Han-Wen Chang

463 citations
13 papers · 339 indexed · h-index 9
Topics
DNA Repair Mechanisms (6 papers)Genomics and Chromatin Dynamics (6 papers)RNA and protein synthesis mechanisms (5 papers)

In The Last Decade

Han-Wen Chang

13 papers receiving 337 citations

Peers

Han-Wen Chang
Comparison fields: 5 of 42
  • Molecular Biology 310
  • Oncology 48
  • Genetics 36
  • Cancer Research 26
  • Pathology and Forensic Medicine 24
Replace Yasunao Kamikawa with:
Yasunao Kamikawa Japan
Xiaochu Chen United States
Reinhard Sigl Austria
Xiaowei Xu China
Bastian Stielow Germany
Marta Vázquez-Cedeira Spain
Sandeep N. Wontakal United States
P. Pavan Kumar India
Joseph J. Kaberlein United States
Sini Rautio Finland
Han-Wen Chang relative to Yasunao Kamikawa Japan Yasunao Kamikawa's profile →
Citations per field
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Yasunao Kamikawa · 1×
Citations per year

Countries citing papers authored by Han-Wen Chang

Since Specialization
Citations

This map shows the geographic impact of Han-Wen Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Han-Wen Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Han-Wen Chang more than expected).

Fields of papers citing papers by Han-Wen Chang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Han-Wen Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Han-Wen Chang. The network helps show where Han-Wen Chang may publish in the future.

Co-authorship network of co-authors of Han-Wen Chang

This figure shows the co-authorship network connecting the top 25 collaborators of Han-Wen Chang. A scholar is included among the top collaborators of Han-Wen Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Han-Wen Chang. Han-Wen Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
#WorkIndexed citations
1 7
2 4
3 14
4 10
5 2
6 61
7 91
8 39
9
Chromatin replication: TRANSmitting the histone code.
1
10 8
11 8
12 37
13
The oncogene qin codes for a transcriptional repressor.
57

About Han-Wen Chang

Han-Wen Chang is a scholar working on Molecular Biology, Oncology and Genetics, having authored 13 papers that have together received 339 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (6 papers), Genomics and Chromatin Dynamics (6 papers) and RNA and protein synthesis mechanisms (5 papers). The work is most often cited by research in Molecular Biology (310 citations), Aging (4 citations) and Developmental Neuroscience (9 citations). Han-Wen Chang has collaborated with scholars based in United States, Russia and Tajikistan. Frequent co-authors include Vasily M. Studitsky, Maria E. Valieva, Katerina V. Gurova, Poorva Sandlesh, Peter K. Vogt, Jian Li, Edmund Lai, М. П. Кирпичников, Olga I. Kulaeva and Alexey V. Feofanov. Their work appears in journals such as Nucleic Acids Research, Journal of Clinical Investigation and International Journal of Molecular Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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