Gui‐Xian Zhao
- Cancer Research top 5%
- Neurology top 5%
- Peripheral Neuropathies and Disorders 4
- Neurological diseases and metabolism 3
- Immunology top 10%
- Immunotherapy and Immune Responses 4
-
- Multiple Sclerosis Research Studies 16
- Physiology top 5%
-
- Systemic Lupus Erythematosus Research 9
-
- Genetic Neurodegenerative Diseases 4
-
- Cytokine Signaling Pathways and Interactions 4
-
- Trace Elements in Health 3
- Cited by
- Cancer ResearchNeurologyImmunology
- Partner nations
- ChinaUnited StatesSwitzerland
In The Last Decade
Gui‐Xian Zhao
34 papers receiving 1.5k citations
Hit Papers
Peers
Comparison fields: 5 of 86
- Cancer Research 417
- Neurology 296
- Immunology 372
- Pathology and Forensic Medicine 315
- Physiology 70
Countries citing papers authored by Gui‐Xian Zhao
This map shows the geographic impact of Gui‐Xian Zhao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gui‐Xian Zhao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gui‐Xian Zhao more than expected).
Fields of papers citing papers by Gui‐Xian Zhao
This network shows the impact of papers produced by Gui‐Xian Zhao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gui‐Xian Zhao. The network helps show where Gui‐Xian Zhao may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Gui‐Xian Zhao, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2022 | 5 | |
| 3 | 2021 | 6 | |
| 4 | 2020 | 6 | |
| 5 | 2020 | 11 | |
| 6 | 2018 | 6 | |
| 7 | 2017 | 48 | |
| 8 | 2016 | 22 | |
| 9 | 2016 | 36 | |
| 10 | 2016 | 4 | |
| 11 | 2016 | 75 | |
| 12 | 2015 | 12 | |
| 13 | 2014 | 8 | |
| 14 | 2013 | 18 | |
| 15 | 2010 | 35 | |
| 16 | MicroRNA miR-326 regulates TH-17 differentiation and is associated with the pathogenesis of multiple sclerosisbreakdown → | 2009 | 650 |
| 17 | 2008 | 33 | |
| 18 | 2008 | 3 | |
| 19 | Mutation analysis of MURRl in Chinese patients with Wilson disease | 2006 | 1 |
| 20 | 2006 | 44 |
About Gui‐Xian Zhao
Gui‐Xian Zhao is a scholar working on Pathology and Forensic Medicine, Rheumatology and Neurology, having authored 35 papers that have together received 1.5k indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (16 papers), Systemic Lupus Erythematosus Research (9 papers), Peripheral Neuropathies and Disorders (4 papers), Genetic Neurodegenerative Diseases (4 papers), Immunotherapy and Immune Responses (4 papers), Cytokine Signaling Pathways and Interactions (4 papers), Neurological diseases and metabolism (3 papers) and Trace Elements in Health (3 papers). The work is most often cited by research in Cancer Research (417 citations), Neurology (296 citations) and Immunology (372 citations). Gui‐Xian Zhao has collaborated with scholars based in China, United States and Switzerland. Frequent co-authors include Zhi‐Ying Wu, Zhenxin Li, Changsheng Du, Zhiqiang Ye, Gang Pei, Shichao Huang, Jiuhong Kang, Chang Liu, Wan‐Jin Chen and Jie Lv. Their work appears in journals such as Journal of Neurology, Scientific Reports, Gene, The Journal of Immunology and Neuroscience Bulletin.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.