Eugenio Grau

464 total citations
11 papers, 403 citations indexed

About

Eugenio Grau is a scholar working on Molecular Biology, Physiology and Clinical Biochemistry. According to data from OpenAlex, Eugenio Grau has authored 11 papers receiving a total of 403 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Physiology and 4 papers in Clinical Biochemistry. Recurrent topics in Eugenio Grau's work include Mitochondrial Function and Pathology (4 papers), Metabolism and Genetic Disorders (4 papers) and Neuroscience and Neuropharmacology Research (3 papers). Eugenio Grau is often cited by papers focused on Mitochondrial Function and Pathology (4 papers), Metabolism and Genetic Disorders (4 papers) and Neuroscience and Neuropharmacology Research (3 papers). Eugenio Grau collaborates with scholars based in Spain and Russia. Eugenio Grau's co-authors include Vicente Felipo, María‐Dolores Miñana, Santiago Grisolı́a, Еlena Kosenko, Goizane Marcaida, Santiago Grisolía, Carmina Montoliú and Hugo Cabedo and has published in prestigious journals such as Hepatology, Journal of Neurochemistry and European Journal of Biochemistry.

In The Last Decade

Eugenio Grau

11 papers receiving 388 citations

Peers

Eugenio Grau

CMN

PHYSI

HEPAT

Santiago Grisolía Spain

L. Ratnakumari Canada

Richard C. Nielsen United States

Kara R. Vogel United States

Viruna Neergheen United Kingdom

Kirsi‐Marja Marnela Finland

T M Badger United States

Tanja Scherer Switzerland

A. W. Linnane Australia

Madhabi Barua United States

Santiago Grisolía Spain

Eugenio Grau

179 ×1.1

94 ×0.7

140 ×1.1

CMN

67 ×0.7

PHYSI

54 ×0.6

HEPAT

Citations per year, relative to Eugenio Grau Eugenio Grau (= 1×) peers Santiago Grisolía

Countries citing papers authored by Eugenio Grau

Since Specialization

Citations

This map shows the geographic impact of Eugenio Grau's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eugenio Grau with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eugenio Grau more than expected).

Fields of papers citing papers by Eugenio Grau

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eugenio Grau. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eugenio Grau. The network helps show where Eugenio Grau may publish in the future.

Co-authorship network of co-authors of Eugenio Grau

This figure shows the co-authorship network connecting the top 25 collaborators of Eugenio Grau. A scholar is included among the top collaborators of Eugenio Grau based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eugenio Grau. Eugenio Grau is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Grau, Eugenio, Goizane Marcaida, Carmina Montoliú, et al.. (1996). Effects of hyperammonemia on brain protein kinase C substrates. Metabolic Brain Disease. 11(3). 205–216. 16 indexed citations

Cabedo, Hugo, María‐Dolores Miñana, Eugenio Grau, Vicente Felipo, & Santiago Grisolı́a. (1996). Protein kinase C isoforms and cell proliferation in neuroblastoma cells. Molecular Brain Research. 37(1-2). 125–133. 7 indexed citations

Kosenko, Еlena, et al.. (1995). Nitroarginine, an inhibitor of nitric oxide synthetase, attenuates ammonia toxicity and ammonia-induced alterations in brain metabolism. Neurochemical Research. 20(4). 451–456. 70 indexed citations

Kosenko, Еlena, Eugenio Grau, María‐Dolores Miñana, et al.. (1994). Brain ATP Depletion Induced by Acute Ammonia Intoxication in Rats Is Mediated by Activation of the NMDA Receptor and Na⁺, K⁺‐ATPase. Journal of Neurochemistry. 63(6). 2172–2178. 165 indexed citations

Felipo, Vicente, Eugenio Grau, María‐Dolores Miñana, & Santiago Grisolı́a. (1993). Ammonium Injection Induces an N‐Methyl‐d‐Aspartate Receptor‐Mediated Proteolysis of the Microtubule‐Associated Protein MAP‐2. Journal of Neurochemistry. 60(5). 1626–1630. 66 indexed citations

Felipo, Vicente, Eugenio Grau, María‐Dolores Miñana, & Santiago Grisolı́a. (1993). Activation of NMDA Receptor Mediates the Toxicity of Ammonia and the Effects of Ammonia on the Microtubule-Associated Protein MAP-2. Advances in experimental medicine and biology. 341. 83–93. 9 indexed citations

Felipo, Vicente, Eugenio Grau, María‐Dolores Miñana, & Santiago Grisolı́a. (1993). Hyperammonemia decreases protein‐kinase‐C‐dependent phosphorylation of microtubule‐associated protein 2 and increases its binding to tubulin. European Journal of Biochemistry. 214(1). 243–249. 32 indexed citations

Grisolı́a, Santiago, María‐Dolores Miñana, Eugenio Grau, & Vicente Felipo. (1993). Control of Urea Synthesis and Ammonia Detoxification. Advances in experimental medicine and biology. 341. 1–12. 2 indexed citations

Grau, Eugenio, Vicente Felipo, María‐Dolores Miñana, & Santiago Grisolía. (1992). Treatment of hyperammonemia with carbamylglutamate in rats. Hepatology. 15(3). 446–448. 18 indexed citations

10.

Grau, Eugenio, et al.. (1992). The susceptibility of MAP-2 to proteolytic degradation increases when bound to tubulin. Neurochemical Research. 17(10). 967–971. 3 indexed citations

11.

Kosenko, Еlena, Vicente Felipo, María‐Dolores Miñana, Eugenio Grau, & Santiago Grisolı́a. (1991). Ammonium ingestion prevents depletion of hepatic energy metabolites induced by acute ammonium intoxication. Archives of Biochemistry and Biophysics. 290(2). 484–488. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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