Goizane Marcaida

999 total citations
20 papers, 829 citations indexed

About

Goizane Marcaida is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Goizane Marcaida has authored 20 papers receiving a total of 829 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 5 papers in Physiology. Recurrent topics in Goizane Marcaida's work include Neuroscience and Neuropharmacology Research (8 papers), Mitochondrial Function and Pathology (3 papers) and Metabolism and Genetic Disorders (3 papers). Goizane Marcaida is often cited by papers focused on Neuroscience and Neuropharmacology Research (8 papers), Mitochondrial Function and Pathology (3 papers) and Metabolism and Genetic Disorders (3 papers). Goizane Marcaida collaborates with scholars based in Spain, Russia and Switzerland. Goizane Marcaida's co-authors include Vicente Felipo, María‐Dolores Miñana, Santiago Grisolı́a, Carlos Hermenegildo, Еlena Kosenko, Santiago Grisolía, Carmina Montoliú, Eugenio Grau, María Burgal and Begoña Laíz and has published in prestigious journals such as Brain Research, FEBS Letters and Journal of Neurochemistry.

In The Last Decade

Goizane Marcaida

20 papers receiving 803 citations

Peers

Goizane Marcaida

CMN

PHYSI

HEPAT

Eugenio Grau Spain

Marion Schneider Germany

F. Seguin France

Aleksandra Sinđić Croatia

Christophe Rocher France

R Mornex France

Ricardo Fernández Chile

Elizabeth M. Gordon United States

Yoshihiro Fujiwara Japan

Sara Missaglia Italy

Eugenio Grau Spain

Goizane Marcaida

156 ×0.5

124 ×0.4

CMN

142 ×0.6

91 ×0.5

PHYSI

87 ×0.6

HEPAT

Citations per year, relative to Goizane Marcaida Goizane Marcaida (= 1×) peers Eugenio Grau

Countries citing papers authored by Goizane Marcaida

Since Specialization

Citations

This map shows the geographic impact of Goizane Marcaida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Goizane Marcaida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Goizane Marcaida more than expected).

Fields of papers citing papers by Goizane Marcaida

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Goizane Marcaida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Goizane Marcaida. The network helps show where Goizane Marcaida may publish in the future.

Co-authorship network of co-authors of Goizane Marcaida

This figure shows the co-authorship network connecting the top 25 collaborators of Goizane Marcaida. A scholar is included among the top collaborators of Goizane Marcaida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Goizane Marcaida. Goizane Marcaida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ferrer, Isidró, et al.. (2016). Susceptibilidad genética del síndrome de Gilbert en población valenciana; eficiencia del test de ayuno. Revista Clínica Española. 217(1). 1–6. 3 indexed citations

Ferrer, Isidró, et al.. (2016). Genetic susceptibility to Gilbert's syndrome in a Valencian population; efficacy of the fasting test. Revista Clínica Española (English Edition). 217(1). 1–6. 3 indexed citations

Salinas, María, Maite López‐Garrigós, M. Teresa Agulló-Ortuño, et al.. (2011). Estudio piloto regional de evaluación del tiempo de respuesta de laboratorio según el tipo de cliente. Revista de Calidad Asistencial. 26(2). 104–110. 1 indexed citations

Salinas, María, Maite López‐Garrigós, Arturo Carratalá, et al.. (2011). Evaluación del patrón de solicitud de hemoglobina glucosilada por Atención Primaria: estudio piloto regional en la Comunidad Valenciana. Endocrinología y Nutrición. 58(5). 219–223. 4 indexed citations

Salinas, María, Maite López‐Garrigós, Julián Ruiz Díaz, et al.. (2011). Regional variations in test requiring patterns of general practitioners in Spain. Upsala Journal of Medical Sciences. 116(4). 247–251. 35 indexed citations

Salinas, María, Maite López‐Garrigós, Cristina Aguado, et al.. (2011). Evaluation of PSA testing by general practitioners: regional study in the autonomic Community of Valencia.. PubMed. 64(5). 435–40. 3 indexed citations

Salinas, María, Maite López‐Garrigós, Arturo Carratalá, et al.. (2011). An evaluation of glycosylated hemoglobin requesting patterns in a primary care setting: a pilot experience in the Valencian Community (Spain). Endocrinología y Nutrición (English Edition). 58(5). 219–223. 3 indexed citations

Treviño, Ana, Juan González‐García, Carmen de Mendoza, et al.. (2010). Prevalence of HTLV-1/2 Infections in Spain: A Cross-Sectional Hospital-Based Survey. AIDS Research and Human Retroviruses. 26(8). 861–864. 14 indexed citations

Diago, M., et al.. (2001). Long-term response to interferon plus ribavirin in patients with chronic hepatitis C refractory to interferon.. PubMed. 93(6). 353–63. 3 indexed citations

10.

Miñana, María‐Dolores, Еlena Kosenko, Goizane Marcaida, et al.. (1997). Modulation of Glutamine Synthesis in Cultured Astrocytes by Nitric Oxide. Cellular and Molecular Neurobiology. 17(4). 433–445. 35 indexed citations

11.

Marcaida, Goizane, María‐Dolores Miñana, Santiago Grisolı́a, & Vicente Felipo. (1997). Determination of intracellular ATP in primary cultures of neurons. Brain Research Protocols. 1(1). 75–78. 16 indexed citations

12.

Hermenegildo, Carlos, Goizane Marcaida, Carmina Montoliú, et al.. (1996). NMDA Receptor antagonists prevent acute ammonia toxicity in mice. Neurochemical Research. 21(10). 1237–1244. 121 indexed citations

13.

Grau, Eugenio, Goizane Marcaida, Carmina Montoliú, et al.. (1996). Effects of hyperammonemia on brain protein kinase C substrates. Metabolic Brain Disease. 11(3). 205–216. 16 indexed citations

14.

Marcaida, Goizane, Еlena Kosenko, María‐Dolores Miñana, Santiago Grisolía, & Vicente Felipo. (1996). Glutamate Induces a Calcineurin‐Mediated Dephosphorylation of Na⁺,K⁺‐ATPase that Results in Its Activation in Cerebellar Neurons in Culture. Journal of Neurochemistry. 66(1). 99–104. 75 indexed citations

15.

Miñana, María‐Dolores, et al.. (1995). Prenatal Exposure of Rats to Ammonia Impairs NMDA Receptor Function and Affords Delayed Protection Against Ammonia Toxicity and Glutamate Neurotoxicity. Journal of Neuropathology & Experimental Neurology. 54(5). 644–650. 19 indexed citations

16.

Marcaida, Goizane, et al.. (1995). Lack of correlation between glutamate-induced depletion of ATP and neuronal death in primary cultures of cerebellum. Brain Research. 695(2). 146–150. 51 indexed citations

17.

Marcaida, Goizane, María‐Dolores Miñana, María Burgal, Santiago Grisolı́a, & Vicente Felipo. (1995). Ammonia Prevents Activation of NMDA Receptors by Glutamate in Rat Cerebellar Neuronal Cultures. European Journal of Neuroscience. 7(12). 2389–2396. 69 indexed citations

18.

Felipo, Vicente, et al.. (1994). Molecular Mechanism of Acute Ammonia Toxicity and of its Prevention by L-Carnitine. Advances in experimental medicine and biology. 368. 65–77. 25 indexed citations

19.

Kosenko, Еlena, Eugenio Grau, María‐Dolores Miñana, et al.. (1994). Brain ATP Depletion Induced by Acute Ammonia Intoxication in Rats Is Mediated by Activation of the NMDA Receptor and Na⁺, K⁺‐ATPase. Journal of Neurochemistry. 63(6). 2172–2178. 165 indexed citations

20.

Marcaida, Goizane, Vicente Felipo, Carlos Hermenegildo, María‐Dolores Miñana, & Santiago Grisolı́a. (1992). Acute ammonia toxicity is mediated by the NMDA type of glutamate receptors. FEBS Letters. 296(1). 67–68. 168 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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