Gustavo González
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 2%
- Genetics top 5%
- Physiology top 10%
- Immunology top 10%
- Co-authors
- Marc MontminyKaren YamamotoRobin E. OffordE. A. LissiCharles W. HewittDonald C. MartinDavid W. FurnasBruce M. Achauer
- Topics
- Erythrocyte Function and Pathophysiology (6 papers)Hemoglobin structure and function (4 papers)Enzyme Production and Characterization (2 papers)
- Partner nations
- United StatesChileArgentina
In The Last Decade
Gustavo González
26 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 122
- Molecular Biology 1.8k
- Cellular and Molecular Neuroscience 748
- Genetics 436
- Physiology 314
- Immunology 292
Countries citing papers authored by Gustavo González
This map shows the geographic impact of Gustavo González's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gustavo González with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gustavo González more than expected).
Fields of papers citing papers by Gustavo González
This network shows the impact of papers produced by Gustavo González. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gustavo González. The network helps show where Gustavo González may publish in the future.
Co-authorship network of co-authors of Gustavo González
This figure shows the co-authorship network connecting the top 25 collaborators of Gustavo González. A scholar is included among the top collaborators of Gustavo González based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gustavo González. Gustavo González is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 15 | |
| 3 | 22 | |
| 4 | 0 | |
| 5 | 4 | |
| 6 | 12 | |
| 7 | 9 | |
| 8 | 17 | |
| 9 | 1 | |
| 10 | 6 | |
| 11 | 13 | |
| 12 | 17 | |
| 13 | 6 | |
| 14 | 24 | |
| 15 | 0 | |
| 16 | 376 | |
| 17 | Cyclic AMP stimulates somatostatin gene transcription by phosphorylation of CREB at serine 133breakdown → | 2204 |
| 18 | 30 | |
| 19 | 2 | |
| 20 | 7 |
About Gustavo González
Gustavo González is a scholar working on Cell Biology, Transplantation and Biotechnology, having authored 29 papers that have together received 3.0k indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (6 papers), Hemoglobin structure and function (4 papers) and Enzyme Production and Characterization (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (748 citations), Developmental Neuroscience (120 citations) and Behavioral Neuroscience (94 citations). Gustavo González has collaborated with scholars based in United States, Chile and Argentina. Frequent co-authors include Marc Montminy, Karen Yamamoto, Robin E. Offord, E. A. Lissi, Charles W. Hewitt, Donald C. Martin, David W. Furnas, Bruce M. Achauer, Edwin B. Howard and Steven F. Dowdy. Their work appears in journals such as Cell, Trends in Neurosciences and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.