Gláucia E. Callera

4.2k total citations
58 papers, 3.5k citations indexed

About

Gláucia E. Callera is a scholar working on Physiology, Endocrinology, Diabetes and Metabolism and Molecular Biology. According to data from OpenAlex, Gláucia E. Callera has authored 58 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Physiology, 18 papers in Endocrinology, Diabetes and Metabolism and 16 papers in Molecular Biology. Recurrent topics in Gláucia E. Callera's work include Nitric Oxide and Endothelin Effects (28 papers), Hormonal Regulation and Hypertension (18 papers) and Renin-Angiotensin System Studies (12 papers). Gláucia E. Callera is often cited by papers focused on Nitric Oxide and Endothelin Effects (28 papers), Hormonal Regulation and Hypertension (18 papers) and Renin-Angiotensin System Studies (12 papers). Gláucia E. Callera collaborates with scholars based in Canada, Brazil and United Kingdom. Gláucia E. Callera's co-authors include Rhian M. Touyz, Álvaro Yogi, Rita C. Tostes, Augusto C. Montezano, Ying He, Ernesto L. Schiffrin, Kevin D. Burns, Ana M. Briones, Tayze T. Antunes and Aurélie Nguyen Dinh Cat and has published in prestigious journals such as PLoS ONE, Circulation Research and Diabetes.

In The Last Decade

Gláucia E. Callera

57 papers receiving 3.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Gláucia E. Callera Canada 36 1.1k 1.1k 1.0k 931 679 58 3.5k
Álvaro Yogi Canada 31 792 0.7× 776 0.7× 655 0.6× 734 0.8× 537 0.8× 45 2.5k
Hirofumi Hitomi Japan 35 1.4k 1.3× 1.2k 1.2× 666 0.6× 942 1.0× 651 1.0× 98 3.7k
Aurélie Nguyen Dinh Cat United Kingdom 27 1.2k 1.1× 975 0.9× 622 0.6× 788 0.8× 699 1.0× 42 2.9k
Fatiha Tabet Australia 25 789 0.7× 662 0.6× 737 0.7× 1.2k 1.3× 875 1.3× 40 3.1k
Guillermo Zalba Spain 39 407 0.4× 1.0k 1.0× 1.6k 1.5× 1.0k 1.1× 348 0.5× 109 4.1k
Khalid Matrougui United States 40 547 0.5× 972 0.9× 963 0.9× 1.6k 1.7× 507 0.7× 90 4.1k
Livius V. d’Uscio United States 36 601 0.6× 1.7k 1.6× 2.2k 2.1× 998 1.1× 357 0.5× 76 4.0k
Takehide Ogihara Japan 48 1.3k 1.2× 873 0.8× 1.7k 1.6× 2.7k 2.9× 1.0k 1.5× 96 5.7k
Jeremy D. Ollerenshaw United States 14 472 0.4× 1.1k 1.1× 1.1k 1.1× 835 0.9× 414 0.6× 27 3.0k
Yoshihiro Taniyama Japan 22 448 0.4× 676 0.6× 1.1k 1.1× 1.4k 1.6× 312 0.5× 32 3.3k

Countries citing papers authored by Gláucia E. Callera

Since Specialization
Citations

This map shows the geographic impact of Gláucia E. Callera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gláucia E. Callera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gláucia E. Callera more than expected).

Fields of papers citing papers by Gláucia E. Callera

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gláucia E. Callera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gláucia E. Callera. The network helps show where Gláucia E. Callera may publish in the future.

Co-authorship network of co-authors of Gláucia E. Callera

This figure shows the co-authorship network connecting the top 25 collaborators of Gláucia E. Callera. A scholar is included among the top collaborators of Gláucia E. Callera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gláucia E. Callera. Gláucia E. Callera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Callera, Gláucia E., Thiago Bruder‐Nascimento, & Rhian M. Touyz. (2017). Assessment of Caveolae/Lipid Rafts in Isolated Cells. Methods in molecular biology. 1527. 251–269. 2 indexed citations
2.
Callera, Gláucia E., Tayze T. Antunes, José Wilson do Nascimento Corrêa, et al.. (2016). Differential renal effects of candesartan at high and ultra-high doses in diabetic mice–potential role of the ACE2/AT2R/Mas axis. Bioscience Reports. 36(5). 33 indexed citations
3.
Simplicio, Janaina A., Gabriel Tavares do Vale, Gláucia E. Callera, et al.. (2016). Acute Ethanol Intake Induces NAD(P)H Oxidase Activation and Rhoa Translocation in Resistance Arteries. Arquivos Brasileiros de Cardiologia. 107(5). 427–436. 6 indexed citations
4.
Antunes, Tayze T., Gláucia E. Callera, Ying He, et al.. (2016). Transient Receptor Potential Melastatin 7 Cation Channel Kinase. Hypertension. 67(4). 763–773. 33 indexed citations
5.
Callera, Gláucia E., et al.. (2015). Vitamin C prevents the endothelial dysfunction induced by acute ethanol intake. Life Sciences. 141. 99–107. 14 indexed citations
6.
Yogi, Álvaro, Gláucia E. Callera, Sarah O’Connor, et al.. (2013). Aldosterone signaling through transient receptor potential melastatin 7 cation channel (TRPM7) and its α-kinase domain. Cellular Signalling. 25(11). 2163–2175. 26 indexed citations
7.
Briones, Ana M., Aurélie Nguyen Dinh Cat, Gláucia E. Callera, et al.. (2012). Adipocytes Produce Aldosterone Through Calcineurin-Dependent Signaling Pathways. Hypertension. 59(5). 1069–1078. 290 indexed citations
8.
Callera, Gláucia E., Álvaro Yogi, Ana M. Briones, et al.. (2011). Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research. 91(4). 720–731. 42 indexed citations
9.
Rocha, Juliana Trevisan da, Gláucia E. Callera, Álvaro Yogi, et al.. (2011). Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways. Vascular Pharmacology. 56(1-2). 74–83. 24 indexed citations
10.
Yogi, Álvaro, Gláucia E. Callera, Sarah O’Connor, et al.. (2011). Dysregulation of renal transient receptor potential melastatin 6/7 but not paracellin-1 in aldosterone-induced hypertension and kidney damage in a model of hereditary hypomagnesemia. Journal of Hypertension. 29(7). 1400–1410. 36 indexed citations
11.
Yogi, Álvaro, Chantal Mercure, Gláucia E. Callera, et al.. (2008). Renal Redox-Sensitive Signaling, but Not Blood Pressure, Is Attenuated by Nox1 Knockout in Angiotensin II–Dependent Chronic Hypertension. Hypertension. 51(2). 500–506. 64 indexed citations
12.
Callera, Gláucia E., Ying He, Álvaro Yogi, et al.. (2008). Regulation of the novel Mg2+ transporter transient receptor potential melastatin 7 (TRPM7) cation channel by bradykinin in vascular smooth muscle cells. Journal of Hypertension. 27(1). 155–166. 62 indexed citations
13.
Yogi, Álvaro, Gláucia E. Callera, Rita C. Tostes, & Rhian M. Touyz. (2008). Bradykinin regulates calpain and proinflammatory signaling through TRPM7-sensitive pathways in vascular smooth muscle cells. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 296(2). R201–R207. 45 indexed citations
14.
Touyz, Rhian M., Ying He, Danesh Javeshghani, et al.. (2005). Angiotensin II-Dependent Chronic Hypertension and Cardiac Hypertrophy Are Unaffected by gp91phox-Containing NADPH Oxidase. Hypertension. 45(4). 530–537. 104 indexed citations
15.
Touyz, Rhian M., Ying He, Augusto C. Montezano, et al.. (2005). Differential regulation of transient receptor potential melastatin 6 and 7 cation channels by ANG II in vascular smooth muscle cells from spontaneously hypertensive rats. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 290(1). R73–R78. 89 indexed citations
16.
Xavier, Fabiano E., Álvaro Yogi, Gláucia E. Callera, et al.. (2004). Contribution of the endothelin and renin–angiotensin systems to the vascular changes in rats chronically treated with ouabain. British Journal of Pharmacology. 143(6). 794–802. 21 indexed citations
17.
Callera, Gláucia E., et al.. (2004). Changes in the vascular β‐adrenoceptor‐activated signalling pathway in 2Kidney‐1Clip hypertensive rats. British Journal of Pharmacology. 141(7). 1151–1158. 14 indexed citations
18.
Callera, Gláucia E., Álvaro Yogi, Rita C. Tostes, Luciana Venturini Rossoni, & Lusiane Maria Bendhack. (2004). Ca2+-Activated K+ Channels Underlying the Impaired Acetylcholine-Induced Vasodilation in 2K-1C Hypertensive Rats. Journal of Pharmacology and Experimental Therapeutics. 309(3). 1036–1042. 36 indexed citations
19.
Callera, Gláucia E. & Lusiane Maria Bendhack. (2003). Mechanisms Underlying the Contractile Response to Endothelin-1 in the Rat Renal Artery. Pharmacology. 68(3). 131–139. 8 indexed citations
20.
Callera, Gláucia E. & Lusiane Maria Bendhack. (1999). Contribution of sarcoplasmic reticulum calcium uptake and L-type calcium channels to altered vascular responsiveness in the aorta of renal hypertensive rats. General Pharmacology The Vascular System. 33(6). 457–466. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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