H J Lüth

716 total citations
11 papers, 610 citations indexed

About

H J Lüth is a scholar working on Physiology, Molecular Biology and Clinical Biochemistry. According to data from OpenAlex, H J Lüth has authored 11 papers receiving a total of 610 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 4 papers in Molecular Biology and 4 papers in Clinical Biochemistry. Recurrent topics in H J Lüth's work include Alzheimer's disease research and treatments (4 papers), Advanced Glycation End Products research (4 papers) and Neuroscience and Neuropharmacology Research (2 papers). H J Lüth is often cited by papers focused on Alzheimer's disease research and treatments (4 papers), Advanced Glycation End Products research (4 papers) and Neuroscience and Neuropharmacology Research (2 papers). H J Lüth collaborates with scholars based in Germany, Netherlands and Australia. H J Lüth's co-authors include Thomas Arendt, Gerald Münch, Peter Riederer, Étienne C. Hirsch, Amanda Wong, Rivka Ravid, H.-J. Gertz, Max Holzer, E Winkelmann and K Brauer and has published in prestigious journals such as Brain Research, Journal of Neurochemistry and Cerebral Cortex.

In The Last Decade

H J Lüth

11 papers receiving 595 citations

Peers

H J Lüth

PHYSI

CB

MB

CMN

NEURO

Junichi Takamatsu Japan

Belinda Giorgetti Italy

Tony Valente Spain

Frank Mastrogiacomo Canada

Jayoung Byun South Korea

Jiazhao Xie China

Samanta Oliveira Loureiro Brazil

Moreno Solazzi Italy

S Dózić Serbia

M. Brunetti Italy

Junichi Takamatsu Japan

H J Lüth

513 ×1.6

PHYSI

167 ×0.7

CB

217 ×1.3

MB

129 ×0.8

CMN

279 ×2.0

NEURO

Citations per year, relative to H J Lüth H J Lüth (= 1×) peers Junichi Takamatsu

Countries citing papers authored by H J Lüth

Since Specialization

Citations

This map shows the geographic impact of H J Lüth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H J Lüth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H J Lüth more than expected).

Fields of papers citing papers by H J Lüth

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H J Lüth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H J Lüth. The network helps show where H J Lüth may publish in the future.

Co-authorship network of co-authors of H J Lüth

This figure shows the co-authorship network connecting the top 25 collaborators of H J Lüth. A scholar is included among the top collaborators of H J Lüth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H J Lüth. H J Lüth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Lüth, H J. (2004). Age- and Stage-dependent Accumulation of Advanced Glycation End Products in Intracellular Deposits in Normal and Alzheimer's Disease Brains. Cerebral Cortex. 15(2). 211–220. 156 indexed citations

2.

Münch, Gerald, et al.. (2003). Advanced glycation endproducts and pro‐inflammatory cytokines in transgenic Tg2576 mice with amyloid plaque pathology. Journal of Neurochemistry. 86(2). 283–289. 37 indexed citations

3.

Münch, Gerald, Björn Kuhla, H J Lüth, Thomas Arendt, & Stephen R. Robinson. (2003). Anti-AGEing defences against Alzheimer's disease. Biochemical Society Transactions. 31(6). 1397–1399. 34 indexed citations

4.

Münch, Gerald, H J Lüth, Amanda Wong, et al.. (2000). Crosslinking of α-synuclein by advanced glycation endproducts — an early pathophysiological step in Lewy body formation?. Journal of Chemical Neuroanatomy. 20(3-4). 253–257. 181 indexed citations

5.

Lüth, H J, Max Holzer, H.-J. Gertz, & Thomas Arendt. (2000). Aberrant expression of nNOS in pyramidal neurons in Alzheimer's disease is highly co-localized with p21ras and p16INK4a. Brain Research. 852(1). 45–55. 95 indexed citations

6.

Lüth, H J & Thomas Arendt. (1998). Nitric oxide and Alzheimer's disease.. PubMed. 39(2). 245–51. 17 indexed citations

7.

Seeger, Gudrun, H J Lüth, E Winkelmann, & K Brauer. (1996). Distribution patterns of Wisteria floribunda agglutinin binding sites and parvalbumin-immunoreactive neurons in the human visual cortex: a double-labelling study.. PubMed. 37(3). 351–66. 43 indexed citations

8.

Lüth, H J, Ingmar Blümcke, E Winkelmann, & Marco R. Celio. (1993). The calcium-binding protein calretinin is localized in a subset of interneurons in the rat cerebral cortex: a light and electron immunohistochemical study.. PubMed. 34(1). 93–103. 23 indexed citations

9.

Lüth, H J, et al.. (1988). Noradrenergic sprouting and beta-adrenergic receptor binding in the lateral geniculate nucleus of rats after unilateral visual cortex ablation.. PubMed. 29(6). 659–67. 1 indexed citations

10.

Schöber, W, K Brauer, L Werner, & H J Lüth. (1988). [Anatomy of the nucleus magnocellularis in the basal forebrain of rodents and rabbits].. PubMed. 29(4). 443–59. 3 indexed citations

11.

Brauer, K, L Werner, E Winkelmann, & H J Lüth. (1981). The dorsal lateral geniculate nucleus of Tupaia glis: a Golgi, Nissl and acetylcholinesterase study.. PubMed. 22(1). 59–74. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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