Thiago Bruder‐Nascimento

1.4k total citations
47 papers, 964 citations indexed

About

Thiago Bruder‐Nascimento is a scholar working on Physiology, Cardiology and Cardiovascular Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Thiago Bruder‐Nascimento has authored 47 papers receiving a total of 964 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Physiology, 15 papers in Cardiology and Cardiovascular Medicine and 12 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Thiago Bruder‐Nascimento's work include Adipose Tissue and Metabolism (12 papers), Adipokines, Inflammation, and Metabolic Diseases (9 papers) and Hormonal Regulation and Hypertension (9 papers). Thiago Bruder‐Nascimento is often cited by papers focused on Adipose Tissue and Metabolism (12 papers), Adipokines, Inflammation, and Metabolic Diseases (9 papers) and Hormonal Regulation and Hypertension (9 papers). Thiago Bruder‐Nascimento collaborates with scholars based in United States, Brazil and United Kingdom. Thiago Bruder‐Nascimento's co-authors include Rita C. Tostes, Eric J. Belin de Chantemèle, Rhian M. Touyz, Rafael M. Costa, Fabíola Mestriner, Jessica Faulkner, Sandra Cordellini, Gláucia E. Callera, Simone Kennard and Karla B Neves and has published in prestigious journals such as Circulation, PLoS ONE and Scientific Reports.

In The Last Decade

Thiago Bruder‐Nascimento

46 papers receiving 956 citations

Peers

Thiago Bruder‐Nascimento
Núbia S. Lobato Brazil
Katarina Lalić Serbia
Avshalom Leibowitz Israel
Rainer U. Pliquett Germany
Wataru Kameda Japan
Ping Gu China
Emilia Ragno Italy
Esther Guiu‐Jurado Spain
Andréia Carvalho Alzamora Brazil
Núbia S. Lobato Brazil
Thiago Bruder‐Nascimento
Citations per year, relative to Thiago Bruder‐Nascimento Thiago Bruder‐Nascimento (= 1×) peers Núbia S. Lobato

Countries citing papers authored by Thiago Bruder‐Nascimento

Since Specialization
Citations

This map shows the geographic impact of Thiago Bruder‐Nascimento's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thiago Bruder‐Nascimento with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thiago Bruder‐Nascimento more than expected).

Fields of papers citing papers by Thiago Bruder‐Nascimento

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thiago Bruder‐Nascimento. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thiago Bruder‐Nascimento. The network helps show where Thiago Bruder‐Nascimento may publish in the future.

Co-authorship network of co-authors of Thiago Bruder‐Nascimento

This figure shows the co-authorship network connecting the top 25 collaborators of Thiago Bruder‐Nascimento. A scholar is included among the top collaborators of Thiago Bruder‐Nascimento based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thiago Bruder‐Nascimento. Thiago Bruder‐Nascimento is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bruder‐Nascimento, Thiago, et al.. (2025). Adipose Tissue‐Derived Adipokines in Vascular Physiology and Pathophysiology: Insights and Implications. Comprehensive physiology. 15(3). e70018–e70018.
2.
Bruder‐Nascimento, Thiago, David W. Stepp, Rodger D. MacArthur, et al.. (2025). CD4 + T Cells Expressing Viral Proteins Induce HIV-Associated Endothelial Dysfunction and Hypertension Through Interleukin 1α–Mediated Increases in Endothelial NADPH Oxidase 1. Circulation. 151(16). 1187–1203. 5 indexed citations
3.
Costa, Rafael M., et al.. (2025). Vascular Contractility Relies on Integrity of Progranulin Pathway: Insights Into Mitochondrial Function. Journal of the American Heart Association. 14(3). e037640–e037640. 2 indexed citations
4.
Alves, Juliano, et al.. (2024). NADPH oxidase 4-derived hydrogen peroxide counterbalances testosterone-induced endothelial dysfunction and migration. American Journal of Physiology-Endocrinology and Metabolism. 327(1). E1–E12. 8 indexed citations
5.
Costa, Rafael M., et al.. (2023). Suppressed vascular Rho-kinase activation is a protective cardiovascular mechanism in obese female mice. Bioscience Reports. 43(7). 5 indexed citations
6.
Costa, Rafael M., et al.. (2023). Melatonin prevents overproduction of reactive oxygen species and vascular dysfunction induced by cyclophosphamide. Life Sciences. 338. 122361–122361. 12 indexed citations
7.
Atawia, Reem T., Simone Kennard, Galina Antonova, et al.. (2023). Type 1 Diabetes Impairs Endothelium-Dependent Relaxation Via Increasing Endothelial Cell Glycolysis Through Advanced Glycation End Products, PFKFB3, and Nox1-Mediated Mechanisms. Hypertension. 80(10). 2059–2071. 8 indexed citations
8.
Alves, Juliano, et al.. (2023). Progranulin Maintains Blood Pressure and Vascular Tone Dependent on EphrinA2 and Sortilin1 Receptors and Endothelial Nitric Oxide Synthase Activation. Journal of the American Heart Association. 12(16). e030353–e030353. 5 indexed citations
9.
Padovan, Júlio C., et al.. (2023). Reactive Oxygen Species Are Central Mediators of Vascular Dysfunction and Hypertension Induced by Ethanol Consumption. Antioxidants. 12(10). 1813–1813. 9 indexed citations
10.
Alves, Juliano, et al.. (2023). Vascular injury associated with ethanol intake is driven by AT1 receptor and mitochondrial dysfunction. Biomedicine & Pharmacotherapy. 169. 115845–115845. 4 indexed citations
11.
Alves, Juliano, et al.. (2022). PROGRESSION OF VASCULAR FUNCTION AND BLOOD PRESSURE IN A MOUSE MODEL OF KAWASAKI DISEASE. Shock. 59(1). 74–81. 2 indexed citations
12.
Alves, Juliano, et al.. (2022). Adipokines: Deciphering the cardiovascular signature of adipose tissue. Biochemical Pharmacology. 206. 115324–115324. 16 indexed citations
13.
Chantemèle, Eric J. Belin de, et al.. (2021). CCR5 antagonist treatment inhibits vascular injury by regulating NADPH oxidase 1. Biochemical Pharmacology. 195. 114859–114859. 14 indexed citations
14.
15.
Legeay, Samuel, Galina Antonova, Simone Kennard, et al.. (2020). Selective deficiency in endothelial PTP1B protects from diabetes and endoplasmic reticulum stress-associated endothelial dysfunction via preventing endothelial cell apoptosis. Biomedicine & Pharmacotherapy. 127. 110200–110200. 18 indexed citations
16.
Callera, Gláucia E., Thiago Bruder‐Nascimento, & Rhian M. Touyz. (2017). Assessment of Caveolae/Lipid Rafts in Isolated Cells. Methods in molecular biology. 1527. 251–269. 2 indexed citations
17.
18.
Bruder‐Nascimento, Thiago, et al.. (2015). Effects of exercise training on stress-induced vascular reactivity alterations: role of nitric oxide and prostanoids. Brazilian Journal of Physical Therapy. 19(3). 177–185. 8 indexed citations
19.
Bruder‐Nascimento, Thiago, Dijon Henrique Salomé de Campos, Antônio Carlos Cicogna, & Sandra Cordellini. (2014). Chronic Stress Improves NO- and Ca2+ Flux-Dependent Vascular Function: A Pharmacological Study. Arquivos Brasileiros de Cardiologia. 104(3). 226–33. 12 indexed citations
20.
Bruder‐Nascimento, Thiago, et al.. (2014). The involvement of aldosterone on vascular insulin resistance: implications in obesity and type 2 diabetes. Diabetology & Metabolic Syndrome. 6(1). 90–90. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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