Gergely Imre

656 total citations
20 papers, 516 citations indexed

About

Gergely Imre is a scholar working on Molecular Biology, Immunology and Infectious Diseases. According to data from OpenAlex, Gergely Imre has authored 20 papers receiving a total of 516 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 7 papers in Immunology and 4 papers in Infectious Diseases. Recurrent topics in Gergely Imre's work include Cell death mechanisms and regulation (11 papers), interferon and immune responses (4 papers) and Inflammasome and immune disorders (4 papers). Gergely Imre is often cited by papers focused on Cell death mechanisms and regulation (11 papers), interferon and immune responses (4 papers) and Inflammasome and immune disorders (4 papers). Gergely Imre collaborates with scholars based in Germany, Hungary and United States. Gergely Imre's co-authors include Rudolf Mihalik, István Peták, Gábor Barna, Zsuzsanna A. Dunai, Krishnaraj Rajalingam, Tamás Korcsmáros, Pál I. Bauer, Volker Dötsch, Jan Heering and Bernd Thiede and has published in prestigious journals such as The EMBO Journal, PLoS ONE and British Journal of Cancer.

In The Last Decade

Gergely Imre

19 papers receiving 511 citations

Peers

Countries citing papers authored by Gergely Imre

Since Specialization

Citations

This map shows the geographic impact of Gergely Imre's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gergely Imre with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gergely Imre more than expected).

Fields of papers citing papers by Gergely Imre

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gergely Imre. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gergely Imre. The network helps show where Gergely Imre may publish in the future.

Co-authorship network of co-authors of Gergely Imre

This figure shows the co-authorship network connecting the top 25 collaborators of Gergely Imre. A scholar is included among the top collaborators of Gergely Imre based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gergely Imre. Gergely Imre is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	ALIX mediates reversible gasdermin-D pore formation via the endosomal pathway to limit pyroptosis by active membrane repair 2025 ·Cell Death and Disease ·(unknown), (unknown), (unknown), John Perry, (unknown), (unknown), Radhey S. Kaushik, Adam D. Hoppe, (unknown), (unknown), Jaime López-Mosqueda, Gergely Imre	0
2	Pyroptosis in health and disease 2024 ·American Journal of Physiology-Cell Physiology ·Gergely Imre	26
3	The E3 Ligase TRIM25 Impairs Apoptotic Cell Death in Colon Carcinoma Cells via Destabilization of Caspase-7 mRNA: A Possible Role of hnRNPH1 2023 ·Cells ·(unknown), (unknown), (unknown), Süleyman Bozkurt, (unknown), Stefan Gauer, Gergely Imre, Christian Münch, Josef Pfeilschifter, W. Eberhardt	4
4	Reciprocal abrogation of PKM isoforms: contradictory outcomes and differing impact of splicing signal on CRISPR /Cas9 mediates gene editing in keratinocytes 2022 ·FEBS Journal ·(unknown), (unknown), Gergely Imre, Dominik C. Fuhrmann, Josef Pfeilschifter, Rajkumar Vutukuri, Frank Schnütgen, Ilka Wittig, (unknown), Stefan L. Frank, Itamar Goren	6
5	The caspase-2 substrate p54nrb exhibits a multifaceted role in tumor cell death susceptibility via gene regulatory functions 2022 ·Cell Death and Disease ·(unknown), Ute Distler, (unknown), (unknown), Manuel Kaulich, Koraljka Husnjak, W. Eberhardt, Krishnaraj Rajalingam, Stefan Tenzer, Josef Pfeilschifter, Gergely Imre	5
6	C6 Ceramide (d18:1/6:0) as a Novel Treatment of Cutaneous T Cell Lymphoma 2021 ·Cancers ·(unknown), (unknown), Gergely Imre, Stefan Kippenberger, Markus Meißner, Dominique Thomas, Sandra Trautmann, Jean‐Philippe Merlio, Edith Chevret, Roland Kaufmann, Josef Pfeilschifter, Alexander Koch, (unknown)	9
7	Application of pseudotyped virus particles to monitor Ebola virus and SARS-CoV-2 viral entry in human cell lines 2021 ·STAR Protocols ·(unknown), (unknown), Josef Pfeilschifter, Gergely Imre	2
8	The sphingosine kinase 1 activator, K6PC-5, attenuates Ebola virus infection 2021 ·iScience ·Gergely Imre, Verena Krähling, (unknown), Sandra Trautmann, Nerea Ferreirós, M. Javad Aman, Fatah Kashanchi, Krishnaraj Rajalingam, Stefan Pöhlmann, Stephan Becker, Dagmar Meyer zu Heringdorf, Josef Pfeilschifter	7
9	Cell death signalling in virus infection 2020 ·Cellular Signalling ·Gergely Imre	61
10	The involvement of regulated cell death forms in modulating the bacterial and viral pathogenesis 2020 ·International review of cell and molecular biology ·Gergely Imre	16
11	Apoptosis inhibitor 5 is an endogenous inhibitor of caspase‐2 2017 ·EMBO Reports ·Gergely Imre, Jean Berthelet, Jan Heering, Sebastian Kehrloesser, (unknown), Byung Il Lee, Bernd Thiede, Volker Dötsch, Krishnaraj Rajalingam	30
12	Transposon-based gene delivery vectors for gene therapy 2015 ·Acta Biologica Szegediensis ·Andrea Nagy, Gergely Imre, Lajos Mátés	2
13	Attenuation of the ELAV1-like protein HuR sensitizes adenocarcinoma cells to the intrinsic apoptotic pathway by increasing the translation of caspase-2L 2014 ·Cell Death and Disease ·Christoph Winkler, Anke Doller, Gergely Imre, A. M. Badawi, Tobias Schmid, Sebastian Schulz, (unknown), Josef Pfeilschifter, Krishnaraj Rajalingam, W. Eberhardt	22
14	Caspase‐2 is an initiator caspase responsible for pore‐forming toxin‐mediated apoptosis 2012 ·The EMBO Journal ·Gergely Imre, Jan Heering, (unknown), Matthias Husmann, Bernd Thiede, Dagmar Meyer zu Heringdorf, Douglas R. Green, Gijs R. van den Brink, Bhanu Sinha, Volker Dötsch, Krishnaraj Rajalingam	73
15	Staurosporine Induces Necroptotic Cell Death under Caspase-Compromised Conditions in U937 Cells 2012 ·PLoS ONE ·Zsuzsanna A. Dunai, Gergely Imre, Gábor Barna, Tamás Korcsmáros, István Peták, Pál I. Bauer, Rudolf Mihalik	85
16	High expression of RelA/p65 is associated with activation of nuclear factor-κB-dependent signaling in pancreatic cancer and marks a patient population with poor prognosis 2007 ·British Journal of Cancer ·(unknown), Markus Boehm, Volker Gekeler, Marcus Bahra, Jan M. Langrehr, P. Neuhaus, Carsten Denkert, Gergely Imre, Claudia M Weller, (unknown), Silvia Niesporek, Joseph M Jacob, (unknown), Claus Scheidereit, Glen Kristiansen	124
17	Cystein cathepsin and Hsp90 activities determine the balance between apoptotic and necrotic cell death pathways in caspase-compromised U937 cells 2007 ·Biochimica et Biophysica Acta (BBA) - Molecular Cell Research ·Gergely Imre, Zsuzsanna A. Dunai, István Peták, Rudolf Mihalik	5
18	In ovo vitelline duct ligation results in transient changes of bursal microenvironments 2005 ·Immunology ·(unknown), Gergely Imre, Botond Z. Igyártó, (unknown), Rudolf Mihalik, (unknown), Imre Oláh, Attila Magyar	8
19	Proteasome inhibitors abolish cell death downstream of caspase activation during anti-microtubule drug-induced apoptosis in leukemia cells. 2005 ·PubMed ·(unknown), István Peták, Gergely Imre, Gábor Barna, (unknown), Anna Sebestyén, Janet A. Houghton, Rudolf Mihalik, László Kopper	4
20	Cathepsin B-independent abrogation of cell death by CA-074-OMe upstream of lysosomal breakdown 2004 ·Cell Death and Differentiation ·Rudolf Mihalik, Gergely Imre, István Peták, B. Szende, L. Kópper	27

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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