Min-Ting Lin

722 total citations
48 papers, 452 citations indexed

About

Min-Ting Lin is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Min-Ting Lin has authored 48 papers receiving a total of 452 indexed citations (citations by other indexed papers that have themselves been cited), including 35 papers in Molecular Biology, 15 papers in Cellular and Molecular Neuroscience and 14 papers in Genetics. Recurrent topics in Min-Ting Lin's work include Muscle Physiology and Disorders (16 papers), Neurogenetic and Muscular Disorders Research (14 papers) and Genetic Neurodegenerative Diseases (14 papers). Min-Ting Lin is often cited by papers focused on Muscle Physiology and Disorders (16 papers), Neurogenetic and Muscular Disorders Research (14 papers) and Genetic Neurodegenerative Diseases (14 papers). Min-Ting Lin collaborates with scholars based in China, United States and Japan. Min-Ting Lin's co-authors include Shen-Xing Murong, Wan‐Jin Chen, Ning Wang, Zhi‐Ying Wu, Zhiqiang Wang, Ning Wang, Ning Wang, Ming Jin, Lin Feng and Xinyi Liu and has published in prestigious journals such as Brain, Neurology and Gene.

In The Last Decade

Min-Ting Lin

47 papers receiving 444 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Min-Ting Lin China 13 279 122 110 70 69 48 452
Boris Pantic Italy 10 467 1.7× 17 0.1× 120 1.1× 40 0.6× 21 0.3× 10 569
Meerim K. Nurbaeva Germany 14 313 1.1× 16 0.1× 80 0.7× 31 0.4× 64 0.9× 18 490
Marta Elena Roque Argentina 11 186 0.7× 80 0.7× 58 0.5× 5 0.1× 112 1.6× 20 394
Ju Eun Oh South Korea 10 126 0.5× 38 0.3× 50 0.5× 13 0.2× 11 0.2× 24 360
Stephanie M.Y. Kong Australia 11 203 0.7× 22 0.2× 32 0.3× 22 0.3× 33 0.5× 14 464
Shiroh Maguchi Japan 11 128 0.5× 52 0.4× 28 0.3× 7 0.1× 23 0.3× 30 567
Claire Vinel France 9 270 1.0× 51 0.4× 38 0.3× 226 3.2× 17 0.2× 13 727
Kei Miyanaka Japan 6 108 0.4× 17 0.1× 44 0.4× 20 0.3× 23 0.3× 7 341
Daise Nunes Queiroz da Cunha Brazil 11 259 0.9× 39 0.3× 68 0.6× 306 4.4× 45 0.7× 29 574
Thomas A. Doser United States 6 321 1.2× 7 0.1× 45 0.4× 69 1.0× 54 0.8× 7 630

Countries citing papers authored by Min-Ting Lin

Since Specialization
Citations

This map shows the geographic impact of Min-Ting Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Min-Ting Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Min-Ting Lin more than expected).

Fields of papers citing papers by Min-Ting Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Min-Ting Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Min-Ting Lin. The network helps show where Min-Ting Lin may publish in the future.

Co-authorship network of co-authors of Min-Ting Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Min-Ting Lin. A scholar is included among the top collaborators of Min-Ting Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Min-Ting Lin. Min-Ting Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Xuan-Yu, Xinyuan Chen, Zhili Chen, et al.. (2025). Associations between CAG repeat size, brain and spinal cord volume loss, and motor symptoms in spinocerebellar ataxia type 3: a cohort study. Orphanet Journal of Rare Diseases. 20(1). 35–35. 1 indexed citations
2.
Cao, Chunyan, Feng Xu, Wenqi Lv, et al.. (2025). Bi-allelic loss-of function variants in ATP2A1 cause autosomal recessive Brody disease. QJM. 118(10). 786–788.
3.
Chen, Xinyuan, Mengcheng Li, Wei Lin, et al.. (2024). Disease Progression and Multiparametric Imaging Characteristics of Spinocerebellar Ataxia Type 3 With Spastic Paraplegia. Neurology Genetics. 10(3). e200162–e200162. 1 indexed citations
4.
Chen, Long, Ying Zheng, Xiaodan Lin, et al.. (2023). Association of 4qA-Specific Distal D4Z4 Hypomethylation With Disease Severity and Progression in Facioscapulohumeral Muscular Dystrophy. Neurology. 101(3). e225–e237. 9 indexed citations
5.
Cao, Chunyan, Long Chen, Xuejiao Chen, et al.. (2023). CGG repeat expansion in LOC642361/NUTM2B-AS1 typically presents as oculopharyngodistal myopathy. Journal of genetics and genomics. 51(2). 184–196. 4 indexed citations
6.
Feng, Lin, Ming Jin, Long Chen, et al.. (2023). Clinical features, imaging findings and molecular data of limb-girdle muscular dystrophies in a cohort of Chinese patients. Orphanet Journal of Rare Diseases. 18(1). 356–356. 5 indexed citations
7.
Yang, Jinshan, Pingping Chen, Min-Ting Lin, et al.. (2020). Ataxic Severity Is Positively Correlated With Fatigue in Spinocerebellar Ataxia Type 3 Patients. Frontiers in Neurology. 11. 266–266. 6 indexed citations
8.
Jin, Ming, Xiaodan Lin, Xin-Yi Liu, et al.. (2019). Rhabdomyolysis and respiratory insufficiency due to the common ETFDH mutation of c.250G>A in two patients with late-onset multiple acyl-CoA dehydrogenase deficiency. Chinese Medical Journal. 132(13). 1615–1618. 8 indexed citations
9.
Xu, Guorong, Lin Feng, Ming Jin, et al.. (2019). Clinical and genetic characterization of limb girdle muscular dystrophy R7 telethonin-related patients from three unrelated Chinese families. Neuromuscular Disorders. 30(2). 137–143. 14 indexed citations
10.
Xu, Guorong, Wei Hu, Chong Wang, et al.. (2018). High frequency of the TARDBP p.M337 V mutation among south-eastern Chinese patients with familial amyotrophic lateral sclerosis. BMC Neurology. 18(1). 35–35. 7 indexed citations
11.
Shang, Xianjin, Jinshan Yang, Pingping Chen, et al.. (2018). Homozygote of spinocerebellar Ataxia type 3 correlating with severe phenotype based on analyses of clinical features. Journal of the Neurological Sciences. 390. 111–114. 4 indexed citations
12.
Wang, Danni, Zhiqiang Wang, Lei Yan, et al.. (2017). Clinical and mutational characteristics of Duchenne muscular dystrophy patients based on a comprehensive database in South China. Neuromuscular Disorders. 27(8). 715–722. 15 indexed citations
13.
Liu, Xin-Yi, Ming Jin, Zhiqiang Wang, et al.. (2016). Skeletal Muscle Magnetic Resonance Imaging of the Lower Limbs in Late-onset Lipid Storage Myopathy with Electron Transfer Flavoprotein Dehydrogenase Gene Mutations. Chinese Medical Journal. 129(12). 1425–1431. 11 indexed citations
16.
Wei, Wei, et al.. (2012). [Prevalence of CYP2C19 polymorphisms involved in clopidogrel metabolism in Fujian Han population].. PubMed. 29(4). 420–5. 6 indexed citations
17.
Wang, Ning, et al.. (2009). Mutation and polymorphism analysis of SPG4 and SPG3A in Chinese patients with hereditary spastic paraplegia. Chin J Neurol. 42(4). 253–257. 1 indexed citations
19.
Wang, Ning, Chaodong Wang, Zhiqiang Wang, et al.. (2003). [Mechanism of translocation between chromosomes 4q and 10q in facioscapulohumeral muscular dystrophy].. PubMed. 83(8). 650–3. 2 indexed citations
20.
Lin, Min-Ting. (2001). Detection and analysis of mutations in Chinese patients with Wilson disease. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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