–380. 39 indexed citations
17.
Wiederholt, T., Edgar Dahl, Claus Hellerbrand, et al.. (2006). Identification of the chemokine CXCL9 (MIG) as a genetic modifier of liver fibrosis in patients with hepatitis C. Zeitschrift für Gastroenterologie. 44(8). 1 indexed citations
18.
Mueller, Tobias, Andreas Mas Marques, Christoph Sarrazin, et al.. (2004). Influence of interleukin 12B (IL12B) polymorphisms on spontaneous and treatment-induced recovery from hepatitis C virus infection. Journal of Hepatology. 41(4). 652–658. 48 indexed citations
19.
Büning, Carsten, Juliane Halangk, Axel Dignaß, et al.. (2004). Keratin 8 Y54H and G62C mutations are not associated with inflammatory bowel disease. Digestive and Liver Disease. 36(6). 388–391. 16 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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